Deletion of the pH sensor GPR4 decreases renal acid excretion.
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Collecting duct intercalated cell function and regulationAcidosis activation of the proton-sensing GPR4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysisActivation of GPR4 by acidosis increases endothelial cell adhesion through the cAMP/Epac pathwayPHYSIOLOGY. Regulation of breathing by CO₂ requires the proton-activated receptor GPR4 in retrotrapezoid nucleus neurons.Autophagic clearance of mitochondria in the kidney copes with metabolic acidosis.pH-dependent regulation of the α-subunit of H+-K+-ATPase (HKα2).Ischemia/reperfusion-induced CHOP expression promotes apoptosis and impairs renal function recovery: the role of acidosis and GPR4Molecular mechanisms of acid-base sensing by the kidneyThe GPCR OGR1 (GPR68) mediates diverse signalling and contraction of airway smooth muscle in response to small reductions in extracellular pHSDF1 induction by acidosis from principal cells regulates intercalated cell subtype distribution.Increased bone density in mice lacking the proton receptor OGR1.Effect of acute acid-base disturbances on ErbB1/2 tyrosine phosphorylation in rabbit renal proximal tubules.Bicarbonate-sensing soluble adenylyl cyclase is present in the cell cytoplasm and nucleus of multiple shark tissues.Differentiation of intercalated cells in the kidney.Cell biology of the intercalated cell in the kidney.Acidic tumor microenvironment and pH-sensing G protein-coupled receptors.Glucagon actions on the kidney revisited: possible role in potassium homeostasis.Unsung renal receptors: orphan G-protein-coupled receptors play essential roles in renal development and homeostasis.Comparative study of 3D morphology and functions on genetically engineered mouse melanoma cells.Role of Receptor Protein Tyrosine Phosphatase γ in Sensing Extracellular CO2 and HCO3.Acid stress increases gene expression of proinflammatory cytokines in Madin-Darby canine kidney cells.Deletion of proton-sensing receptor GPR4 associates with lower blood pressure and lower binding of angiotensin II receptor in SFO.Identifying the localization and exploring a functional role for Gprc5c in the kidney.The proton-activated receptor GPR4 modulates intestinal inflammation.GPR4 knockout improves renal ischemia-reperfusion injury and inhibits apoptosis via suppressing the expression of CHOP.Manganese and cobalt activate zebrafish ovarian cancer G-protein-coupled receptor 1 but not GPR4.GPR4 deficiency alleviates intestinal inflammation in a mouse model of acute experimental colitis.Soluble adenylyl cyclase is an acid-base sensor in epithelial base-secreting cells.Kidney Response to the Spectrum of Diet-Induced Acid Stress.Acid retention accompanies reduced GFR in humans and increases plasma levels of endothelin and aldosteroneAcidosis promotes cell apoptosis through the G protein-coupled receptor 4/CCAAT/enhancer-binding protein homologous protein pathway
P2860
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P2860
Deletion of the pH sensor GPR4 decreases renal acid excretion.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Deletion of the pH sensor GPR4 decreases renal acid excretion.
@en
Deletion of the pH sensor GPR4 decreases renal acid excretion.
@nl
type
label
Deletion of the pH sensor GPR4 decreases renal acid excretion.
@en
Deletion of the pH sensor GPR4 decreases renal acid excretion.
@nl
prefLabel
Deletion of the pH sensor GPR4 decreases renal acid excretion.
@en
Deletion of the pH sensor GPR4 decreases renal acid excretion.
@nl
P2093
P2860
P356
P1476
Deletion of the pH sensor GPR4 decreases renal acid excretion.
@en
P2093
Brian C Tiegs
Dennis W McGraw
Lois J Arend
Raymond B Penn
Snezana Petrovic
Xuming Sun
P2860
P304
P356
10.1681/ASN.2009050477
P50
P577
2010-08-26T00:00:00Z