SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells. - Wikidata - awgldk - TriplyDB

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

http://www.wikidata.org/entity/Q39454272

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Dynamic aberrant NF-κB spurs tumorigenesis: a new model encompassing the microenvironment Cell fusion induced by ERVWE1 or measles virus causes cellular senescence Role of the beta catenin destruction complex in mediating chemotherapy-induced senescence-associated secretory phenotype Metformin-induced metabolic reprogramming of chemoresistant ALDHbright breast cancer cells Senescence and apoptosis: dueling or complementary cell fates?Zoledronic acid overcomes chemoresistance and immunosuppression of malignant mesothelioma Cisplatin-resistant cells in malignant pleural mesothelioma cell lines show ALDH(high)CD44(+) phenotype and sphere-forming capacity.Antagonizing Effects of Aspartic Acid against Ultraviolet A-Induced Downregulation of the Stemness of Human Adipose Tissue-Derived Mesenchymal Stem Cells Chemoresistance to doxorubicin induces epithelial-mesenchymal transition via upregulation of transforming growth factor β signaling in HCT116 colon cancer cells.The requirement for freshly isolated human colorectal cancer (CRC) cells in isolating CRC stem cells.Expression of the Stem Cell Factor Nestin in Malignant Pleural Mesothelioma Is Associated with Poor Prognosis A STAT3-NFkB/DDIT3/CEBPβ axis modulates ALDH1A3 expression in chemoresistant cell subpopulations.Prolonged pemetrexed pretreatment augments persistence of cisplatin-induced DNA damage and eliminates resistant lung cancer stem-like cells associated with EMT The p53 codon 72 Pro/Pro genotype identifies poor-prognosis neuroblastoma patients: correlation with reduced apoptosis and enhanced senescence by the p53-72P isoform.Cynara scolymus affects malignant pleural mesothelioma by promoting apoptosis and restraining invasion.Protumorigenic effects of mir-145 loss in malignant pleural mesothelioma.Cisplatin-induced mesenchymal stromal cells-mediated mechanism contributing to decreased antitumor effect in breast cancer cells.MLL1 is essential for the senescence-associated secretory phenotype ATM kinase enables the functional axis of YAP, PML and p53 to ameliorate loss of Werner protein-mediated oncogenic senescence.A senescence secretory switch mediated by PI3K/AKT/mTOR activation controls chemoprotective endothelial secretory responses.Redox control of the senescence regulator interleukin-1α and the secretory phenotype.NF-κB-HOTAIR axis links DNA damage response, chemoresistance and cellular senescence in ovarian cancer.Secretome from senescent melanoma engages the STAT3 pathway to favor reprogramming of naive melanoma towards a tumor-initiating cell phenotype.Inhibition of the colony-stimulating-factor-1 receptor affects the resistance of lung cancer cells to cisplatin Exemestane blocks mesothelioma growth through downregulation of cAMP, pCREB and CD44 implicating new treatment option in patients affected by this disease Immune regulation of therapy-resistant niches: emerging targets for improving anticancer drug responses.EZH2 inhibition re-sensitizes multidrug resistant B-cell lymphomas to etoposide mediated apoptosis.Premature aging/senescence in cancer cells facing therapy: good or bad?Cyclin A1 expression and paclitaxel resistance in human ovarian cancer cells.Blocking the epithelial-to-mesenchymal transition pathway abrogates resistance to anti-folate chemotherapy in lung cancer.Cellular lifespan and senescence: a complex balance between multiple cellular pathways.The Regulation of Cellular Functions by the p53 Protein: Cellular Senescence.Melanocyte-protective effect of afzelin is mediated by the Nrf2-ARE signalling pathway via GSK-3β inactivation.Schedule-dependent increased efficiency of pemetrexed-ionizing radiation combination therapy elicits a differential DNA damage response in lung cancer cells.4th international conference on tumor progression and therapeutic resistance: meeting report.Stem cell senescence drives age-attenuated induction of pituitary tumours in mouse models of paediatric craniopharyngioma.DNA damage response-initiated cytokine secretion in bone marrow stromal cells promotes chemoresistance of myeloma cells.p38 MAPK inhibits nonsense-mediated RNA decay in response to persistent DNA damage in noncycling cells.HSP90 inhibition alters the chemotherapy-driven rearrangement of the oncogenic secretome.Live-Cell Mesothelioma Biobank to Explore Mechanisms of Tumor Progression.

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SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

http://www.wikidata.org/entity/Q39454272

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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2011年學術文章

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name

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

@en

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

@nl

type

label

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

@en

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

@nl

prefLabel

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

@en

SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

@nl

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SASP mediates chemoresistance and tumor-initiating-activity of mesothelioma cells.

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A Cambria

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A Diamantini

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C Canino

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P2860

A microRNA component of the p53 tumour suppressor network

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

The senescence-associated secretory phenotype: the dark side of tumor suppression

The effects and mechanisms of blockage of STAT3 signaling pathway on IL-6 inducing EMT in human pancreatic cancer cells in vitro.

Aldehyde dehydrogenase 1-positive cancer stem cells mediate metastasis and poor clinical outcome in inflammatory breast cancer.

STAT family of transcription factors in cytokine-mediated biological responses.

Inflammation precedes the development of human malignant mesotheliomas in a SCID mouse xenograft model

SASP reflects senescence.

Breast cancer stem cells are regulated by mesenchymal stem cells through cytokine networks

Expression of a functional VEGFR-1 in tumor cells is a major determinant of anti-PlGF antibodies efficacy

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3148-3163

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10.1038/ONC.2011.485

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26

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22020330

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