Blockade of the 5-HT3 receptor for days causes sustained relief from mechanical allodynia following spinal cord injury.
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Neurogenic bowel dysfunction in patients with spinal cord injury, myelomeningocele, multiple sclerosis and Parkinson's diseaseSpinal cord transection-induced allodynia in rats--behavioral, physiopathological and pharmacological characterizationLack of analgesic efficacy of spinal ondansetron on thermal and mechanical hypersensitivity following spinal nerve ligation in the rat.Activation of descending pain-facilitatory pathways from the rostral ventromedial medulla by cholecystokinin elicits release of prostaglandin-E₂ in the spinal cord.Trigeminal-rostral ventromedial medulla circuitry is involved in orofacial hyperalgesia contralateral to tissue injury.Ondansetron reverses antihypersensitivity from clonidine in rats after peripheral nerve injury: role of γ-aminobutyric acid in α2-adrenoceptor and 5-HT3 serotonin receptor analgesia.Serotonin enhances urinary bladder nociceptive processing via a 5-HT3 receptor mechanism.Ion channel blockers for the treatment of neuropathic pain.Ondansetron does not prevent physical dependence in patients taking opioid medications chronically for pain control.
P2860
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P2860
Blockade of the 5-HT3 receptor for days causes sustained relief from mechanical allodynia following spinal cord injury.
description
2009 nî lūn-bûn
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name
Blockade of the 5-HT3 receptor ...... following spinal cord injury.
@en
Blockade of the 5-HT3 receptor ...... following spinal cord injury.
@nl
type
label
Blockade of the 5-HT3 receptor ...... following spinal cord injury.
@en
Blockade of the 5-HT3 receptor ...... following spinal cord injury.
@nl
prefLabel
Blockade of the 5-HT3 receptor ...... following spinal cord injury.
@en
Blockade of the 5-HT3 receptor ...... following spinal cord injury.
@nl
P2093
P356
P1476
Blockade of the 5-HT3 receptor ...... following spinal cord injury.
@en
P2093
Lynne C Weaver
Mark A Oatway
Yuhua Chen
P304
P356
10.1002/JNR.21860
P577
2009-02-01T00:00:00Z