Cholesterol-enriched membrane microdomains are required for inducing host cell cytoskeleton rearrangements in response to attaching-effacing Escherichia coli.
about
Type Three Secretion System in Attaching and Effacing PathogensRNAi screen of Salmonella invasion shows role of COPI in membrane targeting of cholesterol and Cdc42'Clustering' SIRPα into the plasma membrane lipid microdomains is required for activated monocytes and macrophages to mediate effective cell surface interactions with CD47.The N-terminal amphipathic region of the Escherichia coli type III secretion system protein EspD is required for membrane insertion and functionHuman Genome-Wide RNAi Screen for Host Factors That Facilitate Salmonella Invasion Reveals a Role for Potassium Secretion in Promoting Internalization.How cholesterol regulates endothelial biomechanics.Enteropathogenic Escherichia coli subverts phosphatidylinositol 4,5-bisphosphate and phosphatidylinositol 3,4,5-trisphosphate upon epithelial cell infectionThe Bordetella type III secretion system effector BteA contains a conserved N-terminal motif that guides bacterial virulence factors to lipid rafts.Hijacking and Use of Host Lipids by Intracellular Pathogens.Protein kinase C mediates enterohemorrhagic Escherichia coli O157:H7-induced attaching and effacing lesions.Enterohemorrhagic Escherichia coli O157:H7 Shiga toxins inhibit gamma interferon-mediated cellular activationDetergent-resistant microdomains mediate activation of host cell signaling in response to attaching-effacing bacteria.Lipid raft-dependent uptake, signalling and intracellular fate of Porphyromonas gingivalis in mouse macrophages.Lactobacilli antagonize biological effects of enterohaemorrhagic Escherichia coli in vitro.Surface-layer protein extracts from Lactobacillus helveticus inhibit enterohaemorrhagic Escherichia coli O157:H7 adhesion to epithelial cells.Amino acid residues within enterohemorrhagic Escherichia coli O157:H7 Tir involved in phosphorylation, alpha-actinin recruitment, and Nck-independent pedestal formation.Disruption of tight junctions and induction of proinflammatory cytokine responses in colonic epithelial cells by Campylobacter jejuni.Role of host cell polarity and leading edge properties in Pseudomonas type III secretion.Enteropathogenic Escherichia coli, Samonella, Shigella and Yersinia: cellular aspects of host-bacteria interactions in enteric diseases.Pore-forming Activity of the Escherichia coli Type III Secretion System Protein EspD.Conditioned medium from enterohemorrhagic Escherichia coli-infected T84 cells inhibits signal transducer and activator of transcription 1 activation by gamma interferon.
P2860
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P2860
Cholesterol-enriched membrane microdomains are required for inducing host cell cytoskeleton rearrangements in response to attaching-effacing Escherichia coli.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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name
Cholesterol-enriched membrane ...... ing-effacing Escherichia coli.
@en
Cholesterol-enriched membrane ...... ing-effacing Escherichia coli.
@nl
type
label
Cholesterol-enriched membrane ...... ing-effacing Escherichia coli.
@en
Cholesterol-enriched membrane ...... ing-effacing Escherichia coli.
@nl
prefLabel
Cholesterol-enriched membrane ...... ing-effacing Escherichia coli.
@en
Cholesterol-enriched membrane ...... ing-effacing Escherichia coli.
@nl
P2860
P1476
Cholesterol-enriched membrane ...... ing-effacing Escherichia coli.
@en
P2093
Jason D Riff
John W Callahan
P2860
P304
P356
10.1128/IAI.73.11.7113-7125.2005
P407
P577
2005-11-01T00:00:00Z