Multiple factors independently regulate hilA and invasion gene expression in Salmonella enterica serovar typhimurium.
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CHEMOTAXIS-GUIDED MOVEMENTS IN BACTERIASensing wetness: a new role for the bacterial flagellum.Detection and characterization of the S. typhimurium HilA proteinRegulatory principles governing Salmonella and Yersinia virulenceNon-genetic diversity shapes infectious capacity and host resistanceSalmonella enterica serovars Typhimurium and Typhi as model organisms: revealing paradigm of host-pathogen interactionsThe phosphate regulon and bacterial virulence: a regulatory network connecting phosphate homeostasis and pathogenesisIdentification of Mycobacterium tuberculosis counterimmune (cim) mutants in immunodeficient mice by differential screeningMultiple FadD acyl-CoA synthetases contribute to differential fatty acid degradation and virulence in Pseudomonas aeruginosaThe Salmonella SPI1 type three secretion system responds to periplasmic disulfide bond status via the flagellar apparatus and the RcsCDB systemFadD is required for utilization of endogenous fatty acids released from membrane lipidsMycobacterium avium genes MAV_5138 and MAV_3679 are transcriptional regulators that play a role in invasion of epithelial cells, in part by their regulation of CipA, a putative surface protein interacting with host cell signaling pathwaysDNA-binding activities of the HilC and HilD virulence regulatory proteins of Salmonella enterica serovar TyphimuriumNovel attenuated Salmonella enterica serovar Choleraesuis strains as live vaccine candidates generated by signature-tagged mutagenesisRole of flagella in host cell invasion by Burkholderia cepaciaAlterations in Vibrio fischeri motility correlate with a delay in symbiosis initiation and are associated with additional symbiotic colonization defects.Functional genomics approach to the identification of virulence genes involved in Edwardsiella tarda pathogenesis.Salmonella enterica Serovar Typhi conceals the invasion-associated type three secretion system from the innate immune system by gene regulation.Extensive alanine scanning reveals protein-protein and protein-DNA interaction surfaces in the global regulator FlhD from Escherichia coli.Use of in vivo-induced antigen technology for identification of Escherichia coli O157:H7 proteins expressed during human infection.Identification of Mycobacterium avium genes that affect invasion of the intestinal epithelium.Systematic analysis of the regulation of type three secreted effectors in Salmonella enterica serovar Typhimurium.Contribution of the pst-phoU operon to cell adherence by atypical enteropathogenic Escherichia coli and virulence of Citrobacter rodentiumThe role of coupled positive feedback in the expression of the SPI1 type three secretion system in Salmonella.Analysis of factors that affect FlgM-dependent type III secretion for protein purification with Salmonella enterica serovar Typhimurium.Microarray analysis and motif detection reveal new targets of the Salmonella enterica serovar Typhimurium HilA regulatory protein, including hilA itself.Regulation of a type III and a putative secretion system in Edwardsiella tarda by EsrC is under the control of a two-component system, EsrA-EsrBppGpp conjures bacterial virulenceThe high-affinity phosphate transporter Pst in Proteus mirabilis HI4320 and its importance in biofilm formation.Unravelling the mysteries of virulence gene regulation in Salmonella typhimurium.Hha is a negative modulator of transcription of hilA, the Salmonella enterica serovar Typhimurium invasion gene transcriptional activatorAbsence of all components of the flagellar export and synthesis machinery differentially alters virulence of Salmonella enterica serovar Typhimurium in models of typhoid fever, survival in macrophages, tissue culture invasiveness, and calf enterocolOpsonized virulent Edwardsiella tarda strains are able to adhere to and survive and replicate within fish phagocytes but fail to stimulate reactive oxygen intermediatesIn vivo genetic analysis indicates that PhoP-PhoQ and the Salmonella pathogenicity island 2 type III secretion system contribute independently to Salmonella enterica serovar Typhimurium virulence.Antivirulence activity of the human gut metabolome.A stationary-phase acyl-coenzyme A synthetase of Streptomyces coelicolor A3(2) is necessary for the normal onset of antibiotic production.Role of cross talk in regulating the dynamic expression of the flagellar Salmonella pathogenicity island 1 and type 1 fimbrial genesThe flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.Pathways leading from BarA/SirA to motility and virulence gene expression in Salmonella.Role of Salmonella enterica serovar Typhimurium two-component system PreA/PreB in modulating PmrA-regulated gene transcription.
P2860
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P2860
Multiple factors independently regulate hilA and invasion gene expression in Salmonella enterica serovar typhimurium.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Multiple factors independently ...... enterica serovar typhimurium.
@en
Multiple factors independently ...... enterica serovar typhimurium.
@nl
type
label
Multiple factors independently ...... enterica serovar typhimurium.
@en
Multiple factors independently ...... enterica serovar typhimurium.
@nl
prefLabel
Multiple factors independently ...... enterica serovar typhimurium.
@en
Multiple factors independently ...... enterica serovar typhimurium.
@nl
P2093
P2860
P1476
Multiple factors independently ...... enterica serovar typhimurium.
@en
P2093
B L Wanner
C C DiRusso
C P Lostroh
M P Spector
P2860
P304
P356
10.1128/JB.182.7.1872-1882.2000
P407
P577
2000-04-01T00:00:00Z