Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
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The primary vascular dysregulation syndrome: implications for eye diseasesEndothelin-1 and its role in the pathogenesis of infectious diseasesEndothelin-converting enzymes and related metalloproteases in Alzheimer's diseaseVascular dysfunction as a target for adjuvant therapy in cerebral malariaBiomarkers in Multiple Sclerosis: An Up-to-Date OverviewTargeting endothelin receptors A and B attenuates the inflammatory response and improves locomotor function following spinal cord injury in mice.The role of the blood-brain barrier in the development and treatment of migraine and other pain disorders.Milano summer particulate matter (PM10) triggers lung inflammation and extra pulmonary adverse events in mice.A novel preclinical method to quantitatively evaluate early-stage metastatic events at the murine blood-brain barrierEvidence of different mediators of central inflammation in dysfunctional and inflammatory pain--interleukin-8 in fibromyalgia and interleukin-1 β in rheumatoid arthritisDecreased endothelin-1 plasma levels in multiple sclerosis patients: a possible factor of vascular dysregulation?Sphingosine 1-phosphate receptor 5 mediates the immune quiescence of the human brain endothelial barrier.Neurovascular unit in chronic pain.Overview of experimental models of the blood-brain barrier in CNS drug discovery.ECIS technology reveals that monocytes isolated by CD14+ve selection mediate greater loss of BBB integrity than untouched monocytes, which occurs to a greater extent with IL-1β activated endothelium in comparison to TNFα.Reduced expression of PGC-1α partly underlies mitochondrial changes and correlates with neuronal loss in multiple sclerosis cortex.Cdc42 GTPases facilitate TNF-α-mediated secretion of CCL2 from peripheral nerve microvascular endoneurial endothelial cells.MS risk genes are transcriptionally regulated in CSF leukocytes at relapse.Biomaterial strategies for limiting the impact of secondary events following spinal cord injury.Widespread microglial activation in patients deceased from traumatic brain injury.Early-stage minimally invasive procedures decrease perihematomal endothelin-1 levels and improve neurological functioning in a rabbit model of intracerebral hemorrhage.Roles for HB-EGF and CD9 in multiple sclerosis.
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P2860
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
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2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
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2011年學術文章
@zh-hant
name
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
@en
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
@nl
type
label
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
@en
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
@nl
prefLabel
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
@en
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
@nl
P2093
P2860
P1476
Brain endothelial barrier passage by monocytes is controlled by the endothelin system.
@en
P2093
Arie Reijerkerk
Bert van Het Hof
Gijs Kooij
Helga E de Vries
Joost A R Drexhage
Kim A M Lakeman
Susanne M A van der Pol
Yolanda van Wijck
P2860
P304
P356
10.1111/J.1471-4159.2011.07393.X
P407
P50
P577
2011-08-12T00:00:00Z