Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
about
Modulation of NK cell function by genetically coupled C-type lectin-like receptor/ligand pairs encoded in the human natural killer gene complexStructure of NKp65 bound to its keratinocyte ligand reveals basis for genetically linked recognition in natural killer gene complexRecognition of tumors by the innate immune system and natural killer cells.Suppression of natural killer cells by sorafenib contributes to prometastatic effects in hepatocellular carcinomaAnti-inflammatory activity of IgG1 mediated by Fc galactosylation and association of FcγRIIB and dectin-1.HER2-specific immunoligands engaging NKp30 or NKp80 trigger NK-cell-mediated lysis of tumor cells and enhance antibody-dependent cell-mediated cytotoxicityControlling natural killer cell responses: integration of signals for activation and inhibition.Human dNK cell function is differentially regulated by extrinsic cellular engagement and intrinsic activating receptors in first and second trimester pregnancyVis-à-vis in the NKC: genetically linked natural killer cell receptor/ligand pairs in the natural killer gene complex (NKC).Novel platelet activation receptor CLEC-2: from discovery to prospects.Non-catalytic tyrosine-phosphorylated receptors.NK cells and poxvirus infection.Regulation of self-ligands for activating natural killer cell receptors.The Activating C-type Lectin-like Receptor NKp65 Signals through a Hemi-immunoreceptor Tyrosine-based Activation Motif (hemITAM) and Spleen Tyrosine Kinase (Syk).Attenuated natural killer (NK) cell activation through C-type lectin-like receptor NKp80 is due to an anomalous hemi-immunoreceptor tyrosine-based activation motif (HemITAM) with impaired Syk kinase recruitment capacity.Differential Requirements for Src-Family Kinases in SYK or ZAP70-Mediated SLP-76 Phosphorylation in Lymphocytes.Protein Kinase C-θ (PKC-θ) in Natural Killer Cell Function and Anti-Tumor ImmunityComparison of Host Gene Expression Profiles in Spleen Tissues of Genetically Susceptible and Resistant Mice during ECTV Infection.Transferrin‘ activation: Bonding with transferrin receptors tunes KLRG1 function
P2860
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P2860
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
@en
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
@nl
type
label
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
@en
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
@nl
prefLabel
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
@en
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
@nl
P356
P1476
Cutting edge: NKp80 uses an atypical hemi-ITAM to trigger NK cytotoxicity.
@en
P2093
Kevin M Dennehy
Sascha N Klimosch
P304
P356
10.4049/JIMMUNOL.0904117
P407
P577
2010-12-13T00:00:00Z