Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen.
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Multifaceted Role of Sialylation in Prion DiseasesThe Good, the Bad, and the Ugly of Dendritic Cells during Prion DiseaseThe diverse roles of mononuclear phagocytes in prion disease pathogenesisNeuroinvasion of α-Synuclein Prionoids after Intraperitoneal and Intraglossal Inoculation.Lymphotoxin, but not TNF, is required for prion invasion of lymph nodes.A lympho-follicular microenvironment is required for pathological prion protein deposition in chronically inflamed tissues from scrapie-affected sheepB cell-specific S1PR1 deficiency blocks prion dissemination between secondary lymphoid organsIncunabular immunological events in prion trafficking.Increased Abundance of M Cells in the Gut Epithelium Dramatically Enhances Oral Prion Disease SusceptibilityFollicular dendritic cells in health and disease.Genetic depletion of complement receptors CD21/35 prevents terminal prion disease in a mouse model of chronic wasting disease.Post-conversion sialylation of prions in lymphoid tissues.Prions and lymphoid organs: solved and remaining mysteries.Complement protein C3 exacerbates prion disease in a mouse model of chronic wasting disease.Role of palatine tonsils as a prion entry site in classical and atypical experimental sheep scrapie.Prion disease and the innate immune system.Determining the role of mononuclear phagocytes in prion neuroinvasion from the skin.Prion disease pathogenesis in the absence of the commensal microbiota.Sialylation of Glycosylphosphatidylinositol (GPI) Anchors of Mammalian Prions Is Regulated in a Host-, Tissue-, and Cell-specific MannerOral Prion Disease Pathogenesis Is Impeded in the Specific Absence of CXCR5-Expressing Dendritic Cells.MicroRNA-100-5p indirectly modulates the expression of Il6, Ptgs1/2 and Tlr4 mRNA in the mouse follicular dendritic cell-like cell line, FL-Y.Prion pathogenesis and secondary lymphoid organs (SLO): tracking the SLO spread of prions to the brainAnalyses of N-linked glycans of PrPSc revealed predominantly 2,6-linked sialic acid residues.The effect of PrP(Sc) accumulation on inflammatory gene expression within sheep peripheral lymphoid tissueSialylation Controls Prion Fate in Vivo.Prion pathogenesis is unaltered following down-regulation of SIGN-R1.The Gut-Associated Lymphoid Tissues in the Small Intestine, Not the Large Intestine, Play a Major Role in Oral Prion Disease Pathogenesis.M cell-depletion blocks oral prion disease pathogenesis.Peripheral prion disease pathogenesis is unaltered in the absence of sialoadhesin (Siglec-1/CD169).How do PrPSc Prions Spread between Host Species, and within Hosts?Prion Strains and Transmission Barrier Phenomena.Complement Regulatory Protein Factor H Is a Soluble Prion Receptor That Potentiates Peripheral Prion Pathogenesis.Oral Prion Neuroinvasion Occurs Independently of PrP Expression in the Gut Epithelium
P2860
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P2860
Follicular dendritic cell-specific prion protein (PrP) expression alone is sufficient to sustain prion infection in the spleen.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Follicular dendritic cell-spec ...... prion infection in the spleen.
@en
Follicular dendritic cell-specific prion protein
@nl
type
label
Follicular dendritic cell-spec ...... prion infection in the spleen.
@en
Follicular dendritic cell-specific prion protein
@nl
prefLabel
Follicular dendritic cell-spec ...... prion infection in the spleen.
@en
Follicular dendritic cell-specific prion protein
@nl
P2093
P2860
P50
P1433
P1476
Follicular dendritic cell-spec ...... prion infection in the spleen.
@en
P2093
John Hopkins
Karen L Brown
Laura McCulloch
Mick Bailey
P2860
P304
P356
10.1371/JOURNAL.PPAT.1002402
P577
2011-12-01T00:00:00Z