All-trans-retinoic acid inhibits Jun N-terminal kinase by increasing dual-specificity phosphatase activity.
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Retinoic acid receptors inhibit AP1 activation by regulating extracellular signal-regulated kinase and CBP recruitment to an AP1-responsive promoter.Induced cytoskeletal changes in bovine pulmonary artery endothelial cells by resveratrol and the accompanying modified responses to arterial shear stressCharacterization of retinoic acid receptor-deficient keratinocytesMitogen-activated protein kinases and mitogen-activated protein kinase phosphatases mediate the inhibitory effects of all-trans retinoic acid on the hypertrophic growth of cardiomyocytesInterleukin-1 beta-mediated suppression of RXR:RAR transactivation of the Ntcp promoter is JNK-dependentTranscriptional induction of mitogen-activated protein kinase phosphatase 1 by retinoids. Selective roles of nuclear receptors and contribution to the antiapoptotic effectGlucocorticoids antagonize AP-1 by inhibiting the Activation/phosphorylation of JNK without affecting its subcellular distribution.c-Jun N-terminal kinase contributes to aberrant retinoid signaling in lung cancer cells by phosphorylating and inducing proteasomal degradation of retinoic acid receptor alphaTranscriptional and posttranslational regulation of insulin-like growth factor binding protein-3 by Akt3.Cross-talk between glucocorticoid receptor and AP-1.Orchestration of multiple arrays of signal cross-talk and combinatorial interactions for maturation and cell death: another vision of t(15;17) preleukemic blast and APL-cell maturation.All-Trans Retinoic Acid Induces TGF-β2 in Intestinal Epithelial Cells via RhoA- and p38α MAPK-Mediated Activation of the Transcription Factor ATF2.Animal models in carotenoids research and lung cancer prevention.Combined α-tocopherol and ascorbic acid protects against smoke-induced lung squamous metaplasia in ferrets.Signalling pathways activated by all-trans-retinoic acid in acute promyelocytic leukemia cells.The role of retinoic acid in tolerance and immunity.Molecular pathways: current role and future directions of the retinoic acid pathway in cancer prevention and treatmentRetinoic acid-elicited RARα/RXRα signaling attenuates Aβ production by directly inhibiting γ-secretase-mediated cleavage of amyloid precursor proteinRetinoic acid treatment of human leiomyoma cells transformed the cell phenotype to one strongly resembling myometrial cells.Glucocorticoid receptor-JNK interaction mediates inhibition of the JNK pathway by glucocorticoids.Retinoic acid induced repression of AP-1 activity is mediated by protein phosphatase 2A in ovarian carcinoma cells.Regulation of ERK1 gene expression by coactivator proteins.Retinoids arrest breast cancer cell proliferation: retinoic acid selectively reduces the duration of receptor tyrosine kinase signaling.Retinoic acid inhibits interleukin-4-induced eotaxin production in a human bronchial epithelial cell line.Gene expression profiling during all-trans retinoic acid-induced cell differentiation of acute promyelocytic leukemia cellsAP-1 transrepressing retinoic acid does not deplete coactivators or AP-1 monomers but may target specific Jun or Fos containing dimers.Compartment-specific regulation of extracellular signal-regulated kinase (ERK) and c-Jun N-terminal kinase (JNK) mitogen-activated protein kinases (MAPKs) by ERK-dependent and non-ERK-dependent inductions of MAPK phosphatase (MKP)-3 and MKP-1 in difActivation of Rac1 and the p38 mitogen-activated protein kinase pathway in response to all-trans-retinoic acid.12-O-tetradecanoylphorbol-13-acetate (TPA)-induced c-Jun N-terminal kinase (JNK) phosphatase renders immortalized or transformed epithelial cells refractory to TPA-inducible JNK activity.Kinase-dependent, retinoic acid receptor-independent up-regulation of cyclooxygenase-2 by all-trans retinoic acid in human mesangial cells.Retinoic acid activation of the ERK pathway is required for embryonic stem cell commitment into the adipocyte lineage.Insulin-like growth factor binding protein-6 inhibits the growth of human bronchial epithelial cells and increases in abundance with all-trans-retinoic acid treatment.Vitamin A inhibits pancreatic stellate cell activation: implications for treatment of pancreatic fibrosisSelective roles of retinoic acid receptor and retinoid x receptor in the suppression of apoptosis by all-trans-retinoic acid.Retinoic acid inhibits hepatic Jun N-terminal kinase-dependent signaling pathway in ethanol-fed rats.Retinoic acid decreases fetal lung mesenchymal cell proliferation in vivo and in vitro.Ectopic expression of nonliganded retinoic acid receptor beta abrogates AP-1 activity by selective degradation of c-Jun in cervical carcinoma cells.Large-scale reprogramming of cranial neural crest gene expression by retinoic acid exposure.Methyl viologen induces neural differentiation on murine P19 cells.In vitro culture significantly alters gene expression profiles and reduces differences between myometrial and fibroid smooth muscle cells.
P2860
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P2860
All-trans-retinoic acid inhibits Jun N-terminal kinase by increasing dual-specificity phosphatase activity.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh
1999年學術文章
@zh-hant
name
All-trans-retinoic acid inhibi ...... cificity phosphatase activity.
@en
All-trans-retinoic acid inhibi ...... cificity phosphatase activity.
@nl
type
label
All-trans-retinoic acid inhibi ...... cificity phosphatase activity.
@en
All-trans-retinoic acid inhibi ...... cificity phosphatase activity.
@nl
prefLabel
All-trans-retinoic acid inhibi ...... cificity phosphatase activity.
@en
All-trans-retinoic acid inhibi ...... cificity phosphatase activity.
@nl
P2093
P2860
P356
P1476
All-trans-retinoic acid inhibi ...... cificity phosphatase activity.
@en
P2093
P2860
P304
P356
10.1128/MCB.19.3.1973
P407
P577
1999-03-01T00:00:00Z