Epithelial-mesenchymal transition in breast cancer correlates with high histological grade and triple-negative phenotype.
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The Role of Forkhead Box Protein M1 in Breast Cancer Progression and Resistance to TherapyPanepoxydone targets NF-kB and FOXM1 to inhibit proliferation, induce apoptosis and reverse epithelial to mesenchymal transition in breast cancerRole of SMC1 in overcoming drug resistance in triple negative breast cancerCirculating breast tumor cells exhibit dynamic changes in epithelial and mesenchymal compositionLuminal breast cancer cell lines overexpressing ZNF703 are resistant to tamoxifen through activation of Akt/mTOR signalingPLA2G7 associates with hormone receptor negativity in clinical breast cancer samples and regulates epithelial-mesenchymal transition in cultured breast cancer cellsGD2 expression in breast cancer.Can biomarker assessment on circulating tumor cells help direct therapy in metastatic breast cancer?Synergistic antitumor effects of S-1 with eribulin in vitro and in vivo for triple-negative breast cancer cell lines.Relation between radiographic BI-RADS scores and triple negativity in patients with ductal carcinomas.The BCL9-2 proto-oncogene governs estrogen receptor alpha expression in breast tumorigenesisEMT and EGFR in CTCs cytokeratin negative non-metastatic breast cancerHRG-β1-driven ErbB3 signaling induces epithelial-mesenchymal transition in breast cancer cells.The role of microRNA-200 in progression of human colorectal and breast cancer.Cancer-associated fibroblasts induce high mobility group box 1 and contribute to resistance to doxorubicin in breast cancer cells.KLF6-SV1 drives breast cancer metastasis and is associated with poor survivalAn Autoimmune Response Signature Associated with the Development of Triple-Negative Breast Cancer Reflects Disease Pathogenesis.Acantholytic squamous cell carcinoma in upper aerodigestive tract: histopathology, immunohistochemical profile and epithelial mesenchymal transition phenotype change.GLI1 regulates a novel neuropilin-2/α6β1 integrin based autocrine pathway that contributes to breast cancer initiation.Clinical Significance of CK19 Negative Breast Cancer.The fragile X protein binds mRNAs involved in cancer progression and modulates metastasis formation.Inflammatory factors of the tumor microenvironment induce plasticity in nontransformed breast epithelial cells: EMT, invasion, and collapse of normally organized breast texturesAre synchronous and metachronous bilateral breast cancers different? An immunohistochemical analysis aimed at intrinsic tumor phenotype.MiR-155-mediated loss of C/EBPβ shifts the TGF-β response from growth inhibition to epithelial-mesenchymal transition, invasion and metastasis in breast cancer.Superior mesenteric artery margin in pancreaticoduodenectomy for pancreatic adenocarcinomaEGF induces epithelial-mesenchymal transition through phospho-Smad2/3-Snail signaling pathway in breast cancer cells.MicroRNA-222 Expression as a Predictive Marker for Tumor Progression in Hormone Receptor-Positive Breast CancerBiochemical and molecular mechanisms for the association between obesity, chronic inflammation, and breast cancer.BRMS1L suppresses breast cancer metastasis by inducing epigenetic silence of FZD10.Vimentin as a poor prognostic factor for triple-negative breast cancer.Brachyury, a vaccine target, is overexpressed in triple-negative breast cancer.E-cadherin expression phenotypes associated with molecular subtypes in invasive non-lobular breast cancer: evidence from a retrospective study and meta-analysis.Anterior gradient 2 downregulation in a subset of pancreatic ductal adenocarcinoma is a prognostic factor indicative of epithelial-mesenchymal transition.Pathological expression of tissue factor confers promising antitumor response to a novel therapeutic antibody SC1 in triple negative breast cancer and pancreatic adenocarcinoma.Primary breast tumor-derived cellular models: characterization of tumorigenic, metastatic, and cancer-associated fibroblasts in dissociated tumor (DT) cultures.Cancer stem cell and epithelial-mesenchymal transition markers predict worse outcome in metaplastic carcinoma of the breast.Vimentin, zeb1 and Sip1 are up-regulated in triple-negative and basal-like breast cancers: association with an aggressive tumour phenotype.Overcoming Resistance to Endocrine Therapy in Breast Cancer: New Approaches to a Nagging Problem.UNC0638, a G9a inhibitor, suppresses epithelial‑mesenchymal transition‑mediated cellular migration and invasion in triple negative breast cancer.Endothelial‑to‑mesenchymal transition in human idiopathic dilated cardiomyopathy.
P2860
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P2860
Epithelial-mesenchymal transition in breast cancer correlates with high histological grade and triple-negative phenotype.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Epithelial-mesenchymal transit ...... and triple-negative phenotype.
@en
Epithelial-mesenchymal transit ...... and triple-negative phenotype.
@nl
type
label
Epithelial-mesenchymal transit ...... and triple-negative phenotype.
@en
Epithelial-mesenchymal transit ...... and triple-negative phenotype.
@nl
prefLabel
Epithelial-mesenchymal transit ...... and triple-negative phenotype.
@en
Epithelial-mesenchymal transit ...... and triple-negative phenotype.
@nl
P2093
P2860
P1433
P1476
Epithelial-mesenchymal transit ...... and triple-negative phenotype
@en
P2093
Jungsuk An
Young-joon Ryu
Youngseok Lee
P2860
P304
P356
10.1111/J.1365-2559.2012.04195.X
P577
2012-03-22T00:00:00Z