Type-III interferon, not type-I, is the predominant interferon induced by respiratory viruses in nasal epithelial cells. - Wikidata - awgldk - TriplyDB

Type-III interferon, not type-I, is the predominant interferon induced by respiratory viruses in nasal epithelial cells.

Type-III interferon, not type-I, is the predominant interferon induced by respiratory viruses in nasal epithelial cells.

http://www.wikidata.org/entity/Q39495155

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The host immune response in respiratory virus infection: balancing virus clearance and immunopathology.Interferon-λ: Immune Functions at Barrier Surfaces and Beyond Induction and function of type I and III interferon in response to viral infection Leukocyte-derived IFN-α/β and epithelial IFN-λ constitute a compartmentalized mucosal defense system that restricts enteric virus infections IFN-λ: A New Inducer of Local Immunity against Cancer and Infections Respiratory syncytial virus can infect basal cells and alter human airway epithelial differentiation In vitro modeling of respiratory syncytial virus infection of pediatric bronchial epithelium, the primary target of infection in vivo Mast cells are permissive for rhinovirus replication: potential implications for asthma exacerbations Reactive oxygen species induce antiviral innate immune response through IFN-λ regulation in human nasal epithelial cells.IFN-λ exerts opposing effects on T cell responses depending on the chronicity of the virus infection.Bovine type III interferon significantly delays and reduces the severity of foot-and-mouth disease in cattle.Type III interferon attenuates a vesicular stomatitis virus-based vaccine vector.Human kinome analysis reveals novel kinases contributing to virus infection and retinoic-acid inducible gene I-induced type I and type III IFN gene expression.Interferon response and respiratory virus control are preserved in bronchial epithelial cells in asthma.Curcumin prevents replication of respiratory syncytial virus and the epithelial responses to it in human nasal epithelial cells Respiratory syncytial virus induced type I IFN production by pDC is regulated by RSV-infected airway epithelial cells, RSV-exposed monocytes and virus specific antibodies Suppression of interferon lambda signaling by SOCS-1 results in their excessive production during influenza virus infection Human parainfluenza virus type 3 (HPIV3) induces production of IFNγ and RANTES in human nasal epithelial cells (HNECs).Efficacy of IFN-λ1 to protect human airway epithelial cells against human rhinovirus 1B infection.The unique regulation and functions of type III interferons in antiviral immunity.Cytokine Profiles in Human Metapneumovirus Infected Children: Identification of Genes Involved in the Antiviral Response and Pathogenesis.The role of airway epithelial cells and innate immune cells in chronic respiratory disease.Parainfluenza Virus 3 Blocks Antiviral Mediators Downstream of the Interferon Lambda Receptor by Modulating Stat1 Phosphorylation α1-Antitrypsin reduces rhinovirus infection in primary human airway epithelial cells exposed to cigarette smoke.Impaired secretion of interferons by dendritic cells from aged subjects to influenza : role of histone modifications.Respiratory syncytial virus--a comprehensive review.Host-Viral Interactions: Role of Pattern Recognition Receptors (PRRs) in Human Pneumovirus Infections The Central Conserved Region (CCR) of Respiratory Syncytial Virus (RSV) G Protein Modulates Host miRNA Expression and Alters the Cellular Response to Infection.Sustained activation of interferon regulatory factor 3 during infection by paramyxoviruses requires MDA5.Contribution of type III interferons to antiviral immunity: location, location, location.Clarithromycin suppresses human respiratory syncytial virus infection-induced Streptococcus pneumoniae adhesion and cytokine production in a pulmonary epithelial cell line.Impaired virus replication and decreased innate immune responses to viral infections in nasal epithelial cells from patients with allergic rhinitis.RIG-I Activation Protects and Rescues from Lethal Influenza Virus Infection and Bacterial Superinfection.Induction and Antagonism of Antiviral Responses in Respiratory Syncytial Virus-Infected Pediatric Airway Epithelium.The innate immune response to RSV: Advances in our understanding of critical viral and host factors.Human Rhinovirus-induced Proinflammatory Cytokine and Interferon-β Responses in Nasal Epithelial Cells From Chronic Rhinosinusitis Patients Respiratory syncytial virus G protein CX3C motif impairs human airway epithelial and immune cell responses.Marked induction of matrix metalloproteinase-10 by respiratory syncytial virus infection in human nasal epithelial cells.Relative respiratory syncytial virus cytopathogenesis in upper and lower respiratory tract epithelium.Interferon-λs: Front-Line Guardians of Immunity and Homeostasis in the Respiratory Tract.

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P2860

Type-III interferon, not type-I, is the predominant interferon induced by respiratory viruses in nasal epithelial cells.

http://www.wikidata.org/entity/Q39495155

description

2011 nî lūn-bûn

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2011年の論文

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2011年學術文章

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2011年學術文章

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name

Type-III interferon, not type- ...... ses in nasal epithelial cells.

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Type-III interferon, not type- ...... ses in nasal epithelial cells.

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type

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Type-III interferon, not type- ...... ses in nasal epithelial cells.

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Type-III interferon, not type- ...... ses in nasal epithelial cells.

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prefLabel

Type-III interferon, not type- ...... ses in nasal epithelial cells.

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Type-III interferon, not type- ...... ses in nasal epithelial cells.

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J.VIRUSRES.2011.07.011

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Type-III interferon, not type- ...... ses in nasal epithelial cells.

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Hiroyuki Tsutsumi

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Nobuhiro Fujii

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Norimasa Sawada

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Shin-Ichi Yokota

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Tadaatsu Imaizumi

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Takashi Kojima

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Tamaki Okabayashi

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Tetsuo Himi

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Tomoyuki Masaki

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360-366

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10.1016/J.VIRUSRES.2011.07.011

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1-2

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21816185

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