Four danger response programs determine glomerular and tubulointerstitial kidney pathology: clotting, inflammation, epithelial and mesenchymal healing.
about
The Immune System in Tissue Environments Regaining Homeostasis after Injury: Is "Inflammation" Always Inflammation?Induction monotherapy with sirolimus has selected beneficial effects on glomerular and tubulointersititial injury in nephrotoxic serum nephritis.Pattern-recognition receptor signaling regulator mRNA expression in humans and mice, and in transient inflammation or progressive fibrosisToll-like receptor 4-induced IL-22 accelerates kidney regeneration.Tissues use resident dendritic cells and macrophages to maintain homeostasis and to regain homeostasis upon tissue injury: the immunoregulatory role of changing tissue environments.Immunopathology of lupus nephritis.Beyond tissue injury-damage-associated molecular patterns, toll-like receptors, and inflammasomes also drive regeneration and fibrosis.Histones and Neutrophil Extracellular Traps Enhance Tubular Necrosis and Remote Organ Injury in Ischemic AKI.The effect of zinc on healing of renal damage in rats.Innate versus adaptive immunity in kidney immunopathology.FTY720 attenuates tubulointerstitial inflammation and fibrosis in subtotally nephrectomized rats.Molecular mechanisms of crystal-related kidney inflammation and injury. Implications for cholesterol embolism, crystalline nephropathies and kidney stone disease.Nrf2 signalling promotes ex vivo tubular epithelial cell survival and regeneration via murine double minute (MDM)-2.Podocyte loss involves MDM2-driven mitotic catastrophe.
P2860
Q28066426-E3B1841E-A69A-4719-87EF-0056882975DFQ33952749-173BA99F-1782-4845-A7CE-840668B22BA7Q37225102-FE197B02-82E3-4FE4-B671-88E86DDBB99AQ37733573-05C32CC0-8E0D-4AF4-B525-E6C8DD8217BFQ38068399-978D6FDC-8DCF-452A-8AB7-B99756FCDBF9Q38176819-7361B84C-DBF5-4344-A615-8627A96BFB32Q38206875-42DA8A8D-57E7-480D-A590-645BCECD48CEQ39028315-570D7203-FA59-43BC-A266-151B190146C5Q41761706-8FBD411E-EDF5-4A57-BE41-7427E5224488Q43099963-B9EFF2CE-970C-4936-BB12-CCA4A71245AEQ44062977-8F867A31-4F95-4E2A-BFA3-DD7198072F00Q44855203-7E73375A-5227-445E-BD8B-3126DB86EA8FQ45729835-5C42817B-40F7-4132-B8E4-EFAEE1320A87Q46048434-2E16551E-C890-4589-B8CD-2D74368FF534
P2860
Four danger response programs determine glomerular and tubulointerstitial kidney pathology: clotting, inflammation, epithelial and mesenchymal healing.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Four danger response programs ...... elial and mesenchymal healing.
@en
Four danger response programs ...... elial and mesenchymal healing.
@nl
type
label
Four danger response programs ...... elial and mesenchymal healing.
@en
Four danger response programs ...... elial and mesenchymal healing.
@nl
altLabel
Four danger response programs ...... ointerstitial kidney pathology
@en
prefLabel
Four danger response programs ...... elial and mesenchymal healing.
@en
Four danger response programs ...... elial and mesenchymal healing.
@nl
P2860
P356
P1433
P1476
Four danger response programs ...... elial and mesenchymal healing.
@en
P2093
Hans-Joachim Anders
P2860
P356
10.4161/ORG.20342
P577
2012-04-01T00:00:00Z