A novel NOD1- and CagA-independent pathway of interleukin-8 induction mediated by the Helicobacter pylori type IV secretion system.
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Differential inflammatory response to Helicobacter pylori infection: etiology and clinical outcomesExploring alternative treatments for Helicobacter pylori infectionPathobiology of Helicobacter pylori-Induced Gastric Cancer.Helicobacter pylori downregulates expression of human β-defensin 1 in the gastric mucosa in a type IV secretion-dependent fashion.Presence of terminal EPIYA phosphorylation motifs in Helicobacter pylori CagA contributes to IL-8 secretion, irrespective of the number of repeats.Functional plasticity in the type IV secretion system of Helicobacter pyloriHelicobacter pylori cag pathogenicity island (cagPAI) involved in bacterial internalization and IL-8 induced responses via NOD1- and MyD88-dependent mechanisms in human biliary epithelial cells.Helicobacter pylori CagL Y58/E59 mutation turns-off type IV secretion-dependent delivery of CagA into host cellsNucleotide Binding Oligomerization Domain 1 Is an Essential Signal Transducer in Human Epithelial Cells Infected with Helicobacter pylori That Induces the Transepithelial Migration of Neutrophils.Mutual reinforcement of inflammation and carcinogenesis by the Helicobacter pylori CagA oncoprotein.Coiled coil rich proteins (Ccrp) influence molecular pathogenicity of Helicobacter pyloriPreservation of Helicobacter pylori CagA Translocation and Host Cell Proinflammatory Responses in the Face of CagL Hypervariability at Amino Acid Residues 58/59.The Helicobacter pylori cytotoxin CagA is essential for suppressing host heat shock protein expression.Mechanism and Function of Type IV Secretion During Infection of the Human Host.Life in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori.The Helicobacter pylori Autotransporter ImaA Tempers the Bacterium's Interaction with α5β1 Integrin.H. pylori virulence factors: influence on immune system and pathology.Inflammation, immunity, vaccines for Helicobacter pylori infection.Signal transduction of Helicobacter pylori during interaction with host cell protein receptors of epithelial and immune cells.How Helicobacter pylori senses, targets and interacts with the gastric epithelium.Helicobacter pylori CagA induced interleukin-8 secretion in gastric epithelial cellsInflammatory signaling pathways induced by Helicobacter pylori in primary human gastric epithelial cells.Expressional profiles of transcription factors in the progression of Helicobacter pylori-associated gastric carcinoma based on protein/DNA array analysis.The Human Stomach in Health and Disease: Infection Strategies by Helicobacter pylori.NF-κB Signaling in Gastric Cancer.Helicobacter pylori utilizes DNA shuffling to modulate the gastric inflammatory response.TIFA Signaling in Gastric Epithelial Cells Initiates the cag Type 4 Secretion System-Dependent Innate Immune Response to Helicobacter pylori Infection.The Middle Fragment of Helicobacter pylori CagA Induces Actin Rearrangement and Triggers Its Own Uptake into Gastric Epithelial Cells.A role for the tfs3 ICE-encoded type IV secretion system in pro-inflammatory signalling by the Helicobacter pylori Ser/Thr kinase, CtkA.Helicobacter pylori modulates host cell responses by CagT4SS-dependent translocation of an intermediate metabolite of LPS inner core heptose biosynthesis.Systematic site-directed mutagenesis of the Helicobacter pylori CagL protein of the Cag type IV secretion system identifies novel functional domains.Integrin engagement by the helical RGD motif of the Helicobacter pylori CagL protein is regulated by pH-induced displacement of a neighboring helix.Macrophages recognize the Helicobacter pylori type IV secretion system in the absence of toll-like receptor signalling.Analysis of the Relative Contribution of Phagocytosis, LC3-Associated Phagocytosis, and Canonical Autophagy During Helicobacter pylori Infection of Macrophages.Helicobacter pylori induces vascular endothelial growth factor production in gastric epithelial cells through hypoxia-inducible factor-1α-dependent pathway.Chemokines and antimicrobial peptides have a cag-dependent early response to Helicobacter pylori infection in primary human gastric epithelial cells.Helicobacter pylori antigen HP0986 (TieA) interacts with cultured gastric epithelial cells and induces IL8 secretion via NF-κB mediated pathway.Gastric lymphoma: association with Helicobacter pylori outer membrane protein Q (HopQ) and cytotoxic-pathogenicity activity island (CPAI) genes.Helicobacter pylori Type IV Secretion System and Its Adhesin Subunit, CagL, Mediate Potent Inflammatory Responses in Primary Human Endothelial Cells.The Helicobacter pylori adhesin protein HopQ exploits the dimer interface of human CEACAMs to facilitate translocation of the oncoprotein CagA.
P2860
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P2860
A novel NOD1- and CagA-independent pathway of interleukin-8 induction mediated by the Helicobacter pylori type IV secretion system.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
A novel NOD1- and CagA-indepen ...... lori type IV secretion system.
@en
A novel NOD1- and CagA-indepen ...... lori type IV secretion system.
@nl
type
label
A novel NOD1- and CagA-indepen ...... lori type IV secretion system.
@en
A novel NOD1- and CagA-indepen ...... lori type IV secretion system.
@nl
prefLabel
A novel NOD1- and CagA-indepen ...... lori type IV secretion system.
@en
A novel NOD1- and CagA-indepen ...... lori type IV secretion system.
@nl
P2093
P2860
P50
P356
P1476
A novel NOD1- and CagA-indepen ...... ylori type IV secretion system
@en
P2093
Jyeswei Guan
Mona Anoushiravani Tafreshi
P2860
P304
P356
10.1111/CMI.12055
P577
2012-11-21T00:00:00Z