Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
about
LB100, a small molecule inhibitor of PP2A with potent chemo- and radio-sensitizing potentialPP2A regulatory subunit Bα controls endothelial contractility and vessel lumen integrity via regulation of HDAC7.Regulating Set-β's Subcellular Localization Toggles Its Function between Inhibiting and Promoting Axon Growth and Regeneration.Endothelial permeability and VE-cadherin: a wacky comradeship.Semaphorin3A elevates vascular permeability and contributes to cerebral ischemia-induced brain damage.Endothelial secreted factors suppress mitogen deprivation-induced autophagy and apoptosis in glioblastoma stem-like cells.The N-terminal Set-β Protein Isoform Induces Neuronal DeathLuteolin Impacts on the DNA Damage Pathway in Oral Squamous Cell Carcinoma.Semaphorin-3C signals through Neuropilin-1 and PlexinD1 receptors to inhibit pathological angiogenesis.Desert Hedgehog/Patch2 Axis Contributes to Vascular Permeability and Angiogenesis in Glioblastoma.Emerging roles of Semaphorins in the regulation of epithelial and endothelial junctionsPhosphatase regulation of intercellular junctions.Critical multiple angiogenic factors secreted by glioblastoma stem-like cells underline the need for combinatorial anti-angiogenic therapeutic strategies.Vascular permeability and drug delivery in cancers.Morphogens and blood-brain barrier function in health and disease.The C-terminus region of β-arrestin1 modulates VE-cadherin expression and endothelial cell permeability.Glioblastoma stem-like cells secrete the pro-angiogenic VEGF-A factor in extracellular vesicles.β-escin selectively targets the glioblastoma-initiating cell population and reduces cell viability.Protein phosphatase 2A activity is required for functional adherent junctions in endothelial cells.The E3 ubiquitin ligase MARCH3 controls the endothelial barrier.Extracellular vesicle-transported Semaphorin3A promotes vascular permeability in glioblastoma.Apelin, the Devil Inside Brain Tumors.[Tumor angiogenesis: when the Tree of Life turns bad].Isthmin is a novel vascular permeability inducer that functions through cell-surface GRP78-mediated Src activation.
P2860
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P2860
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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name
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
@en
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
@nl
type
label
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
@en
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
@nl
prefLabel
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
@en
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
@nl
P2093
P2860
P356
P1476
Semaphorin 3A elevates endothelial cell permeability through PP2A inactivation.
@en
P2093
Cedric Auffray
Eva-Maria Galan-Moya
Garance Kettler
Herve Chneiweiss
Jagoda K Hebda
Julie Dwyer
Julie Gavard
Nicolas Bidere
Sonia Dubois
P2860
P304
P356
10.1242/JCS.108282
P407
P577
2012-06-08T00:00:00Z