Coexpression of Oct4 and Nanog enhances malignancy in lung adenocarcinoma by inducing cancer stem cell-like properties and epithelial-mesenchymal transdifferentiation.
about
The PRKCI and SOX2 oncogenes are coamplified and cooperate to activate Hedgehog signaling in lung squamous cell carcinomaMolecular biology of lung cancer: clinical implicationsYin Yang 1 is associated with cancer stem cell transcription factors (SOX2, OCT4, BMI1) and clinical implicationCancer Stem Cell Hierarchy in Glioblastoma MultiformeEpigenetic modulators, modifiers and mediators in cancer aetiology and progressionRedox Modulating NRF2: A Potential Mediator of Cancer Stem Cell ResistanceCommon stemness regulators of embryonic and cancer stem cellsThe genomic stability of induced pluripotent stem cellsLinks between cancer stem cells and epithelial-mesenchymal transitionEMT and stem cell-like properties associated with HIF-2α are involved in arsenite-induced transformation of human bronchial epithelial cellsInsights into the Nanog gene: A propeller for stemness in primitive stem cellsIn vitro models of cancer stem cells and clinical applicationsThe role of NANOG transcriptional factor in the development of malignant phenotype of cancer cellsConcise Review: NANOG in Cancer Stem Cells and Tumor Development: An Update and Outstanding QuestionsInhibition of stearoyl-coA desaturase selectively eliminates tumorigenic Nanog-positive cells: improving the safety of iPS cell transplantation to myocardiumFormation of Tumorspheres with Increased Stemness without External Mitogens in a Lung Cancer ModelElevated expression of Nrf2 mediates multidrug resistance in CD133(+) head and neck squamous cell carcinoma stem cellsNew approaches of PARP-1 inhibitors in human lung cancer cells and cancer stem-like cells by some selected anthraquinone-derived small moleculesNanog1 in NTERA-2 and recombinant NanogP8 from somatic cancer cells adopt multiple protein conformations and migrate at multiple M.W speciesA novel anticancer agent SNG1153 inhibits growth of lung cancer stem/progenitor cellsSnail contributes to the maintenance of stem cell-like phenotype cells in human pancreatic cancer.Phosphorylation of Nanog is essential to regulate Bmi1 and promote tumorigenesis.Tumour-initiating capacity is independent of epithelial-mesenchymal transition status in breast cancer cell lines.Increased expression of the pluripotency markers sex-determining region Y-box 2 and Nanog homeobox in ovarian endometriosis.The meaning of PIWI proteins in cancer development.Immunohistochemical analysis of NANOG expression and epithelial-mesenchymal transition in pulmonary sarcomatoid carcinoma.HIF1α regulates single differentiated glioma cell dedifferentiation to stem-like cell phenotypes with high tumorigenic potential under hypoxiaSustained activation of DNA damage response in irradiated apoptosis-resistant cells induces reversible senescence associated with mTOR downregulation and expression of stem cell markersInfluence of adriamycin on changes in Nanog, Oct-4, Sox2, ARID1 and Wnt5b expression in liver cancer stem cellsSOX2 expression is an early event in a murine model of EGFR mutant lung cancer and promotes proliferation of a subset of EGFR mutant lung adenocarcinoma cell lines.Emerging role of nanog in tumorigenesis and cancer stem cells.Snail regulates Nanog status during the epithelial-mesenchymal transition via the Smad1/Akt/GSK3β signaling pathway in non-small-cell lung cancer.Snail1 induces epithelial-to-mesenchymal transition and tumor initiating stem cell characteristicsTranscription factors related to epithelial mesenchymal transition in tumor center and margin in invasive lung adenocarcinoma.Transitions between epithelial and mesenchymal states during cell fate conversions.Computational analysis of expression of human embryonic stem cell-associated signatures in tumors.CD133/Src axis mediates tumor initiating property and epithelial-mesenchymal transition of head and neck cancerThe side population in human lung cancer cell line NCI-H460 is enriched in stem-like cancer cells.Properties of resistant cells generated from lung cancer cell lines treated with EGFR inhibitors.Matrix metalloproteinase-10 is required for lung cancer stem cell maintenance, tumor initiation and metastatic potential.
P2860
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P2860
Coexpression of Oct4 and Nanog enhances malignancy in lung adenocarcinoma by inducing cancer stem cell-like properties and epithelial-mesenchymal transdifferentiation.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Coexpression of Oct4 and Nanog ...... enchymal transdifferentiation.
@en
Coexpression of Oct4 and Nanog ...... enchymal transdifferentiation.
@nl
type
label
Coexpression of Oct4 and Nanog ...... enchymal transdifferentiation.
@en
Coexpression of Oct4 and Nanog ...... enchymal transdifferentiation.
@nl
prefLabel
Coexpression of Oct4 and Nanog ...... enchymal transdifferentiation.
@en
Coexpression of Oct4 and Nanog ...... enchymal transdifferentiation.
@nl
P2093
P1433
P1476
Coexpression of Oct4 and Nanog ...... senchymal transdifferentiation
@en
P2093
Cheng-Wen Wu
Chi-Jen Chen
Chun-Fu Hong
Han-Sui Hsu
Hsin-Tzu Chang
Mong-Lien Wang
Shih-Hwa Chiou
Tzu-Wei Lin
Wang-Ju Hsieh
Ying-Shan Chen
P304
10433-10444
P356
10.1158/0008-5472.CAN-10-2638
P407
P50
P577
2010-12-01T00:00:00Z