about
Human tribbles, a protein family controlling mitogen-activated protein kinase cascadesA novel mammalian expression screen exploiting green fluorescent protein-based transcription detection in single cells.Feedback loops in intracellular signal processing and their potential for identifying novel signalling proteins.Advanced technologies for studies on protein interactomes.Bioinformatics Analysis of the FREM1 Gene-Evolutionary Development of the IL-1R1 Co-Receptor, TILRR.Enhanced macrophage tribbles-1 expression in murine experimental atherosclerosisTILRR, a novel IL-1RI co-receptor, potentiates MyD88 recruitment to control Ras-dependent amplification of NF-kappaBIdentification of 34 novel proinflammatory proteins in a genome-wide macrophage functional screenTRIB2 confers resistance to anti-cancer therapy by activating the serine/threonine protein kinase AKT.Tribbles: a family of kinase-like proteins with potent signalling regulatory function.A TNF variant that associates with susceptibility to musculoskeletal disease modulates thyroid hormone receptor binding to control promoter activationHunting for genes by functional screens.Multi-Compartmentalisation in the MAPK Signalling Pathway Contributes to the Emergence of Oscillatory Behaviour and to Ultrasensitivity.A46R and A52R from vaccinia virus are antagonists of host IL-1 and toll-like receptor signaling.Differential IL-1β secretion by monocyte subsets is regulated by Hsp27 through modulating mRNA stability.The tribbles gene family and lipoprotein metabolism.Tribbles at the cross-roads….Tribbles in inflammation.Tribbles role in reproduction.Oncosuppressive functions of tribbles pseudokinase 3.Evidence for a role of TRIB3 in the regulation of megakaryocytopoiesisLoss of Tribbles pseudokinase-3 promotes Akt-driven tumorigenesis via FOXO inactivation.Regulating STING in health and diseaseTribbles-1 and -2 are tumour suppressors, down-regulated in human acute myeloid leukaemia.Expression of tak1 and tram induces synergistic pro-inflammatory signalling and adjuvants DNA vaccines.Tribbles-2 is a novel regulator of inflammatory activation of monocytes.LDL uptake by monocytes in response to inflammation is MAPK dependent but independent of tribbles protein expression.Synergistic effect of Avemar on proinflammatory cytokine production and Ras-mediated cell activation.Member of the CREB/ATF protein family, but not CREB alpha plays an active role in BLV tax trans activation in vivo.Competition between members of the tribbles pseudokinase protein family shapes their interactions with mitogen activated protein kinase pathways.TRIB3 suppresses tumorigenesis by controlling mTORC2/AKT/FOXO signaling.Rapid secretion of interleukin-1beta by microvesicle shedding.The IL-1RI Co-Receptor TILRR (FREM1 Isoform 2) Controls Aberrant Inflammatory Responses and Development of Vascular Disease.Distinct control of MyD88 adapter-dependent and Akt kinase-regulated responses by the interleukin (IL)-1RI co-receptor, TILRRGeneration of a novel mouse model for the inducible depletion of macrophages in vivo.Ultrasound-enhanced transgene expression in vascular cells is not dependent upon cavitation-induced free radicals.A downstream regulatory element activates the bovine leukemia virus promoter.Analysis of innate immune signal transduction with autocatalytic expression vectors.Particle-Induced Osteolysis Is Mediated by TIRAP/Mal in Vitro and in Vivo: Dependence on Adherent Pathogen-Associated Molecular Patterns.Individual susceptibility to periprosthetic osteolysis is associated with altered patterns of innate immune gene expression in response to pro-inflammatory stimuli.
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description
hulumtues
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onderzoeker
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հետազոտող
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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Endre Kiss-Toth
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P1053
A-8596-2014
P106
P21
P31
P3829
P496
0000-0003-4406-4017