Degradation of myogenic transcription factor MyoD by the ubiquitin pathway in vivo and in vitro: regulation by specific DNA binding.
about
Notch signaling induces rapid degradation of achaete-scute homolog 1.Multiple phosphorylation events control mitotic degradation of the muscle transcription factor Myf5.The beneficial role of proteolysis in skeletal muscle growth and stress adaptationRegulation of E-box DNA binding during in vivo and in vitro activation of rat and human hepatic stellate cellsDown-regulation of MyoD by calpain 3 promotes generation of reserve cells in C2C12 myoblastsThe ubiquitin-proteasome pathway: on protein death and cell lifeAbduction and asylum in the lives of transcription factorsp57(Kip2) stabilizes the MyoD protein by inhibiting cyclin E-Cdk2 kinase activity in growing myoblasts.A novel conserved isoform of the ubiquitin ligase UFD2a/UBE4B is expressed exclusively in mature striated muscle cells.Identification of novel MyoD gene targets in proliferating myogenic stem cellsp27kip1 independently promotes neuronal differentiation and migration in the cerebral cortex.The N-terminal domain of MyoD is necessary and sufficient for its nuclear localization-dependent degradation by the ubiquitin systemMitogen-activated protein kinase kinase 1 (MEK1) stabilizes MyoD through direct phosphorylation at tyrosine 156 during myogenic differentiation.Regulation of ubiquitin-proteasome system mediated degradation by cytosolic stress.Reciprocal inhibition between Pax7 and muscle regulatory factors modulates myogenic cell fate determination.Ubiquitin-Dependent Modification of Skeletal Muscle by the Parasitic Nematode, Trichinella spiralisEffects of myogenin on expression of late muscle genes through MyoD-dependent chromatin remodeling ability of myogenin.Marking the tempo for myogenesis: Pax7 and the regulation of muscle stem cell fate decisionsProtein 4.1R Influences Myogenin Protein Stability and Skeletal Muscle Differentiation.Identification of the immunoproteasome as a novel regulator of skeletal muscle differentiation.Structural motifs involved in ubiquitin-mediated processing of the NF-kappaB precursor p105: roles of the glycine-rich region and a downstream ubiquitination domain.Constitutive instability of muscle regulatory factor Myf5 is distinct from its mitosis-specific disappearance, which requires a D-box-like motif overlapping the basic domain.MyoD-dependent induction during myoblast differentiation of p204, a protein also inducible by interferon.Pax-7 up-regulation inhibits myogenesis and cell cycle progression in satellite cells: a potential mechanism for self-renewalHigh avidity binding to DNA protects ubiquitylated substrates from proteasomal degradation.An isogenetic myoblast expression screen identifies DUX4-mediated FSHD-associated molecular pathologies.Multiple novel functions of lamina associated polypeptide 2α in striated muscle
P2860
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P2860
Degradation of myogenic transcription factor MyoD by the ubiquitin pathway in vivo and in vitro: regulation by specific DNA binding.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Degradation of myogenic transc ...... ation by specific DNA binding.
@en
Degradation of myogenic transc ...... ation by specific DNA binding.
@nl
type
label
Degradation of myogenic transc ...... ation by specific DNA binding.
@en
Degradation of myogenic transc ...... ation by specific DNA binding.
@nl
prefLabel
Degradation of myogenic transc ...... ation by specific DNA binding.
@en
Degradation of myogenic transc ...... ation by specific DNA binding.
@nl
P2093
P2860
P1476
Degradation of myogenic transc ...... ation by specific DNA binding.
@en
P2093
K Breitschopf
O Abu Hatoum
S Gross-Mesilaty
P2860
P304
P407
P577
1998-10-01T00:00:00Z