Human coronavirus 229E infects polarized airway epithelia from the apical surface
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ACE2 receptor expression and severe acute respiratory syndrome coronavirus infection depend on differentiation of human airway epithelia.Coronaviruses and the human airway: a universal system for virus-host interaction studiesApical entry and release of severe acute respiratory syndrome-associated coronavirus in polarized Calu-3 lung epithelial cellsFoot-and-mouth disease virus replicates only transiently in well-differentiated porcine nasal epithelial cells.Culturing the unculturable: human coronavirus HKU1 infects, replicates, and produces progeny virions in human ciliated airway epithelial cell cultures.Severe acute respiratory syndrome coronavirus infection of human ciliated airway epithelia: role of ciliated cells in viral spread in the conducting airways of the lungs.Respiratory syncytial virus infection of human airway epithelial cells is polarized, specific to ciliated cells, and without obvious cytopathologySuppression of adenosine-activated chloride transport by ethanol in airway epitheliaThe RNA binding domain of influenza A virus NS1 protein affects secretion of tumor necrosis factor alpha, interleukin-6, and interferon in primary murine tracheal epithelial cellsInfection of human alveolar macrophages by human coronavirus strain 229E.SARS-CoV replication and pathogenesis in an in vitro model of the human conducting airway epitheliumIsolation and characterization of current human coronavirus strains in primary human epithelial cell cultures reveal differences in target cell tropismHost-pathogen interactions during coronavirus infection of primary alveolar epithelial cells.Glycocalyx restricts adenoviral vector access to apical receptors expressed on respiratory epithelium in vitro and in vivo: role for tethered mucins as barriers to lumenal infection.Coronavirus entry and release in polarized epithelial cells: a review.TMPRSS2 activates the human coronavirus 229E for cathepsin-independent host cell entry and is expressed in viral target cells in the respiratory epithelium.Identification of a receptor-binding domain of the spike glycoprotein of human coronavirus HCoV-229E.Measles virus preferentially transduces the basolateral surface of well-differentiated human airway epithelia.In vitro modeling of human bocavirus 1 infection of polarized primary human airway epithelia.Small interfering RNA inhibits SARS-CoV nucleocapsid gene expression in cultured cells and mouse muscles.Human bocavirus 1 infects commercially available primary human airway epithelium cultures productively.Bidirectional virus secretion and nonciliated cell tropism following Andes virus infection of primary airway epithelial cell cultures.Apical barriers to airway epithelial cell gene transfer with amphotropic retroviral vectors.
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Human coronavirus 229E infects polarized airway epithelia from the apical surface
description
2000 nî lūn-bûn
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2000年の論文
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2000年論文
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2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
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2000年論文
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name
Human coronavirus 229E infects polarized airway epithelia from the apical surface
@en
Human coronavirus 229E infects polarized airway epithelia from the apical surface.
@nl
type
label
Human coronavirus 229E infects polarized airway epithelia from the apical surface
@en
Human coronavirus 229E infects polarized airway epithelia from the apical surface.
@nl
prefLabel
Human coronavirus 229E infects polarized airway epithelia from the apical surface
@en
Human coronavirus 229E infects polarized airway epithelia from the apical surface.
@nl
P2093
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Human coronavirus 229E infects polarized airway epithelia from the apical surface
@en
P2093
B L Davidson
K V Holmes
P B McCray
P2860
P304
P356
10.1128/JVI.74.19.9234-9239.2000
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P577
2000-10-01T00:00:00Z