Brucella abortus and its closest phylogenetic relative, Ochrobactrum spp., differ in outer membrane permeability and cationic peptide resistance.
about
The differential interaction of Brucella and ochrobactrum with innate immunity reveals traits related to the evolution of stealthy pathogensBrucella abortus ornithine lipids are dispensable outer membrane components devoid of a marked pathogen-associated molecular pattern.Brucella melitensis: a nasty bug with hidden credentials for virulence.Recombinant Ochrobactrum anthropi expressing Brucella abortus Cu,Zn superoxide dismutase protects mice against B. abortus infection only after switching of immune responses to Th1 typeThe lipopolysaccharide core of Brucella abortus acts as a shield against innate immunity recognitionDifferent surface charge of colistin-susceptible and -resistant Acinetobacter baumannii cells measured with zeta potential as a function of growth phase and colistin treatmentThe epitopic and structural characterization of Brucella suis biovar 2 O-polysaccharide demonstrates the existence of a new M-negative C-negative smooth Brucella serovarCharacterization of Brucella abortus O-polysaccharide and core lipopolysaccharide mutants and demonstration that a complete core is required for rough vaccines to be efficient against Brucella abortus and Brucella ovis in the mouse model.Secondary serological response of patients with chronic hepatosplenic suppurative brucellosisBrucella abortus Induces the Premature Death of Human Neutrophils through the Action of Its Lipopolysaccharide.CCL20 and Beta-Defensin 2 Production by Human Lung Epithelial Cells and Macrophages in Response to Brucella abortus Infection.Structural Studies of Lipopolysaccharide-defective Mutants from Brucella melitensis Identify a Core Oligosaccharide Critical in Virulence.Brucellosis vaccines: assessment of Brucella melitensis lipopolysaccharide rough mutants defective in core and O-polysaccharide synthesis and exportVirulence factors in brucellosis: implications for aetiopathogenesis and treatment.Interplay between two RND systems mediating antimicrobial resistance in Brucella suis.Key role of Toll-like receptor 2 in the inflammatory response and major histocompatibility complex class ii downregulation in Brucella abortus-infected alveolar macrophages.An evolutionary strategy for a stealthy intracellular Brucella pathogen.Brucella central carbon metabolism: an update.Adrenal steroids modulate the immune response during Brucella abortus infection by a mechanism that depends on the regulation of cytokine production.Pathogenic Yersinia enterocolitica strains increase the outer membrane permeability in response to environmental stimuli by modulating lipopolysaccharide fluidity and lipid A structure.B. abortus RNA is the component involved in the down-modulation of MHC-I expression on human monocytes via TLR8 and the EGFR pathway.Temperature-dependent regulation of the Ochrobactrum anthropi proteome.Efficacy of several serological tests and antigens for diagnosis of bovine brucellosis in the presence of false-positive serological results due to Yersinia enterocolitica O:9.Differential inductions of TNF-alpha and IGTP, IIGP by structurally diverse classic and non-classic lipopolysaccharides.Brucella abortus inhibits major histocompatibility complex class II expression and antigen processing through interleukin-6 secretion via Toll-like receptor 2Brucella melitensis MucR, an orthologue of Sinorhizobium meliloti MucR, is involved in resistance to oxidative, detergent, and saline stresses and cell envelope modifications.Thermodynamic analysis of the lipopolysaccharide-dependent resistance of gram-negative bacteria against polymyxin B.The lipopolysaccharide of Brucella abortus BvrS/BvrR mutants contains lipid A modifications and has higher affinity for bactericidal cationic peptides.Brucella abortus induces intracellular retention of MHC-I molecules in human macrophages down-modulating cytotoxic CD8(+) T cell responses.Identification of lptA, lpxE, and lpxO, Three Genes Involved in the Remodeling of Brucella Cell Envelope.Immunomodulatory properties of Brucella melitensis lipopolysaccharide determinants on mouse dendritic cells in vitro and in vivo.Lipopolysaccharide interaction is decisive for the activity of the antimicrobial peptide NK-2 against Escherichia coli and Proteus mirabilis.Synthesis of phosphatidylcholine, a typical eukaryotic phospholipid, is necessary for full virulence of the intracellular bacterial parasite Brucella abortus
P2860
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P2860
Brucella abortus and its closest phylogenetic relative, Ochrobactrum spp., differ in outer membrane permeability and cationic peptide resistance.
description
2000 nî lūn-bûn
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2000年の論文
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2000年論文
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2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
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2000年論文
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2000年论文
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2000年论文
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name
Brucella abortus and its close ...... d cationic peptide resistance.
@en
Brucella abortus and its close ...... d cationic peptide resistance.
@nl
type
label
Brucella abortus and its close ...... d cationic peptide resistance.
@en
Brucella abortus and its close ...... d cationic peptide resistance.
@nl
prefLabel
Brucella abortus and its close ...... d cationic peptide resistance.
@en
Brucella abortus and its close ...... d cationic peptide resistance.
@nl
P2093
P2860
P50
P1476
Brucella abortus and its close ...... d cationic peptide resistance.
@en
P2093
P2860
P304
P356
10.1128/IAI.68.6.3210-3218.2000
P407
P577
2000-06-01T00:00:00Z