Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR. - Wikidata - awgldk - TriplyDB

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

http://www.wikidata.org/entity/Q39378953

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Peptide neuromodulation in invertebrate model systems Serotonin control of thermotaxis memory behavior in nematode Caenorhabditis elegans Recovery from an acute infection in C. elegans requires the GATA transcription factor ELT-2 Neuronal Goα and CAPS regulate behavioral and immune responses to bacterial pore-forming toxins Neuronal reprograming of protein homeostasis by calcium-dependent regulation of the heat shock response RFX transcription factor DAF-19 regulates 5-HT and innate immune responses to pathogenic bacteria in Caenorhabditis elegans Genes that act downstream of sensory neurons to influence longevity, dauer formation, and pathogen responses in Caenorhabditis elegans Phosphorylation of the conserved transcription factor ATF-7 by PMK-1 p38 MAPK regulates innate immunity in Caenorhabditis elegans Nucleolar proteins suppress Caenorhabditis elegans innate immunity by inhibiting p53/CEP-1 It takes nerves to fight infections: insights on neuro-immune interactions from C. elegans Vulnerability-Based Critical Neurons, Synapses, and Pathways in the Caenorhabditis elegans Connectome Sensory perception and aging in model systems: from the outside in.Quantitative mapping of a digenic behavioral trait implicates globin variation in C. elegans sensory behaviors Catecholamine receptor polymorphisms affect decision-making in C. elegans.Counterbalance between BAG and URX neurons via guanylate cyclases controls lifespan homeostasis in C. elegans Analysis of NPR-1 reveals a circuit mechanism for behavioral quiescence in C. elegans.Alterations in gene expression in Caenorhabditis elegans associated with organophosphate pesticide intoxication and recovery.Increased sleep promotes survival during a bacterial infection in Drosophila.The Natural Biotic Environment of Caenorhabditis elegans.C. elegans germline-deficient mutants respond to pathogen infection using shared and distinct mechanisms.An essential role for XBP-1 in host protection against immune activation in C. elegans.The FMRFamide-Like Peptide Family in Nematodes A conserved PMK-1/p38 MAPK is required in caenorhabditis elegans tissue-specific immune response to Yersinia pestis infection.Chemosensory and hyperoxia circuits in C. elegans males influence sperm navigational capacity.Insulin production and signaling in renal tubules of Drosophila is under control of tachykinin-related peptide and regulates stress resistance.Caenorhabditis elegans as an alternative model host for legionella pneumophila, and protective effects of Bifidobacterium infantis.Measurements of behavioral quiescence in Caenorhabditis elegans.Dissection of genetic pathways in C. elegans Role of the nervous system in the control of proteostasis during innate immune activation: insights from C. elegans Neuropeptide receptors NPR-1 and NPR-2 regulate Caenorhabditis elegans avoidance response to the plant stress hormone methyl salicylate Genetic dissection of Anopheles gambiae gut epithelial responses to Serratia marcescens Infection intensity-dependent responses of Anopheles gambiae to the African malaria parasite Plasmodium falciparum.Contrasting invertebrate immune defense behaviors caused by a single gene, the Caenorhabditis elegans neuropeptide receptor gene npr-1 Neuronal energy-sensing pathway promotes energy balance by modulating disease tolerance.Genetics of Intraspecies Variation in Avoidance Behavior Induced by a Thermal Stimulus in Caenorhabditis elegans.A secreted bacterial peptidoglycan hydrolase enhances tolerance to enteric pathogens.Genetic Screen Reveals Link between the Maternal Effect Sterile Gene mes-1 and Pseudomonas aeruginosa-induced Neurodegeneration in Caenorhabditis elegans.Feeding and the rhodopsin family g-protein coupled receptors in nematodes and arthropods FLP-4 neuropeptide and its receptor in a neuronal circuit regulate preference choice through functions of ASH-2 trithorax complex in Caenorhabditis elegans Neuropeptidergic Signaling and Active Feeding State Inhibit Nociception in Caenorhabditis elegans

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Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

http://www.wikidata.org/entity/Q39378953

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

@en

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

@nl

type

label

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

@en

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

@nl

prefLabel

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

@en

Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

@nl

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Innate immunity in Caenorhabditis elegans is regulated by neurons expressing NPR-1/GPCR.

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Alejandro Aballay

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Evan Macosko

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Katie L Styer

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Sarah E Steele

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Varsha Singh

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P2860

Regulation of DAF-2 receptor signaling by human insulin and ins-1, a member of the unusually large and diverse C. elegans insulin gene family

p38 MAPK regulates expression of immune response genes and contributes to longevity in C. elegans

A distributed chemosensory circuit for oxygen preference in C. elegans

Natural variation in a neuropeptide Y receptor homolog modifies social behavior and food response in C. elegans

Molecular mechanisms of bacterial virulence elucidated using a Pseudomonas aeruginosa-Caenorhabditis elegans pathogenesis model

Killing of Caenorhabditis elegans by Pseudomonas aeruginosa used to model mammalian bacterial pathogenesis

Two neurons mediate diet-restriction-induced longevity in C. elegans

A conserved p38 MAP kinase pathway in Caenorhabditis elegans innate immunity

Social feeding in Caenorhabditis elegans is induced by neurons that detect aversive stimuli.

GATA transcription factor required for immunity to bacterial and fungal pathogens.

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10.1126/SCIENCE.1163673

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322

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Cornelia I Bargmann

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23265380

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18801967

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G protein-coupled receptor

Caenorhabditis elegans

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2831475

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