about
Current targeted therapies in the treatment of advanced colorectal cancer: a reviewAltered mitochondrial function and energy metabolism is associated with a radioresistant phenotype in oesophageal adenocarcinomaMaternal topoisomerase II alpha, not topoisomerase II beta, enables embryonic development of zebrafish top2a-/- mutants.Tumour tissue microenvironment can inhibit dendritic cell maturation in colorectal cancerArray-based comparative genomic hybridization in ulcerative colitis neoplasia: single non-dysplastic biopsies distinguish progressors from non-progressors.Tumour microenvironment of both early- and late-stage colorectal cancer is equally immunosuppressive.Decreased mitochondrial DNA mutagenesis in human colorectal cancer.A prospective investigation of predictive and modifiable risk factors for breast cancer in unaffected BRCA1 and BRCA2 gene carriers.Excess visceral adiposity induces alterations in mitochondrial function and energy metabolism in esophageal adenocarcinoma.Predicting response to vascular endothelial growth factor inhibitor and chemotherapy in metastatic colorectal cancer.Polymorphisms near TBX5 and GDF7 are associated with increased risk for Barrett's esophagus.Overexpression of the microRNA miR-433 promotes resistance to paclitaxel through the induction of cellular senescence in ovarian cancer cells.LC3B globular structures correlate with survival in esophageal adenocarcinoma.Changes in mitochondrial stability during the progression of the Barrett's esophagus disease sequence.Heterogeneity of tumor-induced gene expression changes in the human metabolic network.Genomic biomarkers to improve ulcerative colitis neoplasia surveillance.Nuclear oxidative damage correlates with poor survival in colorectal cancer.Protein kinase C beta II suppresses colorectal cancer by regulating IGF-1 mediated cell survivalCellular senescence induced by aberrant MAD2 levels impacts on paclitaxel responsiveness in vitroCOX-derived prostanoid pathways in gastrointestinal cancer development and progression: novel targets for prevention and intervention.Barrett's to oesophageal cancer sequence: a model of inflammatory-driven upper gastrointestinal cancer.Could signal transducer and activator of transcription 3 be a therapeutic target in obesity-related gastrointestinal malignancy?The role of inflammation in cancer of the esophagus.Quantitative fluorescence in situ hybridization (QFISH) of telomere lengths in tissue and cells.A Quininib Analogue and Cysteinyl Leukotriene Receptor Antagonist Inhibits Vascular Endothelial Growth Factor (VEGF)-independent Angiogenesis and Exerts an Additive Antiangiogenic Response with Bevacizumab.Examining the connectivity between different cellular processes in the Barrett tissue microenvironment.Emerging Concepts Linking Obesity with the Hallmarks of Cancer.Differential expression of mitochondrial energy metabolism profiles across the metaplasia-dysplasia-adenocarcinoma disease sequence in Barrett's oesophagus.Clusterin and chemotherapy sensitivity under normoxic and graded hypoxic conditions in colorectal cancer.Synovial tissue hypoxia and inflammation in vivo.Generation of an epigenetic signature by chronic hypoxia in prostate cells.Successful tumour necrosis factor (TNF) blocking therapy suppresses oxidative stress and hypoxia-induced mitochondrial mutagenesis in inflammatory arthritis.Epigallocatechin-3-gallate and bicalutamide cause growth arrest and apoptosis in NRP-152 and NRP-154 prostate epithelial cells.Impact of the inflammatory microenvironment on T-cell phenotype in the progression from reflux oesophagitis to Barrett oesophagus and oesophageal adenocarcinoma.Tamoxifen exerts agonistic effects on clusterin and complement C3 gene expression in RUCA-I primary xenografts and metastases but not normal uterus.Telomere length assessment in tissue sections by quantitative FISH: image analysis algorithms.Preclinical validation of the small molecule drug quininib as a novel therapeutic for colorectal cancer.Dysregulated bioenergetics: a key regulator of joint inflammation.The effect of green tea on oxidative damage and tumour formation in Lobund-Wistar rats.Tumor conditioned media from colorectal cancer patients inhibits dendritic cell maturation.
P50
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P50
description
hulumtuese
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researcher
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wetenschapper
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հետազոտող
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name
Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
@nl
Jacintha O'Sullivan
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type
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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prefLabel
Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
@es
Jacintha O'Sullivan
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Jacintha O'Sullivan
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Jacintha O'Sullivan
@sl
P106
P21
P31
P496
0000-0001-8622-9858