Arsenic induces apoptosis in myoblasts through a reactive oxygen species-induced endoplasmic reticulum stress and mitochondrial dysfunction pathway. - Wikidata - awgldk - TriplyDB

Arsenic induces apoptosis in myoblasts through a reactive oxygen species-induced endoplasmic reticulum stress and mitochondrial dysfunction pathway.

Arsenic induces apoptosis in myoblasts through a reactive oxygen species-induced endoplasmic reticulum stress and mitochondrial dysfunction pathway.

http://www.wikidata.org/entity/Q39343288

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Role of endoplasmic reticulum stress in drug-induced toxicity Low-Concentration Arsenic Trioxide Inhibits Skeletal Myoblast Cell Proliferation via a Reactive Oxygen Species-Independent Pathway Effects of arsenic on osteoblast differentiation in vitro and on bone mineral density and microstructure in rats.The roles of mitoferrin-2 in the process of arsenic trioxide-induced cell damage in human gliomas.Thimerosal-induced apoptosis in mouse C2C12 myoblast cells occurs through suppression of the PI3K/Akt/survivin pathway Protective role of AMP-activated protein kinase-evoked autophagy on an in vitro model of ischemia/reperfusion-induced renal tubular cell injury.Tetramethylpyrazine (TMP) protects against sodium arsenite-induced nephrotoxicity by suppressing ROS production, mitochondrial dysfunction, pro-inflammatory signaling pathways and programed cell death.Schisandrae Fructus Supplementation Ameliorates Sciatic Neurectomy-Induced Muscle Atrophy in Mice Arsenic-exposed Keratinocytes Exhibit Differential microRNAs Expression Profile; Potential Implication of miR-21, miR-200a and miR-141 in Melanoma Pathway.Oxidative stress-mediated intrinsic apoptosis in human promyelocytic leukemia HL-60 cells induced by organic arsenicals.Nephroprotective effect of astaxanthin against trivalent inorganic arsenic-induced renal injury in wistar rats.Cytotoxin-induced NADPH oxides activation: roles in regulation of cell death.Toxicogenomics-based identification of mechanisms for direct immunotoxicity.ATF4 regulates arsenic trioxide-mediated NADPH oxidase, ER-mitochondrial crosstalk and apoptosis.Activating transcription factor 4 underlies the pathogenesis of arsenic trioxide-mediated impairment of macrophage innate immune functions.Arsenite-induced endoplasmic reticulum-dependent apoptosis through disturbance of calcium homeostasis in HBE cell line.Taurine resumed neuronal differentiation in arsenite-treated N2a cells through reducing oxidative stress, endoplasmic reticulum stress, and mitochondrial dysfunction.Arsenic inhibits hedgehog signaling during P19 cell differentiation.Retardation of C2C12 myoblast cell proliferation by exposure to low-temperature atmospheric plasma.Antiapoptotic effect of a novel synthetic peptide from bovine muscle and MPG peptide on H2O2-induced C2C12 cells.Arsenic trioxide co-exposure potentiates benzo(a)pyrene genotoxicity by enhancing the oxidative stress in human lung adenocarcinoma cell.Unfolded protein response (UPR) signaling regulates arsenic trioxide-mediated macrophage innate immune function disruption.Arsenic trioxide induces unfolded protein response in vascular endothelial cells.Inorganic arsenic exposure induces E2F-dependent G0/G1 arrest via an increase in retinoblastoma family protein p130 in B-cell lymphoma A20 cells.The chemical disruption of human metabolism.Effects of a Sublethal and Transient Stress of the Endoplasmic Reticulum on the Mitochondrial Population.Sodium Arsenite-Induced Learning and Memory Impairment Is Associated with Endoplasmic Reticulum Stress-Mediated Apoptosis in Rat Hippocampus.Arsenic Trioxide Induces Apoptosis via Specific Signaling Pathways in HT-29 Colon Cancer Cells.Toxicity of Arsenic (III) on Isolated Liver Mitochondria: A New Mechanistic Approach.Biochemical and Molecular Alterations Following Arsenic-Induced Oxidative Stress and Mitochondrial Dysfunction in Rat Brain.Arsenic increases Pi-mediated vascular calcification and induces premature senescence in vascular smooth muscle cells.Citrinin induces apoptosis in human HCT116 colon cancer cells through endoplasmic reticulum stress.Isorhynchophylline Attenuates MPP+-Induced Apoptosis Through Endoplasmic Reticulum Stress- and Mitochondria-Dependent Pathways in PC12 Cells: Involvement of Antioxidant Activity.Inorganic Arsenic Induces NRF2-Regulated Antioxidant Defenses in Both Cerebral Cortex and Hippocampus in Vivo.Extracellular polysaccharides purified from Aureobasidium pullulans SM‑2001 (Polycan) inhibit dexamethasone‑induced muscle atrophy in mice.Low-level arsenic causes proteotoxic stress and not oxidative stress.The nanotoxicology revolution.

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P2860

Arsenic induces apoptosis in myoblasts through a reactive oxygen species-induced endoplasmic reticulum stress and mitochondrial dysfunction pathway.

http://www.wikidata.org/entity/Q39343288

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Arsenic induces apoptosis in m ...... chondrial dysfunction pathway.

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Arsenic induces apoptosis in m ...... chondrial dysfunction pathway.

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Arsenic induces apoptosis in m ...... chondrial dysfunction pathway.

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Chun-Fa Huang

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Keh-Sung Tsai

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Rong-Sen Yang

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Shing-Hwa Liu

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Ya-Wen Chen

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Yuan-Peng Yen

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P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

Evidence that Ser87 of BimEL is phosphorylated by Akt and regulates BimEL apoptotic function

An endoplasmic reticulum stress-specific caspase cascade in apoptosis. Cytochrome c-independent activation of caspase-9 by caspase-12

Roles of CHOP/GADD153 in endoplasmic reticulum stress

The Bcl-2 apoptotic switch in cancer development and therapy

AKT/PKB signaling: navigating downstream

Signal integration in the endoplasmic reticulum unfolded protein response

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Emerging roles of caspase-3 in apoptosis

Cellular and molecular regulation of muscle regeneration

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