Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
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Mice Lacking Ras-GRF1 Show Contextual Fear Conditioning but not Spatial Memory Impairments: Convergent Evidence from Two Independently Generated Mouse Mutant Lines.Differential involvement of Ras-GRF1 and Ras-GRF2 in L-DOPA-induced dyskinesiaRASGRF2 regulates alcohol-induced reinforcement by influencing mesolimbic dopamine neuron activity and dopamine release.A translational systems biology approach in both animals and humans identifies a functionally related module of accumbal genes involved in the regulation of reward processing and binge drinking in males.A single nucleotide polymorphism associated with reduced alcohol intake in the RASGRF2 gene predicts larger cortical volumes but faster longitudinal ventricular expansion in the elderly.Convergence of dopamine and glutamate signaling onto striatal ERK activation in response to drugs of abuse.NF1 Is a Direct G Protein Effector Essential for Opioid Signaling to Ras in the Striatum.ERK/MAPK Signaling Is Required for Pathway-Specific Striatal Motor Functions.Lovastatin corrects ERK pathway hyperactivation in fragile X syndrome: potential of platelet's signaling cascades as new outcome measures in clinical trials.The endocrine-brain-aging triad where many paths meet: female reproductive hormone changes at midlife and their influence on circuits important for learning and memory.Acquisition and expression of conditioned taste aversion differentially affects extracellular signal regulated kinase and glutamate receptor phosphorylation in rat prefrontal cortex and nucleus accumbens.Dependence-induced ethanol drinking and GABA neurotransmission are altered in Alk deficient mice.Impairment of cocaine-mediated behaviours in mice by clinically relevant Ras-ERK inhibitors.Neuronal signaling and behavior.Behavioral and biochemical evidence of the role of acetaldehyde in the motivational effects of ethanol.Synaptic GluN2B/CaMKII-α Signaling Induces Synapto-Nuclear Transport of ERK and Jacob.Long-term behavioral and biochemical effects of an ultra-low dose of Δ9-tetrahydrocannabinol (THC): neuroprotection and ERK signaling.Genetic enhancement of Ras-ERK pathway does not aggravate L-DOPA-induced dyskinesia in mice but prevents the decrease induced by lovastatin
P2860
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P2860
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
description
2011 nî lūn-bûn
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2011年の論文
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2011年論文
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2011年論文
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2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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2011年论文
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2011年论文
@zh-cn
name
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
@en
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
@nl
type
label
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
@en
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
@nl
prefLabel
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
@en
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
@nl
P2860
P356
P1476
Ras-ERK Signaling in Behavior: Old Questions and New Perspectives.
@en
P2093
Riccardo Brambilla
Stefania Fasano
P2860
P356
10.3389/FNBEH.2011.00079
P577
2011-11-24T00:00:00Z