Mitochondrial dynamics following global cerebral ischemia. - Wikidata - awgldk - TriplyDB

Mitochondrial dynamics following global cerebral ischemia.

Mitochondrial dynamics following global cerebral ischemia.

http://www.wikidata.org/entity/Q39447297

about

Cannabidiol attenuates OGD/R-induced damage by enhancing mitochondrial bioenergetics and modulating glucose metabolism via pentose-phosphate pathway in hippocampal neurons.Fusion or Fission: The Destiny of Mitochondria In Traumatic Brain Injury of Different Severities.Mitochondrial Quality Control and Disease: Insights into Ischemia-Reperfusion Injury.Inhibition of mitochondrial fission as a molecular target for cardioprotection: critical importance of the timing of treatment.Lycium Barbarum Polysaccharides Decrease Hyperglycemia-Aggravated Ischemic Brain Injury through Maintaining Mitochondrial Fission and Fusion Balance.Significance of Mitochondrial Protein Post-translational Modifications in Pathophysiology of Brain Injury.Inhibitory modulation of cytochrome c oxidase activity with specific near-infrared light wavelengths attenuates brain ischemia/reperfusion injury.Non-toxic engineered carbon nanodiamond concentrations induce oxidative/nitrosative stress, imbalance of energy metabolism, and mitochondrial dysfunction in microglial and alveolar basal epithelial cells.

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Mitochondrial dynamics following global cerebral ischemia.

http://www.wikidata.org/entity/Q39447297

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh

2016年學術文章

@zh-hant

name

Mitochondrial dynamics following global cerebral ischemia.

@en

Mitochondrial dynamics following global cerebral ischemia.

@nl

type

label

Mitochondrial dynamics following global cerebral ischemia.

@en

Mitochondrial dynamics following global cerebral ischemia.

@nl

prefLabel

Mitochondrial dynamics following global cerebral ischemia.

@en

Mitochondrial dynamics following global cerebral ischemia.

@nl

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J.MCN.2016.08.010

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Molecular and Cellular Neuroscience

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Mitochondrial dynamics following global cerebral ischemia

@en

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Bradley Lepore

http://www.w3.org/2001/XMLSchema#string

Christian A Reynolds

http://www.w3.org/2001/XMLSchema#string

Joseph M Wider

http://www.w3.org/2001/XMLSchema#string

Karin Przyklenk

http://www.w3.org/2001/XMLSchema#string

Lesley Calo

http://www.w3.org/2001/XMLSchema#string

Melissa J Bukowski

http://www.w3.org/2001/XMLSchema#string

Michelle Jones

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Renee Tousignant

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Loss of OPA1 perturbates the mitochondrial inner membrane structure and integrity, leading to cytochrome c release and apoptosis

The dynamin-related GTPase, Mgm1p, is an intermembrane space protein required for maintenance of fusion competent mitochondria.

OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion

The OPA1-dependent mitochondrial cristae remodeling pathway controls atrophic, apoptotic, and ischemic tissue damage

Blue native PAGE

Neuronal hypoxia disrupts mitochondrial fusion

Cytochrome C is tyrosine 97 phosphorylated by neuroprotective insulin treatment

Release of mitochondrial Opa1 following oxidative stress in HT22 cells

Mitochondrial dysfunction is associated with a pro-apoptotic cellular environment in senescent cardiac muscle

Cerebral ischemia-reperfusion-induced autophagy protects against neuronal injury by mitochondrial clearance

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68-75

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scholarly article

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10.1016/J.MCN.2016.08.010

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76

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Thomas H. Sanderson

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2016-08-25T00:00:00Z

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27567688

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cerebral infarction

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5056829

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