Lytic but not latent replication of epstein-barr virus is associated with PML and induces sequential release of nuclear domain 10 proteins.
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EBV tegument protein BNRF1 disrupts DAXX-ATRX to activate viral early gene transcriptionMediation of Epstein-Barr virus EBNA-LP transcriptional coactivation by Sp100The Epstein-Barr virus replication protein BBLF2/3 provides an origin-tethering function through interaction with the zinc finger DNA binding protein ZBRK1 and the KAP-1 corepressorEpstein-Barr virus (EBV) SM protein induces and recruits cellular Sp110b to stabilize mRNAs and enhance EBV lytic gene expressionAbility of the human cytomegalovirus IE1 protein to modulate sumoylation of PML correlates with its functional activities in transcriptional regulation and infectivity in cultured fibroblast cellsEbola virus infection inversely correlates with the overall expression levels of promyelocytic leukaemia (PML) protein in cultured cellsHerpesvirus Late Gene Expression: A Viral-Specific Pre-initiation Complex Is KeyThe potential link between PML NBs and ICP0 in regulating lytic and latent infection of HSV-1An Adenovirus DNA Replication Factor, but Not Incoming Genome Complexes, Targets PML Nuclear Bodies.The Role of Nuclear Antiviral Factors against Invading DNA Viruses: The Immediate Fate of Incoming Viral GenomesMutual inhibition between Kaposi's sarcoma-associated herpesvirus and Epstein-Barr virus lytic replication initiators in dually-infected primary effusion lymphomaKaposi's sarcoma-associated herpesvirus K-Rta exhibits SUMO-targeting ubiquitin ligase (STUbL) like activity and is essential for viral reactivationGenome-wide screen of three herpesviruses for protein subcellular localization and alteration of PML nuclear bodiesEpstein-Barr virus immediate-early protein Zta co-opts mitochondrial single-stranded DNA binding protein to promote viral and inhibit mitochondrial DNA replication.A role for cytoplasmic PML in cellular resistance to viral infectionEpstein-Barr nuclear antigen 1 contributes to nasopharyngeal carcinoma through disruption of PML nuclear bodies.Architecture of replication compartments formed during Epstein-Barr virus lytic replication.The Kaposi's sarcoma-associated herpesvirus K8 protein interacts with CREB-binding protein (CBP) and represses CBP-mediated transcription.Accumulation and intranuclear distribution of unintegrated human immunodeficiency virus type 1 DNA.Nucleoprotein structure of immediate-early promoters Zp and Rp and of oriLyt of latent Epstein-Barr virus genomesDNA viruses and viral proteins that interact with PML nuclear bodies.Postinternalization inhibition of adenovirus gene expression and infectious virus production in human T-cell linesFunctional reorganization of promyelocytic leukemia nuclear bodies during BK virus infection.Efficient induction of nuclear aggresomes by specific single missense mutations in the DNA-binding domain of a viral AP-1 homolog.Determination of minimum herpes simplex virus type 1 components necessary to localize transcriptionally active DNA to ND10.Nuclear domain 10 of the viral aspect.Cell cycle arrest by Kaposi's sarcoma-associated herpesvirus replication-associated protein is mediated at both the transcriptional and posttranslational levels by binding to CCAAT/enhancer-binding protein alpha and p21(CIP-1)Assembly of Epstein-Barr Virus Capsid in Promyelocytic Leukemia Nuclear Bodies.Human cytomegalovirus persistence.Epstein-Barr virus protein EB2 stimulates cytoplasmic mRNA accumulation by counteracting the deleterious effects of SRp20 on viral mRNAsEmerging Cellular Functions of Cytoplasmic PML.Epstein-Barr viral productive amplification reprograms nuclear architecture, DNA replication, and histone deposition.Interplay between Herpesvirus Infection and Host Defense by PML Nuclear BodiesDNA virus replication compartments.Rabies virus P and small P products interact directly with PML and reorganize PML nuclear bodies.Stimulation of the Replication of ICP0-Null Mutant Herpes Simplex Virus 1 and pp71-Deficient Human Cytomegalovirus by Epstein-Barr Virus Tegument Protein BNRF1.Components of promyelocytic leukemia nuclear bodies (ND10) act cooperatively to repress herpesvirus infection.Multivalent sequence recognition by Epstein-Barr virus Zta requires cysteine 171 and an extension of the canonical B-ZIP domain.Arsenic mediated disruption of promyelocytic leukemia protein nuclear bodies induces ganciclovir susceptibility in Epstein-Barr positive epithelial cells.Identification of acidic and aromatic residues in the Zta activation domain essential for Epstein-Barr virus reactivation
P2860
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P2860
Lytic but not latent replication of epstein-barr virus is associated with PML and induces sequential release of nuclear domain 10 proteins.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Lytic but not latent replicati ...... of nuclear domain 10 proteins.
@en
Lytic but not latent replicati ...... of nuclear domain 10 proteins.
@nl
type
label
Lytic but not latent replicati ...... of nuclear domain 10 proteins.
@en
Lytic but not latent replicati ...... of nuclear domain 10 proteins.
@nl
prefLabel
Lytic but not latent replicati ...... of nuclear domain 10 proteins.
@en
Lytic but not latent replicati ...... of nuclear domain 10 proteins.
@nl
P2093
P2860
P1433
P1476
Lytic but not latent replicati ...... of nuclear domain 10 proteins.
@en
P2093
P2860
P304
11800-11810
P356
10.1128/JVI.74.24.11800-11810.2000
P407
P577
2000-12-01T00:00:00Z