Longitudinal β-Amyloid Deposition and Hippocampal Volume in Preclinical Alzheimer Disease and Suspected Non-Alzheimer Disease Pathophysiology.

Longitudinal β-Amyloid Deposition and Hippocampal Volume in Preclinical Alzheimer Disease and Suspected Non-Alzheimer Disease Pathophysiology.

Item
http://www.wikidata.org/entity/Q39466317
The Function of the Mitochondrial Calcium Uniporter in Neurodegenerative Disorders.Hippocampal and Clinical Trajectories of Mild Cognitive Impairment with Suspected Non-Alzheimer's Disease Pathology.Age-specific and sex-specific prevalence of cerebral β-amyloidosis, tauopathy, and neurodegeneration in cognitively unimpaired individuals aged 50-95 years: a cross-sectional study.Alzheimer Disease Signature Neurodegeneration and APOE Genotype in Mild Cognitive Impairment With Suspected Non-Alzheimer Disease Pathophysiology.Hippocampal shape alterations are associated with regional Aβ load in cognitively normal elderly individuals.Progressive medial temporal lobe atrophy during preclinical Alzheimer's disease.Regionally specific changes in the hippocampal circuitry accompany progression of cerebrospinal fluid biomarkers in preclinical Alzheimer's disease.Cerebrospinal fluid and blood biomarkers for neurodegenerative dementias: An update of the Consensus of the Task Force on Biological Markers in Psychiatry of the World Federation of Societies of Biological Psychiatry.The Effects of Cortical Hypometabolism and Hippocampal Atrophy on Clinical Trajectories in Mild Cognitive Impairment with Suspected Non-Alzheimer's Pathology: A Brief Report.Amyloid β Deposition and Suspected Non-Alzheimer Pathophysiology and Cognitive Decline Patterns for 12 Years in Oldest Old Participants Without Dementia.Defining SNAP by cross-sectional and longitudinal definitions of neurodegeneration.Age-accelerated cognitive decline in asymptomatic adults with CSF β-amyloid.Association of Neuropathological Markers in the Parietal Cortex With Antemortem Cognitive Function in Persons With Mild Cognitive Impairment and Alzheimer Disease.NIA-AA Research Framework: Toward a biological definition of Alzheimer's disease.Cross-sectional and longitudinal atrophy is preferentially associated with tau rather than amyloid β positron emission tomography pathology.
P2860

Longitudinal β-Amyloid Deposition and Hippocampal Volume in Preclinical Alzheimer Disease and Suspected Non-Alzheimer Disease Pathophysiology.

Item
http://www.wikidata.org/entity/Q39466317
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Longitudinal β-Amyloid Deposit ...... eimer Disease Pathophysiology.
@en
Longitudinal β-Amyloid Deposit ...... eimer Disease Pathophysiology.
@nl
type
label
Longitudinal β-Amyloid Deposit ...... eimer Disease Pathophysiology.
@en
Longitudinal β-Amyloid Deposit ...... eimer Disease Pathophysiology.
@nl
prefLabel
Longitudinal β-Amyloid Deposit ...... eimer Disease Pathophysiology.
@en
Longitudinal β-Amyloid Deposit ...... eimer Disease Pathophysiology.
@nl
P1433
P1476
P2093
P2860
P304
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P356
P407
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P478
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P698
P921
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P1433
P1476
Longitudinal β-Amyloid Deposit ...... eimer Disease Pathophysiology.
@en
P2093
P2860
P304
P31
P356
10.1001/JAMANEUROL.2016.2642
P407
P433
P478
P50
P577
P698
P921
P932