The "go or grow" potential of gliomas is linked to the neuropeptide processing enzyme carboxypeptidase E and mediated by metabolic stress.
about
Carboxypeptidase E: a negative regulator of the canonical Wnt signaling pathwayTumor Metabolism, the Ketogenic Diet and β-Hydroxybutyrate: Novel Approaches to Adjuvant Brain Tumor TherapyA genome-wide systematic analysis reveals different and predictive proliferation expression signatures of cancerous vs. non-cancerous cellsExpression Analysis of All Protease Genes Reveals Cathepsin K to Be Overexpressed in GlioblastomaCombined treatment with Metformin and 2-deoxy glucose induces detachment of viable MDA-MB-231 breast cancer cells in vitro.Dissecting carboxypeptidase E: properties, functions and pathophysiological roles in disease.The CREB-miR-9 negative feedback minicircuitry coordinates the migration and proliferation of glioma cellsThe molecular profiles of neural stem cell niche in the adult subventricular zone.carboxypeptidase E-ΔN, a neuroprotein transiently expressed during development protects embryonic neurons against glutamate neurotoxicity.Effects of anti-angiogenesis on glioblastoma growth and migration: model to clinical predictions.Carboxypeptidase E/NFα1: a new neurotrophic factor against oxidative stress-induced apoptotic cell death mediated by ERK and PI3-K/AKT pathways.Upregulation of CPE promotes cell proliferation and tumorigenicity in colorectal cancer.Glucose-based regulation of miR-451/AMPK signaling depends on the OCT1 transcription factor.Spatio-Temporal Dynamics of Hypoxia during Radiotherapy.ZEB1 Promotes Invasion in Human Fetal Neural Stem Cells and Hypoxic Glioma Neurospheres.Silencing of carboxypeptidase E inhibits cell proliferation, tumorigenicity, and metastasis of osteosarcoma cellsCarboxypeptidase E promotes cancer cell survival, but inhibits migration and invasionGlycolysis and the pentose phosphate pathway are differentially associated with the dichotomous regulation of glioblastoma cell migration versus proliferation.Targeting cancer stem-like cells in glioblastoma and colorectal cancer through metabolic pathways.Zinc and zinc-containing biomolecules in childhood brain tumors.Carboxypeptidase E is a prediction marker for tumor recurrence in early-stage hepatocellular carcinoma.Why one-size-fits-all vaso-modulatory interventions fail to control glioma invasion: in silico insightsEffects of soluble CPE on glioma cell migration are associated with mTOR activation and enhanced glucose flux.Density-Dependent Regulation of Glioma Cell Proliferation and Invasion Mediated by miR-9.Overexpression of CPE-ΔN predicts poor prognosis in colorectal cancer patients.The biology and mathematical modelling of glioma invasion: a review.Developmentally regulated signaling pathways in glioma invasion.Carboxypeptidase E (CPE) inhibits the secretion and activity of Wnt3a.Chronophin is a glial tumor modifier involved in the regulation of glioblastoma growth and invasiveness.
P2860
Q24294190-3D2B39B3-2E2F-4A1A-BC0A-7AE0C80F2836Q28069398-43DB5465-8A49-42C4-A614-BEEA1D108045Q28533665-0C1CE061-F85B-4CE5-ADC0-9F743176C82BQ29302290-AD163FE9-6261-460E-A05D-C4D8DAD5A619Q33687560-13176310-311C-4392-8D3B-5E087EA64310Q33697577-94564FA5-8432-44F3-859C-902AE4715CBCQ34490171-FCB2D2BD-C9CD-4B6A-A6B3-6CD21EAAC17DQ34500384-C292F3D3-CDE9-4773-B456-A1339935BB7CQ34575955-AA8AAD5D-CB6B-49AA-85F9-E5CB537584D5Q34703888-3C699917-DC0F-44F2-804E-5C5BC1C4347EQ34971532-4B994E92-92C4-4EA8-9AD7-F9F13A71817BQ34978670-E61EC0A9-6CF4-4364-AE1D-E896032A4E84Q35607018-AE78B0C0-ADF7-4A0E-8CB9-BC045EFEAF20Q35745894-DFE6BE69-19D5-4B5F-AD74-8FBF6200031DQ35752332-07E05D01-54CE-41D5-B26A-191CFD005F30Q36907950-41889CE7-056E-45FE-ACF0-38DF5F30F459Q37302972-1310EED9-480C-4024-8B40-8CF10212F3D7Q38790923-130AD528-A43B-4A1D-B1B3-08733EA7ABB9Q38889490-9BF123A0-BB1F-404C-89F2-07CB808324E8Q38957110-097813E6-8E25-4B39-A07F-AE2349FC3B88Q40068989-B0AE4207-AB56-4FA4-BF18-6DE17E322F81Q40439684-4091BC7E-05A7-4F0C-AA10-FEFC71A40090Q42004283-07FDBF18-E9E1-4D62-B99E-EB9521B0F173Q42328087-C90C2E30-0197-48CC-9755-ECE3420C40DBQ44594513-8CE04309-285C-4317-B091-AC1EBC6C0D6AQ47124238-B4E44182-0F4A-4D09-946A-ED7F09DE9590Q48148598-5C6B178B-5E19-4B7A-A21B-0E6B62EC6440Q50501066-7D3042D4-E594-4D9B-92FA-DBC6EF2F6907Q52147015-CFFBB8F3-A9C8-4C26-8AFE-2F400728CA97
P2860
The "go or grow" potential of gliomas is linked to the neuropeptide processing enzyme carboxypeptidase E and mediated by metabolic stress.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
The "go or grow" potential of ...... mediated by metabolic stress.
@en
The "go or grow" potential of ...... mediated by metabolic stress.
@nl
type
label
The "go or grow" potential of ...... mediated by metabolic stress.
@en
The "go or grow" potential of ...... mediated by metabolic stress.
@nl
prefLabel
The "go or grow" potential of ...... mediated by metabolic stress.
@en
The "go or grow" potential of ...... mediated by metabolic stress.
@nl
P2093
P2860
P1476
The "go or grow" potential of ...... mediated by metabolic stress.
@en
P2093
Cornelia Zachskorn
Elisabeth Höring
Elke von Hattingen
Hans-Jörg Bühring
Janina Seznec
Patrick Nikolaus Harter
Shohag Bhattacharyya
Ulrike Naumann
P2860
P2888
P356
10.1007/S00401-011-0940-X
P577
2012-01-15T00:00:00Z