B cell-deficient mice are highly resistant to Leishmania donovani infection, but develop neutrophil-mediated tissue pathology.
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In vivo involvement of polymorphonuclear neutrophils in Leishmania infantum infectionThe influence of early neutrophil-Leishmania interactions on the host immune response to infectionB cell: T cell interactions occur within hepatic granulomas during experimental visceral leishmaniasisSingle dose novel Salmonella vaccine enhances resistance against visceralizing L. major and L. donovani infection in susceptible BALB/c miceLessons from other diseases: granulomatous inflammation in leishmaniasisAbortive T follicular helper development is associated with a defective humoral response in Leishmania infantum-infected macaquesRegulation of immunity during visceral Leishmania infectionImipramine is an orally active drug against both antimony sensitive and resistant Leishmania donovani clinical isolates in experimental infectionCritical roles for LIGHT and its receptors in generating T cell-mediated immunity during Leishmania donovani infectionImmune regulation during chronic visceral leishmaniasis.Clinical and experimental advances in treatment of visceral leishmaniasis.Kinetic analysis of ex vivo human blood infection by Leishmania.B cells delay neutrophil migration toward the site of stimulus: tardiness critical for effective bacillus Calmette-Guérin vaccination against tuberculosis infection in mice.Immune evasive mechanisms contributing to persistent Leishmania donovani infection.Murine visceral leishmaniasis: IgM and polyclonal B-cell activation lead to disease exacerbation.Invariant NKT cells drive hepatic cytokinic microenvironment favoring efficient granuloma formation and early control of Leishmania donovani infection.Leishmania pifanoi pathogenesis: selective lack of a local cutaneous response in the absence of circulating antibody.B-cell-deficient mice show an exacerbated inflammatory response in a model of Chlamydophila abortus infection.Does the Leishmania major paradigm of pathogenesis and protection hold for New World cutaneous leishmaniases or the visceral disease?Genetic control of immune cell types in fungal disease.Leukocytes infiltrate the skin and draining lymph nodes in response to the protozoan Leishmania infantum chagasi.Balancing immunity and pathology in visceral leishmaniasis.Immunobiology of visceral leishmaniasis.Myeloid cell function in MRP-14 (S100A9) null mice.To B or not to B: B cells and the Th2-type immune response to helminths.Compartment-specific remodeling of splenic micro-architecture during experimental visceral leishmaniasis.Leishmania infantum amastigotes trigger a subpopulation of human B cells with an immunoregulatory phenotype.Differential Regulation of the Immune Response in the Spleen and Liver of Mice Infected with Leishmania donovani.Dependence of intestinal granuloma formation on unique myeloid DC-like cells.In vitro activity and in vivo efficacy of a combination therapy of diminazene and chloroquine against murine visceral leishmaniasisImmunity and immunosuppression in experimental visceral leishmaniasis.Murine model of chronic L. (Viannia) panamensis infection: role of IL-13 in disease.Central role for B lymphocytes and CD4+ T cells in immunity to infection by the attaching and effacing pathogen Citrobacter rodentium.Infiltration of neutrophils is required for acquisition of metastatic phenotype of benign murine fibrosarcoma cells: implication of inflammation-associated carcinogenesis and tumor progression.The immunopathology of experimental visceral leishmaniasis.New concepts in antibody-mediated immunity.T helper1/t helper2 cells and resistance/susceptibility to leishmania infection: is this paradigm still relevant?Sequential chemoimmunotherapy of experimental visceral leishmaniasis using a single low dose of liposomal amphotericin B and a novel DNA vaccine candidate.Prognostic Impact of Immune Microenvironment in Lung Squamous Cell Carcinoma: Tumor-Infiltrating CD10+ Neutrophil/CD20+ Lymphocyte Ratio as an Independent Prognostic FactorPhenotypic, morphological, and functional heterogeneity of splenic immature myeloid cells in the host response to tularemia.
P2860
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P2860
B cell-deficient mice are highly resistant to Leishmania donovani infection, but develop neutrophil-mediated tissue pathology.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
B cell-deficient mice are high ...... hil-mediated tissue pathology.
@en
B cell-deficient mice are high ...... hil-mediated tissue pathology.
@nl
type
label
B cell-deficient mice are high ...... hil-mediated tissue pathology.
@en
B cell-deficient mice are high ...... hil-mediated tissue pathology.
@nl
prefLabel
B cell-deficient mice are high ...... hil-mediated tissue pathology.
@en
B cell-deficient mice are high ...... hil-mediated tissue pathology.
@nl
P2093
P1476
B cell-deficient mice are high ...... hil-mediated tissue pathology.
@en
P2093
P304
P356
10.4049/JIMMUNOL.164.7.3681
P407
P577
2000-04-01T00:00:00Z