Changes in human immunodeficiency virus type 1 fitness and genetic diversity during disease progression - Wikidata - awgldk - TriplyDB

Changes in human immunodeficiency virus type 1 fitness and genetic diversity during disease progression

Changes in human immunodeficiency virus type 1 fitness and genetic diversity during disease progression

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Variable fitness impact of HIV-1 escape mutations to cytotoxic T lymphocyte (CTL) response Evolution of subtype C HIV-1 Env in a slowly progressing Zambian infant.Modelling the course of an HIV infection: insights from ecology and evolution Origin of the transmitted virus in HIV infection: infected cells versus cell-free virus The Evolutionary Dynamics of a Rapidly Mutating Virus within and between Hosts: The Case of Hepatitis C Virus HIV and antiretroviral therapy in the brain: neuronal injury and repair Escape of HIV-1 from a small molecule CCR5 inhibitor is not associated with a fitness loss Maraviroc: the evidence for its potential in the management of HIV Assessing predicted HIV-1 replicative capacity in a clinical setting HIV-1 Env C2-V4 diversification in a slow-progressor infant reveals a flat but rugged fitness landscape Defining the fitness of HIV-1 isolates with dual/mixed co-receptor usage HIV-1 Genetic Variability and Clinical Implications PAPNC, a novel method to calculate nucleotide diversity from large scale next generation sequencing data.Understanding the slow depletion of memory CD4+ T cells in HIV infection.Dynamic correlation between intrahost HIV-1 quasispecies evolution and disease progression.Latent coinfection and the maintenance of strain diversity.Host HLA B*allele-associated multi-clade Gag T-cell recognition correlates with slow HIV-1 disease progression in antiretroviral therapy-naïve Ugandans.Viral load levels measured at set-point have risen over the last decade of the HIV epidemic in the Netherlands.Characterization of the molecular determinants of primary HIV-1 Vpr proteins: impact of the Q65R and R77Q substitutions on Vpr functions.Using genomic signatures for HIV-1 sub-typing.Replicative capacity of human immunodeficiency virus type 1 transmitted from mother to child is associated with pediatric disease progression rate Initial fitness recovery of HIV-1 is associated with quasispecies heterogeneity and can occur without modifications in the consensus sequence.HIV-1 Transmission, Replication Fitness and Disease Progression.The replicative fitness of primary human immunodeficiency virus type 1 (HIV-1) group M, HIV-1 group O, and HIV-2 isolates.Impaired replication capacity of acute/early viruses in persons who become HIV controllers.A comparative study of HIV-1 clade C env evolution in a Zambian infant with an infected rhesus macaque during disease progression.Analysis of infectious virus clones from two HIV-1 superinfection cases suggests that the primary strains have lower fitness.High viral fitness during acute HIV-1 infection.Gag-protease-mediated replication capacity in HIV-1 subtype C chronic infection: associations with HLA type and clinical parameters Relative replication capacity of phenotypic SIV variants during primary infections differs with route of inoculation.Stochastic model of in-vivo X4 emergence during HIV infection: implications for the CCR5 inhibitor maraviroc.Evolution of virulence in emerging epidemics.HIV-1 p24(gag) derived conserved element DNA vaccine increases the breadth of immune response in mice.Evaluation of a multiple-cycle, recombinant virus, growth competition assay that uses flow cytometry to measure replication efficiency of human immunodeficiency virus type 1 in cell culture.Variations in the Biological Functions of HIV-1 Clade C Envelope in a SHIV-Infected Rhesus Macaque during Disease Progression.The predominance of Human Immunodeficiency Virus type 1 (HIV-1) circulating recombinant form 02 (CRF02_AG) in West Central Africa may be related to its replicative fitness.Gastrointestinal viral load and enteroendocrine cell number are associated with altered survival in HIV-1 infected individuals.In vivo fitness associated with high virulence in a vertebrate virus is a complex trait regulated by host entry, replication, and shedding.Characterization of HIV-1 subtype C envelope glycoproteins from perinatally infected children with different courses of disease.Similar replicative fitness is shared by the subtype B and unique BF recombinant HIV-1 isolates that dominate the epidemic in Argentina.

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Changes in human immunodeficiency virus type 1 fitness and genetic diversity during disease progression

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Changes in human immunodeficie ...... ity during disease progression

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Changes in human immunodeficie ...... ity during disease progression

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JVI.79.14.9006-9018.2005

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Journal of Virology

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Changes in human immunodeficie ...... ity during disease progression

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Awet Abraha

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Dawn M Moore

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Eric J Arts

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Erika Fraundorf

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Guido Vanham

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James I Mullins

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Kalonji R Collins

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Mark A Jensen

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Randall W Krizan

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Robert L Colebunders

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P2860

The CLUSTAL_X windows interface: flexible strategies for multiple sequence alignment aided by quality analysis tools

Positive selection of HIV-1 cytotoxic T lymphocyte escape variants during primary infection

Simple methods for estimating the numbers of synonymous and nonsynonymous nucleotide substitutions

A simple method for estimating evolutionary rates of base substitutions through comparative studies of nucleotide sequences

MEGA2: molecular evolutionary genetics analysis software

Genomic structure of an attenuated quasi species of HIV-1 from a blood transfusion donor and recipients

Antiviral pressure exerted by HIV-1-specific cytotoxic T lymphocytes (CTLs) during primary infection demonstrated by rapid selection of CTL escape virus

Brief report: absence of intact nef sequences in a long-term survivor with nonprogressive HIV-1 infection

Variable sensitivity of CCR5-tropic human immunodeficiency virus type 1 isolates to inhibition by RANTES analogs

Antibody neutralization and escape by HIV-1

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9006-9018

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