Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription.

Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription.

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http://www.wikidata.org/entity/Q39642654
Comparison of Active Drug Concentrations in the Pulmonary Epithelial Lining Fluid and Interstitial Fluid of Calves Injected with Enrofloxacin, Florfenicol, Ceftiofur, or TulathromycinTulathromycin exerts proresolving effects in bovine neutrophils by inhibiting phospholipases and altering leukotriene B4, prostaglandin E2, and lipoxin A4 productionIL-32γ delays spontaneous apoptosis of human neutrophils through MCL-1, regulated primarily by the p38 MAPK pathway.Flavones induce neutrophil apoptosis by down-regulation of Mcl-1 via a proteasomal-dependent pathwayPharmacokinetics of macrolides in foals.Efficacy of Tulathromycin for the Treatment of Foals with Mild to Moderate Bronchopneumonia.Pharmacokinetics and pharmacodynamics of gamithromycin in pulmonary epithelial lining fluid in naturally occurring bovine respiratory disease in multisource commingled feedlot cattle.Direct and indirect anti-inflammatory effects of tulathromycin in bovine macrophages: inhibition of CXCL-8 secretion, induction of apoptosis, and promotion of efferocytosis.Methaphylactic effect of tulathromycin treatment on rumen fluid parameters in feedlot beef cattle.The bacteriocin sublancin attenuates intestinal injury in young mice infected with Staphylococcus aureus.Comparison of direct sampling and brochoalveolar lavage for determining active drug concentrations in the pulmonary epithelial lining fluid of calves injected with enrofloxacin or tilmicosin.Anti-Inflammatory Benefits of Antibiotics: Tylvalosin Induces Apoptosis of Porcine Neutrophils and Macrophages, Promotes Efferocytosis, and Inhibits Pro-Inflammatory CXCL-8, IL1α, and LTB4 Production, While Inducing the Release of Pro-Resolving Lipo
P2860

Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription.

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http://www.wikidata.org/entity/Q39642654
description
2010 nî lūn-bûn
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2010年の論文
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2010年論文
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2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
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2010年論文
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2010年论文
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2010年论文
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2010年论文
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name
Anti-Inflammatory benefits of ...... aling and CXCL8 transcription.
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Anti-Inflammatory benefits of ...... aling and CXCL8 transcription.
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type
label
Anti-Inflammatory benefits of ...... aling and CXCL8 transcription.
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Anti-Inflammatory benefits of ...... aling and CXCL8 transcription.
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prefLabel
Anti-Inflammatory benefits of ...... aling and CXCL8 transcription.
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Anti-Inflammatory benefits of ...... aling and CXCL8 transcription.
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Anti-Inflammatory benefits of ...... aling and CXCL8 transcription.
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P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P921
P932