Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
about
The intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the gamesTumor necrosis factor alpha p55 receptor is important for development of memory responses to blood-stage malaria infection.T. brucei infection reduces B lymphopoiesis in bone marrow and truncates compensatory splenic lymphopoiesis through transitional B-cell apoptosisGamma interferon modulates CD95 (Fas) and CD95 ligand (Fas-L) expression and nitric oxide-induced apoptosis during the acute phase of Trypanosoma cruzi infection: a possible role in immune response controlTumor necrosis factor is a therapeutic target for immunological unbalance and cardiac abnormalities in chronic experimental Chagas' heart disease.TNF-α is involved in the abnormal thymocyte migration during experimental Trypanosoma cruzi infection and favors the export of immature cellsDefective localization of the NADPH phagocyte oxidase to Salmonella-containing phagosomes in tumor necrosis factor p55 receptor-deficient macrophages.Cell-specific activation of nuclear factor-kappaB by the parasite Trypanosoma cruzi promotes resistance to intracellular infection.Interleukin-12 p40- and Fas ligand-dependent apoptotic pathways involving STAT-1 phosphorylation are triggered during infection with a virulent strain of Toxoplasma gondiiCD44-regulated intracellular proliferation of Listeria monocytogenesApoptosis and the balance of homeostatic and pathologic responses to protozoan infection.Burkholderia cenocepacia ET12 strain activates TNFR1 signalling in cystic fibrosis airway epithelial cellsModulation of chemokine production and inflammatory responses in interferon-gamma- and tumor necrosis factor-R1-deficient mice during Trypanosoma cruzi infection.Chagas' disease: an update on immune mechanisms and therapeutic strategiesThe endogenous balance of soluble tumor necrosis factor receptors and tumor necrosis factor modulates cachexia and mortality in mice acutely infected with Trypanosoma cruzi.Tumor necrosis factor (TNF) receptor shedding controls thresholds of innate immune activation that balance opposing TNF functions in infectious and inflammatory diseasesResistant mice lacking interleukin-12 become susceptible to Trypanosoma cruzi infection but fail to mount a T helper type 2 response.Unconventional Pro-inflammatory CD4+ T Cell Response in B Cell-Deficient Mice Infected with Trypanosoma cruzi.Enhancement of natural killer (NK) cell cytotoxicity and induction of NK cell-derived interferon-gamma (IFN-gamma) display different kinetics during experimental infection with Trypanosoma cruzi
P2860
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P2860
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
@en
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
@nl
type
label
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
@en
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
@nl
prefLabel
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
@en
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
@nl
P2093
P2860
P1476
Trypanosoma cruzi infection in tumor necrosis factor receptor p55-deficient mice.
@en
P2093
E Castaños-Velez
L M Osorio
M E Rottenberg
P Biberfeld
P2860
P304
P407
P577
1998-06-01T00:00:00Z