CagA/cytotoxic strains of Helicobacter pylori and interleukin-8 in gastric epithelial cell lines
about
Effect of Helicobacter pylori on gastric epithelial cellsHelicobacter pylori infection potentiates aspirin induced gastric mucosal injury in Mongolian gerbilsHelicobacter pylori infection induces oxidative stress and programmed cell death in human gastric epithelial cells.Iron deficiency accelerates Helicobacter pylori-induced carcinogenesis in rodents and humansIncreased zinc finger protein zFOC1 transcripts in gastric cancer compared with normal gastric tissue.Helicobacter pylori stimulates dendritic cells to induce interleukin-17 expression from CD4+ T lymphocytes.Gastric mucosal secretion of interleukin-10: relations to histopathology, Helicobacter pylori status, and tumour necrosis factor-alpha secretion.Adherence and invasion of mouse-adapted H pylori in different epithelial cell lines.Host cell responses to genotypically similar Helicobacter pylori isolates from United States and Japan.Consensus and variable region PCR analysis of Helicobacter pylori 3' region of cagA gene in isolates from individuals with or without peptic ulcerConcurrent proinflammatory and apoptotic activity of a Helicobacter pylori protein (HP986) points to its role in chronic persistence.Anti-CagA immunoglobulin G responses correlate with interleukin-8 induction in human gastric mucosal biopsy cultureDetermination of Helicobacter pylori virulence by simple gene analysis of the cag pathogenicity islandHelicobacter pylori-associated gastritis in mice is host and strain specific.Multiple genes in the left half of the cag pathogenicity island of Helicobacter pylori are required for tyrosine kinase-dependent transcription of interleukin-8 in gastric epithelial cells.In vivo distribution of Helicobacter felis in the gastric mucus of the mouse: experimental method and results.Induction of monocyte chemoattractant protein 1 by Helicobacter pylori involves NF-kappaB.Differential gene expression from two transcriptional units in the cag pathogenicity island of Helicobacter pylori.Effects of nonsteroidal anti-inflammatory drugs on Helicobacter pylori-infected gastric mucosae of mice: apoptosis, cell proliferation, and inflammatory activity.Helicobacter pylori CagA triggers expression of the bactericidal lectin REG3γ via gastric STAT3 activationResponses of endoscopy patients in Ladakh, India, to Helicobacter pylori whole-cell and Cag A antigensIV. Helicobacter pylori strain-specific activation of signal transduction cascades related to gastric inflammation.Helicobacter pylori exploits cholesterol-rich microdomains for induction of NF-kappaB-dependent responses and peptidoglycan delivery in epithelial cellspathogenesis of Helicobacter pylori-induced gastric inflammation.Association of cytotoxin production and neutrophil activation by strains of Helicobacter pylori isolated from patients with peptic ulceration and chronic gastritisThe relation between gastric vitamin C concentrations, mucosal histology, and CagA seropositivity in the human stomach.Helicobacter pylori with a truncated lipopolysaccharide O chain fails to induce gastritis in SCID mice injected with splenocytes from wild-type C57BL/6J mice.Gastric epithelial expression of IL-12 cytokine family in Helicobacter pylori infection in human: is it head or tail of the coin?Intestinal epithelial responses to enteric pathogens: effects on the tight junction barrier, ion transport, and inflammation.Major virulence factors, VacA and CagA, are commonly positive in Helicobacter pylori isolates in JapanStructure of cag pathogenicity island in Japanese Helicobacter pylori isolates.Interleukin-8 response of gastric epithelial cell lines to Helicobacter pylori stimulation in vitroExpression of the human cell surface glycoconjugates Lewis x and Lewis y by Helicobacter pylori isolates is related to cagA status.Functional analysis of the cag pathogenicity island in Helicobacter pylori isolates from patients with gastritis, peptic ulcer, and gastric cancer.Helicobacter pylori babA2, cagA, and s1 vacA genes work synergistically in causing intestinal metaplasia.Induction of various cytokines and development of severe mucosal inflammation by cagA gene positive Helicobacter pylori strains.Helicobacter pylori infection upregulates interleukin-18 production from gastric epithelial cells.Sequence anomalies in the Cag7 gene of the Helicobacter pylori pathogenicity island.Alterations in gastric mucin synthesis by Helicobacter pylori.Serologic detection of infection with cagA+ Helicobacter pylori strains
P2860
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P2860
CagA/cytotoxic strains of Helicobacter pylori and interleukin-8 in gastric epithelial cell lines
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
CagA/cytotoxic strains of Heli ...... gastric epithelial cell lines
@en
CagA/cytotoxic strains of Heli ...... gastric epithelial cell lines.
@nl
type
label
CagA/cytotoxic strains of Heli ...... gastric epithelial cell lines
@en
CagA/cytotoxic strains of Heli ...... gastric epithelial cell lines.
@nl
prefLabel
CagA/cytotoxic strains of Heli ...... gastric epithelial cell lines
@en
CagA/cytotoxic strains of Heli ...... gastric epithelial cell lines.
@nl
P2093
P2860
P921
P356
P1476
CagA/cytotoxic strains of Heli ...... gastric epithelial cell lines
@en
P2093
P2860
P304
P356
10.1136/JCP.47.10.945
P407
P577
1994-10-01T00:00:00Z