Ras activation induces expression of Raet1 family NK receptor ligands.
about
Immune surveillance of unhealthy cells by natural killer cellsRegulation of ligands for the NKG2D activating receptor.Recognition of tumors by the innate immune system and natural killer cells.Balancing the innate immune system in tumor development.New insights into cancer immunoediting and its three component phases--elimination, equilibrium and escapeImmunosurveillance and therapy of multiple myeloma are CD226 dependent.An Immunocompetent Mouse Model for MLL/AF9 Leukemia Reveals the Potential of Spontaneous Cytotoxic T-Cell Response to an Antigen Expressed in Leukemia CellsMicroenvironment abnormalities and lymphomagenesis: Immunological aspects.Immunosurveillance and immunotherapy of tumors by innate immune cells.NKG2D ligands link oncogenic RAS to innate immunity.NKG2D ligands mediate immunosurveillance of senescent cells.The modulation of the cell-cycle: a sentinel to alert the NK cells of dangers.NK cell activating receptor ligand expression in lymphangioleiomyomatosis is associated with lung function declineA Herpesviral induction of RAE-1 NKG2D ligand expression occurs through release of HDAC mediated repression.Xanthine oxidoreductase is required for genotoxic stress-induced NKG2D ligand expression and gemcitabine-mediated antitumor activityShaping of NK cell responses by the tumor microenvironmentMolecular mechanisms of natural killer cell activation in response to cellular stress.Regulation of self-ligands for activating natural killer cell receptors.Advances in NKG2D ligand recognition and responses by NK cells.Molecular Bases for the Regulation of NKG2D Ligands in Cancer.Does the immune system naturally protect against cancer?RAS Promotes Proliferation and Resistances to Apoptosis in Meningioma.The proto-oncogene Myc drives expression of the NK cell-activating NKp30 ligand B7-H6 in tumor cells.Adjuvant-dependent innate and adaptive immune signatures of risk of SIVmac251 acquisition.IL-18-dependent NKG2D ligand upregulation on accessory cells is mediated by the PI3K/GSK-3 pathway.RB mutation and RAS overexpression induce resistance to NK cell-mediated cytotoxicity in glioma cells.p38 MAPK differentially controls NK activating ligands at transcriptional and post-transcriptional level on multiple myeloma cells.Dysregulated cellular functions and cell stress pathways provide critical cues for activating and targeting natural killer cells to transformed and infected cells.Impaired NK cell recognition of vemurafenib-treated melanoma cells is overcome by simultaneous application of histone deacetylase inhibitors.Oncogenes in immune cells as potential therapeutic targets.
P2860
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P2860
Ras activation induces expression of Raet1 family NK receptor ligands.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Ras activation induces expression of Raet1 family NK receptor ligands.
@en
Ras activation induces expression of Raet1 family NK receptor ligands.
@nl
type
label
Ras activation induces expression of Raet1 family NK receptor ligands.
@en
Ras activation induces expression of Raet1 family NK receptor ligands.
@nl
prefLabel
Ras activation induces expression of Raet1 family NK receptor ligands.
@en
Ras activation induces expression of Raet1 family NK receptor ligands.
@nl
P2093
P356
P1476
Ras activation induces expression of Raet1 family NK receptor ligands
@en
P2093
James J Tan
Neha Kamran
Samantha S W Ho
P304
P356
10.4049/JIMMUNOL.1200965
P407
P577
2012-07-13T00:00:00Z