Establishment of a highly migratory subclone reveals that CD133 contributes to migration and invasion through epithelial-mesenchymal transition in pancreatic cancer.
about
Pancreatic cancer stem cells: emerging target for designing novel therapyLow concentrations of metformin selectively inhibit CD133⁺ cell proliferation in pancreatic cancer and have anticancer actionPolycomb complex protein BMI-1 promotes invasion and metastasis of pancreatic cancer stem cells by activating PI3K/AKT signaling, an ex vivo, in vitro, and in vivo study.Cancer Stem Cells, EMT, and Developmental Pathway Activation in Pancreatic TumorsAnalysis of different components in the peritumoral tissue microenvironment of colorectal cancer: A potential prospect in tumorigenesis.Pancreatic cancer stem cells: regulatory networks in the tumor microenvironment and targeted therapy.CD133 as a biomarker for putative cancer stem cells in solid tumours: limitations, problems and challenges.Epithelial mesenchymal transition in drug resistance and metastasis of lung cancer.CD133 facilitates epithelial-mesenchymal transition through interaction with the ERK pathway in pancreatic cancer metastasis.CD133 Modulate HIF-1α Expression under Hypoxia in EMT Phenotype Pancreatic Cancer Stem-Like Cells.Efficient elimination of pancreatic cancer stem cells by hedgehog/GLI inhibitor GANT61 in combination with mTOR inhibition.miR-30 family promotes migratory and invasive abilities in CD133(+) pancreatic cancer stem-like cells.Slug contributes to gemcitabine resistance through epithelial-mesenchymal transition in CD133(+) pancreatic cancer cells.Neutralizing murine TGFβR2 promotes a differentiated tumor cell phenotype and inhibits pancreatic cancer metastasis.mTOR plays critical roles in pancreatic cancer stem cells through specific and stemness-related functionsThalidomide inhibits proliferation and epithelial-mesenchymal transition by modulating CD133 expression in pancreatic cancer cells.The role of cancer-associated fibroblast MRC-5 in pancreatic cancer.
P2860
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P2860
Establishment of a highly migratory subclone reveals that CD133 contributes to migration and invasion through epithelial-mesenchymal transition in pancreatic cancer.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
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2011年學術文章
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name
Establishment of a highly migr ...... ansition in pancreatic cancer.
@en
Establishment of a highly migr ...... ansition in pancreatic cancer.
@nl
type
label
Establishment of a highly migr ...... ansition in pancreatic cancer.
@en
Establishment of a highly migr ...... ansition in pancreatic cancer.
@nl
prefLabel
Establishment of a highly migr ...... ansition in pancreatic cancer.
@en
Establishment of a highly migr ...... ansition in pancreatic cancer.
@nl
P2093
P2860
P1433
P1476
Establishment of a highly migr ...... ransition in pancreatic cancer
@en
P2093
Makoto Yoshimitsu
Qiang Ding
Shoji Natsugoe
Shyuichiro Matsubara
Sonshin Takao
Taisaku Kuwahata
Tomomi Hayashi
Toru Obara
Yumi Miyazaki
P2860
P2888
P356
10.1007/S13577-011-0037-9
P50
P577
2011-11-23T00:00:00Z