Tissue-resident macrophages protect the liver from ischemia reperfusion injury via a heme oxygenase-1-dependent mechanism. - Wikidata - awgldk - TriplyDB

Tissue-resident macrophages protect the liver from ischemia reperfusion injury via a heme oxygenase-1-dependent mechanism.

Tissue-resident macrophages protect the liver from ischemia reperfusion injury via a heme oxygenase-1-dependent mechanism.

http://www.wikidata.org/entity/Q39501283

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Beyond Preconditioning: Postconditioning as an Alternative Technique in the Prevention of Liver Ischemia-Reperfusion Injury Dual protective role of HO-1 in transplanted liver grafts: a review of experimental and clinical studies Beneficial autoimmunity at body surfaces - immune surveillance and rapid type 2 immunity regulate tissue homeostasis and cancer Gene expression study of monocytes/macrophages during early foreign body reaction and identification of potential precursors of myofibroblasts Classical and alternative macrophage activation in the lung following ozone-induced oxidative stress Adoptive transfer of heme oxygenase-1 (HO-1)-modified macrophages rescues the nuclear factor erythroid 2-related factor (Nrf2) antiinflammatory phenotype in liver ischemia/reperfusion injury Heat shock protein-mediated protection against Cisplatin-induced hair cell death.Microglia - the brain's busy bees.Exuberant sprouting of sensory and sympathetic nerve fibers in nonhealed bone fractures and the generation and maintenance of chronic skeletal pain.Tip-DC development during parasitic infection is regulated by IL-10 and requires CCL2/CCR2, IFN-gamma and MyD88 signaling.Specific expression of heme oxygenase-1 by myeloid cells modulates renal ischemia-reperfusion injury.Plasmacytoid dendritic cell-derived IFN-α promotes murine liver ischemia/reperfusion injury by induction of hepatocyte IRF-1.Adoptive transfer of ex vivo HO-1 modified bone marrow-derived macrophages prevents liver ischemia and reperfusion injury.Contribution of gut bacteria to liver pathobiology.VEGFR1-positive macrophages facilitate liver repair and sinusoidal reconstruction after hepatic ischemia/reperfusion injury.Hybrid biosynthetic gene therapy vector development and dual engineering capacity.Macrophages expressing heme oxygenase-1 improve renal function in ischemia/reperfusion injury.Resident cardiac immune cells and expression of the ectonucleotidase enzymes CD39 and CD73 after ischemic injury.Oval cell response is attenuated by depletion of liver resident macrophages in the 2-AAF/partial hepatectomy rat.Protective role of HO-1 and carbon monoxide in ethanol-induced hepatocyte cell death and liver injury in mice Ischemic preconditioning in the liver is independent of regulatory T cell activity.Remote ischemic preconditioning protects against liver ischemia-reperfusion injury via heme oxygenase-1-induced autophagy Regulation of alternative macrophage activation in the liver following acetaminophen intoxication by stem cell-derived tyrosine kinase.Nilotinib protects the murine liver from ischemia/reperfusion injury.IFN-γ-induced iNOS expression in mouse regulatory macrophages prolongs allograft survival in fully immunocompetent recipients.Disruption of Type-I IFN pathway ameliorates preservation damage in mouse orthotopic liver transplantation via HO-1 dependent mechanism.Heme oxygenase-1 exerts a protective role in ovalbumin-induced neutrophilic airway inflammation by inhibiting Th17 cell-mediated immune response.Cell biology of ischemia/reperfusion injury.Haem oxygenase-1: non-canonical roles in physiology and pathology.The mononuclear phagocyte system in homeostasis and disease: a role for heme oxygenase-1.Heme on innate immunity and inflammation.The Mononuclear Phagocyte System in Organ Transplantation.Prolonged Ischemia Triggers Necrotic Depletion of Tissue-Resident Macrophages To Facilitate Inflammatory Immune Activation in Liver Ischemia Reperfusion Injury.Innate Immune Regulations and Liver Ischemia-Reperfusion Injury.Ischemia/Reperfusion.An Evaluation of Ischaemic Preconditioning as a Method of Reducing Ischaemia Reperfusion Injury in Liver Surgery and Transplantation.Heme oxygenase-1 protects donor livers from ischemia/reperfusion injury: the role of Kupffer cells.Modulation of liver regeneration via myeloid PTEN deficiency.Heme oxygenase system in hepatic ischemia-reperfusion injury Cellular-specific role of toll-like receptor 4 in hepatic ischemia-reperfusion injury in mice.

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Tissue-resident macrophages protect the liver from ischemia reperfusion injury via a heme oxygenase-1-dependent mechanism.

http://www.wikidata.org/entity/Q39501283

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

Tissue-resident macrophages pr ...... ygenase-1-dependent mechanism.

@en

Tissue-resident macrophages pr ...... ygenase-1-dependent mechanism.

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type

label

Tissue-resident macrophages pr ...... ygenase-1-dependent mechanism.

@en

Tissue-resident macrophages pr ...... ygenase-1-dependent mechanism.

@nl

prefLabel

Tissue-resident macrophages pr ...... ygenase-1-dependent mechanism.

@en

Tissue-resident macrophages pr ...... ygenase-1-dependent mechanism.

@nl

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Molecular Therapy

P1476

Tissue-resident macrophages pr ...... xygenase-1-dependent mechanism

@en

P2093

Christopher O Bellamy

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Jeremy Hughes

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Kathryn Sangster

http://www.w3.org/2001/XMLSchema#string

P2860

The danger model: a renewed sense of self

The macrophage F4/80 receptor is required for the induction of antigen-specific efferent regulatory T cells in peripheral tolerance

Tolerance, danger, and the extended family

Blood monocytes consist of two principal subsets with distinct migratory properties

Monitoring of blood vessels and tissues by a population of monocytes with patrolling behavior

Macrophage-restricted molecules: role in differentiation and activation.

Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair.

Kupffer cell-dependent TNF-alpha signaling mediates injury in the arterialized small-for-size liver transplantation in the mouse.

Heme oxygenase 1 mediates an adaptive response to oxidative stress in human skin fibroblasts.

Serial analysis of gene expression provides new insights into regulatory T cells.

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65-72

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scholarly article

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10.1038/MT.2008.237

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1

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17

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David A Ferenbach

Stephen J. Wigmore

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2008-11-11T00:00:00Z

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23466619

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19002167

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P921

reperfusion injury

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2834991

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