Brain phenotypes in two FGFR2 mouse models for Apert syndrome. - Wikidata - awgldk - TriplyDB

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

http://www.wikidata.org/entity/Q39326391

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If the skull fits: magnetic resonance imaging and microcomputed tomography for combined analysis of brain and skull phenotypes in the mouse Craniofacial divergence by distinct prenatal growth patterns in Fgfr2 mutant mice Apert syndrome: temporal lobe abnormalities on fetal brain imaging.Morphological comparison of the craniofacial phenotypes of mouse models expressing the Apert FGFR2 S252W mutation in neural crest- or mesoderm-derived tissues.FGF/FGFR signaling coordinates skull development by modulating magnitude of morphological integration: evidence from Apert syndrome mouse models Beyond the closed suture in apert syndrome mouse models: evidence of primary effects of FGFR2 signaling on facial shape at birth.Fasciculation and guidance of spinal motor axons in the absence of FGFR2 signaling Novel molecular pathways elicited by mutant FGFR2 may account for brain abnormalities in Apert syndrome.Unilateral and bilateral expression of a quantitative trait: asymmetry and symmetry in coronal craniosynostosis.From shape to cells: mouse models reveal mechanisms altering palate development in Apert syndrome Mesodermal expression of Fgfr2S252W is necessary and sufficient to induce craniosynostosis in a mouse model of Apert syndrome.Understanding craniosynostosis as a growth disorder Deformed Skull Morphology Is Caused by the Combined Effects of the Maldevelopment of Calvarias, Cranial Base and Brain in FGFR2-P253R Mice Mimicking Human Apert Syndrome.Embryonic craniofacial bone volume and bone mineral density in Fgfr2(+/P253R) and nonmutant mice.The role of vertebrate models in understanding craniosynostosis.Hand in glove: brain and skull in development and dysmorphogenesis.Integration of Brain and Skull in Prenatal Mouse Models of Apert and Crouzon Syndromes.Tissue-specific responses to aberrant FGF signaling in complex head phenotypes.Loss-of-Function of Gli3 in Mice Causes Abnormal Frontal Bone Morphology and Premature Synostosis of the Interfrontal Suture.Mouse models of Apert syndrome.Apert syndrome with fused thalami.Postnatal brain and skull growth in an Apert syndrome mouse model.Research advances in Apert syndrome

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Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

http://www.wikidata.org/entity/Q39326391

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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2010年學術文章

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2010年學術文章

@zh-hant

name

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

@en

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

@nl

type

label

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

@en

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

@nl

prefLabel

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

@en

Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

@nl

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Developmental Dynamics

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Brain phenotypes in two FGFR2 mouse models for Apert syndrome.

@en

P2093

Cheryl A Hill

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Joan T Richtsmeier

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Jordan R Austin

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Neus Martinez-Abadias

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Thomas Neuberger

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Yingli Wang

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P2860

Self-renewing and differentiating properties of cortical neural stem cells are selectively regulated by basic fibroblast growth factor (FGF) signaling via specific FGF receptors

Phenotypic integration of neurocranium and brain

Cellular dynamics and tissue interactions of the dura mater during head development.

Histological staining methods preparatory to laser capture microdissection significantly affect the integrity of the cellular RNA.

Tissue interactions with underlying dura mater inhibit osseous obliteration of developing cranial sutures.

Cranial sutures require tissue interactions with dura mater to resist osseous obliteration in vitro.

Dura mater maintains rat cranial sutures in vitro by regulating suture cell proliferation and collagen production.

Activation of p38 MAPK pathway in the skull abnormalities of Apert syndrome Fgfr2(+P253R) mice.

Management of craniosynostoses.

Intracranial pressure and intracranial volume in children with craniosynostosis.

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10.1002/DVDY.22218

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3

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239

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Kristina Aldridge

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41030423

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