Late developmental plasticity in the T helper 17 lineage. - Wikidata - awgldk - TriplyDB

Late developmental plasticity in the T helper 17 lineage.

Late developmental plasticity in the T helper 17 lineage.

http://www.wikidata.org/entity/Q39265291

about

IL-1 and T Helper Immune Responses T helper 17 cell heterogeneity and pathogenicity in autoimmune disease Fate mapping of IL-17-producing T cells in inflammatory responses Generation of pathogenic T(H)17 cells in the absence of TGF-β signalling Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling Mitochondrial Stat3, the Need for Design Thinking The Treg/Th17 Axis: A Dynamic Balance Regulated by the Gut Microbiome Advances in T Helper 17 Cell Biology: Pathogenic Role and Potential Therapy in Multiple Sclerosis The First Line of Defense: The Effects of Alcohol on Post-Burn Intestinal Barrier, Immune Cells, and Microbiome Th17 plasticity and its changes associated with inflammatory bowel disease Development and Function of Protective and Pathologic Memory CD4 T Cells T Helper Subsets, Peripheral Plasticity, and the Acute Phase Protein, α1-Antitrypsin Development and survival of Th17 cells within the intestines: the influence of microbiome- and diet-derived signals Review: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyond Contextual functions of antigen-presenting cells in the gastrointestinal tract Diversity of IL-17-producing T lymphocytes Th17 Cell Plasticity and Functions in Cancer Immunity Linking vitamin d deficiency to inflammatory bowel disease Central role of gimap5 in maintaining peripheral tolerance and T cell homeostasis in the gut Contribution of the IL-17/IL-23 axis to the pathogenesis of inflammatory bowel disease Functional and pathogenic differences of Th1 and Th17 cells in experimental autoimmune encephalomyelitis Lithium controls central nervous system autoimmunity through modulation of IFN-γ signaling Protein kinase C θ regulates the phenotype of murine CD4+ Th17 cells Th17 effector cells support B cell responses outside of germinal centres The IL-23-IL-17 immune axis: from mechanisms to therapeutic testing Mechanisms underlying helper T-cell plasticity: implications for immune-mediated disease.Plasticity of T reg at infected sites.Exploiting IL-17-producing CD4+ and CD8+ T cells to improve cancer immunotherapy in the clinic Multiple Sclerosis and Obesity: Possible Roles of Adipokines CD4+ T Cell Fate in Glomerulonephritis: A Tale of Th1, Th17, and Novel Treg Subtypes Along the Axis between Type 1 and Type 2 Immunity; Principles Conserved in Evolution from Fish to Mammals The IL-17 pathway as a major therapeutic target in autoimmune diseases Osteopontin expression by CD103- dendritic cells drives intestinal inflammation IFN-γ-Producing T-Helper 17.1 Cells Are Increased in Sarcoidosis and Are More Prevalent than T-Helper Type 1 Cells Type II Activation of Macrophages and Microglia by Immune Complexes Enhances Th17 Biasing in an IL-6-Independent Manner A validated regulatory network for Th17 cell specification T-bet represses T(H)17 differentiation by preventing Runx1-mediated activation of the gene encoding RORγt Marking and quantifying IL-17A-producing cells in vivo Differentiation of effector CD4 T cell populations (*)Inflammatory bowel disease

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Late developmental plasticity in the T helper 17 lineage.

http://www.wikidata.org/entity/Q39265291

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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2009年學術文章

@zh-hant

name

Late developmental plasticity in the T helper 17 lineage.

@en

Late developmental plasticity in the T helper 17 lineage.

@nl

type

label

Late developmental plasticity in the T helper 17 lineage.

@en

Late developmental plasticity in the T helper 17 lineage.

@nl

prefLabel

Late developmental plasticity in the T helper 17 lineage.

@en

Late developmental plasticity in the T helper 17 lineage.

@nl

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J.IMMUNI.2008.11.005

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Late developmental plasticity in the T helper 17 lineage.

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Casey T Weaver

http://www.w3.org/2001/XMLSchema#string

Charles O Elson

http://www.w3.org/2001/XMLSchema#string

Craig L Maynard

http://www.w3.org/2001/XMLSchema#string

Dongquan Chen

http://www.w3.org/2001/XMLSchema#string

Henrietta Turner

http://www.w3.org/2001/XMLSchema#string

James R Oliver

http://www.w3.org/2001/XMLSchema#string

Yun Kyung Lee

http://www.w3.org/2001/XMLSchema#string

P2860

Monoclonal Anti–Interleukin 23 Reverses Active Colitis in a T Cell–Mediated Model in Mice

Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12

IL-17s adopt a cystine knot fold: structure and activity of a novel cytokine, IL-17F, and implications for receptor binding

A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

A critical role for transforming growth factor-beta but not interleukin 4 in the suppression of T helper type 1-mediated colitis by CD45RB(low) CD4+ T cells

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17

Transforming growth factor-beta induces development of the T(H)17 lineage

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells

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92-107

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10.1016/J.IMMUNI.2008.11.005

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19119024

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