Treatment of inflammatory and neuropathic pain by uncoupling Src from the NMDA receptor complex - Wikidata - awgldk - TriplyDB

Treatment of inflammatory and neuropathic pain by uncoupling Src from the NMDA receptor complex

Treatment of inflammatory and neuropathic pain by uncoupling Src from the NMDA receptor complex

http://www.wikidata.org/entity/Q39573083

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Resolvins RvE1 and RvD1 attenuate inflammatory pain via central and peripheral actions Biological Roles of Resolvins and Related Substances in the Resolution of Pain Identification of a novel ligand binding site in phosphoserine phosphatase from the hyperthermophilic archaeon Thermococcus onnurineus Emerging targets in neuroinflammation-driven chronic pain Emerging roles of resolvins in the resolution of inflammation and pain Targeting the EGFR/PCNA signaling suppresses tumor growth of triple-negative breast cancer cells with cell-penetrating PCNA peptides Roles of the SH2 and SH3 domains in the regulation of neuronal Src kinase functions Disrupting 5-HT(2A) receptor/PDZ protein interactions reduces hyperalgesia and enhances SSRI efficacy in neuropathic pain Cellular and Molecular Mechanisms of Pain The regulation of N-methyl-D-aspartate receptors by Src kinase.β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain.The Src family kinase inhibitor dasatinib delays pain-related behaviour and conserves bone in a rat model of cancer-induced bone pain.Tat-Mediated Peptide Intervention in Analgesia and Anesthesia.The use of ketamine in complex regional pain syndrome: possible mechanisms.Involvement of EphB1 receptors signalling in models of inflammatory and neuropathic pain.Targeting the cis-dimerization of LINGO-1 with low MW compounds affects its downstream signalling Targeting N-methyl-D-aspartate receptors for treatment of neuropathic pain Intracisternal administration of NR2 subunit antagonists attenuates the nociceptive behavior and p-p38 MAPK expression produced by compression of the trigeminal nerve root.Ligands Binding to Cell Surface Ganglioside GD2 Cause Src-Dependent Activation of N-Methyl-D-Aspartate Receptor Signaling and Changes in Cellular Morphology.Sex differences in spinal processing of transient and inflammatory colorectal stimuli in the rat N-methyl-D-aspartate receptor- and calpain-mediated proteolytic cleavage of K+-Cl- cotransporter-2 impairs spinal chloride homeostasis in neuropathic pain.Enhancement by interleukin-1β of AMPA and NMDA receptor-mediated currents in adult rat spinal superficial dorsal horn neurons.Fyn Kinase regulates GluN2B subunit-dominant NMDA receptors in human induced pluripotent stem cell-derived neurons.Inhibition of NMDAR reduces bladder hypertrophy and improves bladder function in cyclophosphamide induced cystitis CCL-1 in the spinal cord contributes to neuropathic pain induced by nerve injury.Inward-rectifying potassium (Kir) channels regulate pacemaker activity in spinal nociceptive circuits during early life.Central sensitization: a generator of pain hypersensitivity by central neural plasticity.Sustained relief of neuropathic pain by AAV-targeted expression of CBD3 peptide in rat dorsal root ganglion.Molecules in pain and sex: a developing story.Synaptic plasticity and pain: role of ionotropic glutamate receptors.Glutamate receptor phosphorylation and trafficking in pain plasticity in spinal cord dorsal horn.Aberrant regulation and function of Src family tyrosine kinases: their potential contributions to glutamate-induced neurotoxicity.Dysregulated Src upregulation of NMDA receptor activity: a common link in chronic pain and schizophrenia.Copper-dependent regulation of NMDA receptors by cellular prion protein: implications for neurodegenerative disorders.ATP P2X3 receptors and neuronal sensitization.Calcium-permeable ion channels in pain signaling.Regulating excitability of peripheral afferents: emerging ion channel targets.Modulation of nociceptive ion channels and receptors via protein-protein interactions: implications for pain relief.Neuronal and microglial mechanisms for neuropathic pain in the spinal dorsal horn and anterior cingulate cortex.Interactions of Pannexin1 channels with purinergic and NMDA receptor channels.

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Treatment of inflammatory and neuropathic pain by uncoupling Src from the NMDA receptor complex

http://www.wikidata.org/entity/Q39573083

description

2008 nî lūn-bûn

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2008年论文

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name

Treatment of inflammatory and ...... from the NMDA receptor complex

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Treatment of inflammatory and ...... rom the NMDA receptor complex.

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type

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Treatment of inflammatory and ...... from the NMDA receptor complex

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Treatment of inflammatory and ...... rom the NMDA receptor complex.

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Treatment of inflammatory and ...... from the NMDA receptor complex

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Treatment of inflammatory and ...... rom the NMDA receptor complex.

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Nature Medicine

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Treatment of inflammatory and ...... from the NMDA receptor complex

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Hoi Ki Ding

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Jeffrey R Gingrich

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Michael W Salter

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Paul W Frankland

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Simon Beggs

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Szu-Han Wang

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Xue Jun Liu

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Yi Na Dong

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P2860

The glutamate receptor ion channels

Molecular mechanisms of nociception

Glutamate-based therapeutic approaches: clinical trials with NMDA antagonists

Fyn kinase-mediated phosphorylation of NMDA receptor NR2B subunit at Tyr1472 is essential for maintenance of neuropathic pain

PSD-95 is a negative regulator of the tyrosine kinase Src in the NMDA receptor complex

Unique domain anchoring of Src to synaptic NMDA receptors via the mitochondrial protein NADH dehydrogenase subunit 2

CAKbeta/Pyk2 kinase is a signaling link for induction of long-term potentiation in CA1 hippocampus

Targeted disruption of the c-src proto-oncogene leads to osteopetrosis in mice

Neuronal plasticity: increasing the gain in pain

In vivo protein transduction: delivery of a biologically active protein into the mouse

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1325-1332

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10.1038/NM.1883

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12

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Ameet S. Sengar

Mariana Vargas-Caballero

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2008-11-16T00:00:00Z

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23475955

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19011637

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neuropathic pain

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3616027

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