lon incompatibility associated with mutations causing SOS induction: null uvrD alleles induce an SOS response in Escherichia coli.
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Answering the Call: Coping with DNA Damage at the Most Inopportune TimeCharacterization of the helicase activity and substrate specificity of Mycobacterium tuberculosis UvrDAnalysis of global gene expression and double-strand-break formation in DNA adenine methyltransferase- and mismatch repair-deficient Escherichia coliEvidence for an arginine exporter encoded by yggA (argO) that is regulated by the LysR-type transcriptional regulator ArgP in Escherichia coliIS186 insertion at a hot spot in the lon promoter as a basis for lon protease deficiency of Escherichia coli B: identification of a consensus target sequence for IS186 transposition.Pathways of resistance to thymineless death in Escherichia coli and the function of UvrD.UvrD controls the access of recombination proteins to blocked replication forksUvrD303, a hyperhelicase mutant that antagonizes RecA-dependent SOS expression by a mechanism that depends on its C terminus.Tolerance of Escherichia coli to fluoroquinolone antibiotics depends on specific components of the SOS response pathway.Specificity in suppression of SOS expression by recA4162 and uvrD303.Inactivation of Cell Division Protein FtsZ by SulA Makes Lon Indispensable for the Viability of a ppGpp0 Strain of Escherichia coli.Stress-based identification and classification of antibacterial agents: second-generation Escherichia coli reporter strains and optimization of detectionThe Escherichia coli dnaN159 mutant displays altered DNA polymerase usage and chronic SOS induction.Isolation of SOS constitutive mutants of Escherichia coli.The protease Lon and the RNA-binding protein Hfq reduce silencing of the Escherichia coli bgl operon by H-NS.Role for tandem duplication and lon protease in AcrAB-TolC- dependent multiple antibiotic resistance (Mar) in an Escherichia coli mutant without mutations in marRAB or acrRAB.A dnaC mutation in Escherichia coli that affects copy number of ColE1-like plasmids and the PriA-PriB (but not Rep-PriC) pathway of chromosomal replication restart.Genetic evidence for the requirement of RecA loading activity in SOS induction after UV irradiation in Escherichia coli.E. coli BW535, a triple mutant for the DNA repair genes xth, nth, and nfo, chronically induces the SOS response.XthA (Exonuclease III) regulates loading of RecA onto DNA substrates in log phase Escherichia coli cells.
P2860
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P2860
lon incompatibility associated with mutations causing SOS induction: null uvrD alleles induce an SOS response in Escherichia coli.
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2000 nî lūn-bûn
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lon incompatibility associated ...... response in Escherichia coli.
@en
lon incompatibility associated ...... response in Escherichia coli.
@nl
type
label
lon incompatibility associated ...... response in Escherichia coli.
@en
lon incompatibility associated ...... response in Escherichia coli.
@nl
prefLabel
lon incompatibility associated ...... response in Escherichia coli.
@en
lon incompatibility associated ...... response in Escherichia coli.
@nl
P2093
P2860
P1476
lon incompatibility associated ...... response in Escherichia coli.
@en
P2093
Gowrishankar J
P2860
P304
P356
10.1128/JB.182.11.3151-3157.2000
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P577
2000-06-01T00:00:00Z