Disintegration of colonic epithelial tight junction in uremia: a likely cause of CKD-associated inflammation. - Wikidata - awgldk - TriplyDB

Disintegration of colonic epithelial tight junction in uremia: a likely cause of CKD-associated inflammation.

Disintegration of colonic epithelial tight junction in uremia: a likely cause of CKD-associated inflammation.

http://www.wikidata.org/entity/Q39598955

about

Iatrogenic Iron Overload in Dialysis Patients at the Beginning of the 21st Century Claudins in intestines: Distribution and functional significance in health and diseases The Gut as a Source of Inflammation in Chronic Kidney Disease Protein- and diabetes-induced glomerular hyperfiltration: role of glucagon, vasopressin, and urea High Fat High Cholesterol Diet (Western Diet) Aggravates Atherosclerosis, Hyperglycemia and Renal Failure in Nephrectomized LDL Receptor Knockout Mice: Role of Intestine Derived Lipopolysaccharide Role of the Gut Microbiome in Uremia: A Potential Therapeutic Target.Expansion of urease- and uricase-containing, indole- and p-cresol-forming and contraction of short-chain fatty acid-producing intestinal microbiota in ESRD.Galacto-oligosaccharides attenuate renal injury with microbiota modification The colon: an overlooked site for therapeutics in dialysis patients.High amylose resistant starch diet ameliorates oxidative stress, inflammation, and progression of chronic kidney disease Role of altered intestinal microbiota in systemic inflammation and cardiovascular disease in chronic kidney disease.Gut bacterial translocation may aggravate microinflammation in hemodialysis patients.Alterations of intestinal barrier and microbiota in chronic kidney disease.Alterations of intercellular junctions in peritoneal mesothelial cells from patients undergoing dialysis: effect of retinoic Acid.Oral Administration of P. gingivalis Induces Dysbiosis of Gut Microbiota and Impaired Barrier Function Leading to Dissemination of Enterobacteria to the Liver Gut microbiome in chronic kidney disease.The Influence of CKD on Colonic Microbial Metabolism.Increased Hepato-Splanchnic Vasoconstriction in Diabetics during Regular Hemodialysis Associations of Soluble CD14 and Endotoxin with Mortality, Cardiovascular Disease, and Progression of Kidney Disease among Patients with CKD.Increased urothelial paracellular transport promotes cystitis.Targeting the transcription factor Nrf2 to ameliorate oxidative stress and inflammation in chronic kidney disease Resistant starch alters gut microbiome and metabolomic profiles concurrent with amelioration of chronic kidney disease in rats Role of urea in intestinal barrier dysfunction and disruption of epithelial tight junction in chronic kidney disease.Uremic plasma impairs barrier function and depletes the tight junction protein constituents of intestinal epithelium.CKD impairs barrier function and alters microbial flora of the intestine: a major link to inflammation and uremic toxicity.From the gastrointestinal tract (GIT) to the kidneys: live bacterial cultures (probiotics) mediating reductions of uremic toxin levels via free radical signaling.Protein Nutrition and Malnutrition in CKD and ESRD.Effects of hemodiafiltration on uremic inflammation.Pharmacokinetic considerations in chronic kidney disease and patients requiring dialysis.Curcumin and chronic kidney disease (CKD): major mode of action through stimulating endogenous intestinal alkaline phosphatase.The intestine and the kidneys: a bad marriage can be hazardous.Altered intestinal microbial flora and impaired epithelial barrier structure and function in CKD: the nature, mechanisms, consequences and potential treatment.The nature, consequences, and management of neurological disorders in chronic kidney disease.Uremia induces adipose tissue inflammation and muscle mitochondrial dysfunction.Urea and chronic kidney disease: the comeback of the century? (in uraemia research).Oral iron supplementation: Potential implications for the gut microbiome and metabolome in patients with CKD.The Cerebrovascular-Chronic Kidney Disease Connection: Perspectives and Mechanisms.Gut microbiome in chronic kidney disease: challenges and opportunities.Gut microbiota in renal physiology: focus on short-chain fatty acids and their receptors.Impact of Dietary Fibers on Nutrient Management and Detoxification Organs: Gut, Liver, and Kidneys.

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P2860

Disintegration of colonic epithelial tight junction in uremia: a likely cause of CKD-associated inflammation.

http://www.wikidata.org/entity/Q39598955

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年论文

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Disintegration of colonic epit ...... f CKD-associated inflammation.

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Disintegration of colonic epit ...... f CKD-associated inflammation.

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Disintegration of colonic epit ...... f CKD-associated inflammation.

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Disintegration of colonic epit ...... f CKD-associated inflammation.

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Disintegration of colonic epit ...... f CKD-associated inflammation.

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Disintegration of colonic epit ...... f CKD-associated inflammation.

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Nephrology Dialysis Transplantation

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Ardeshir Rahimi

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Hyder Said

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Jun Yuan

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Nosratola D Vaziri

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Veedamali S Subramanian

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Zhenmin Ni

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Intestinal mucosal barrier function in health and disease

Molecular physiology and pathophysiology of tight junctions. IV. Regulation of tight junctions by extracellular stimuli: nutrients, cytokines, and immune cells.

Impaired intestinal barrier function measured by differently sized polyethylene glycols in patients with chronic renal failure.

The elephant in uremia: oxidant stress as a unifying concept of cardiovascular disease in uremia.

Occludin regulates macromolecule flux across the intestinal epithelial tight junction barrier

Oxidative stress in uremia: nature, mechanisms, and potential consequences.

Role of epithelial cells in initiation and propagation of intestinal inflammation. Eliminating the static: tight junction dynamics exposed.

Inflammatory bowel disease and the apical junctional complex.

Endotoxemia is related to systemic inflammation and atherosclerosis in peritoneal dialysis patients.

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