Pathogenicity of different rabies virus variants inversely correlates with apoptosis and rabies virus glycoprotein expression in infected primary neuron cultures.
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Mechanisms of escape phenomenon of spinal cord and brainstem in human rabies.Borna disease virus blocks potentiation of presynaptic activity through inhibition of protein kinase C signaling.Subversion of the Immune Response by Rabies VirusRabies Internalizes into Primary Peripheral Neurons via Clathrin Coated Pits and Requires Fusion at the Cell BodyIn vitro and in vivo isolation and characterization of Duvenhage virusNovel Approaches to the Prevention and Treatment of RabiesPathological lesions in the central nervous system and peripheral tissues of ddY mice with street rabies virus (1088 strain).Overexpression of cytochrome C by a recombinant rabies virus attenuates pathogenicity and enhances antiviral immunity.Expression and immunogenicity of human immunodeficiency virus type 1 Gag expressed by a replication-competent rhabdovirus-based vaccine vector.Increased transgene expression level of rabies virus vector for transsynaptic tracingAttenuated rabies virus activates, while pathogenic rabies virus evades, the host innate immune responses in the central nervous system.Molecular basis of neurovirulence of flury rabies virus vaccine strains: importance of the polymerase and the glycoprotein R333Q mutation.A single amino acid change in rabies virus glycoprotein increases virus spread and enhances virus pathogenicity.The 3' untranslated region of the rabies virus glycoprotein mRNA specifically interacts with cellular PCBP2 protein and promotes transcript stability.Sequencing and molecular characterization of CTNCEC25, a China fixed rabies virus vaccine strain CTN-1 adapted to primary chicken embryo cells.Overexpression of the rabies virus glycoprotein results in enhancement of apoptosis and antiviral immune response.Second-generation rabies virus-based vaccine vectors expressing human immunodeficiency virus type 1 gag have greatly reduced pathogenicity but are highly immunogenicEffect of rabies virus infection on gene expression in mouse brainMolecular characterization of Korean rabies virus isolates.Diagnosis and molecular characterization of rabies virus from a buffalo in China: a case reportThe inability of wild-type rabies virus to activate dendritic cells is dependent on the glycoprotein and correlates with its low level of the de novo-synthesized leader RNA.Role of apoptosis in rabies viral encephalitis: a comparative study in mice, canine, and human brain with a review of literature.Generation of a recombinant rabies Flury LEP virus carrying an additional G gene creates an improved seed virus for inactivated vaccine production.Effects of G-gene Deletion and Replacement on Rabies Virus Vector Gene Expression.A Single Amino Acid in the M1 Protein Responsible for the Different Pathogenic Potentials of H5N1 Highly Pathogenic Avian Influenza Virus Strains.iTRAQ protein profile analysis of neuroblastoma (NA) cells infected with the rabies viruses rHep-Flury and Hep-dG.Postexposure treatment with the live-attenuated rabies virus (RV) vaccine TriGAS triggers the clearance of wild-type RV from the Central Nervous System (CNS) through the rapid induction of genes relevant to adaptive immunity in CNS tissues.Dominance of a nonpathogenic glycoprotein gene over a pathogenic glycoprotein gene in rabies virusEvidence of rabies virus exposure among humans in the Peruvian AmazonImmune clearance of attenuated rabies virus results in neuronal survival with altered gene expression.Relationship between within-host fitness and virulence in the vesicular stomatitis virus: correlation with partial decouplingStructural abnormalities in neurons are sufficient to explain the clinical disease and fatal outcome of experimental rabies in yellow fluorescent protein-expressing transgenic mice.The glycoprotein and the matrix protein of rabies virus affect pathogenicity by regulating viral replication and facilitating cell-to-cell spread.Intramuscular inoculation of mice with the live-attenuated recombinant rabies virus TriGAS results in a transient infection of the draining lymph nodes and a robust, long-lasting protective immune response against rabies.Rabies virus glycoprotein is an important determinant for the induction of innate immune responses and the pathogenic mechanisms.Quantitative proteomics for identifying biomarkers for Rabies.Region between the canine distemper virus M and F genes modulates virulence by controlling fusion protein expression.Proteomic analysis reveals selective impediment of neuronal remodeling upon Borna disease virus infection.Interspecies protein substitution to investigate the role of the lyssavirus glycoprotein.Immune modulating effect by a phosphoprotein-deleted rabies virus vaccine vector expressing two copies of the rabies virus glycoprotein gene
P2860
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P2860
Pathogenicity of different rabies virus variants inversely correlates with apoptosis and rabies virus glycoprotein expression in infected primary neuron cultures.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Pathogenicity of different rab ...... ected primary neuron cultures.
@en
Pathogenicity of different rab ...... ected primary neuron cultures.
@nl
type
label
Pathogenicity of different rab ...... ected primary neuron cultures.
@en
Pathogenicity of different rab ...... ected primary neuron cultures.
@nl
prefLabel
Pathogenicity of different rab ...... ected primary neuron cultures.
@en
Pathogenicity of different rab ...... ected primary neuron cultures.
@nl
P2093
P2860
P1433
P1476
Pathogenicity of different rab ...... fected primary neuron cultures
@en
P2093
B Dietzschold
D C Hooper
H Koprowski
K Morimoto
P2860
P304
P407
P577
1999-01-01T00:00:00Z