G Protein-Coupled Receptor-G-Protein βγ-Subunit Signaling Mediates Renal Dysfunction and Fibrosis in Heart Failure.
about
Targeting GPCR-Gβγ-GRK2 signaling as a novel strategy for treating cardiorenal pathologies.Heart Failure Increases the Risk of Adverse Renal Outcomes in Patients With Normal Kidney Function.Structural and pharmacological basis for the induction of mitochondrial biogenesis by formoterol but not clenbuterol.β-Arrestin-1 deficiency ameliorates renal interstitial fibrosis by blocking Wnt1/β-catenin signaling in mice.Pharmacological and Activated Fibroblast Targeting of Gβγ-GRK2 After Myocardial Ischemia Attenuates Heart Failure Progression.Increased susceptibility to structural acute kidney injury in a mouse model of presymptomatic cardiomyopathy.
P2860
G Protein-Coupled Receptor-G-Protein βγ-Subunit Signaling Mediates Renal Dysfunction and Fibrosis in Heart Failure.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
G Protein-Coupled Receptor-G-P ...... and Fibrosis in Heart Failure.
@en
G Protein-Coupled Receptor-G-P ...... and Fibrosis in Heart Failure.
@nl
type
label
G Protein-Coupled Receptor-G-P ...... and Fibrosis in Heart Failure.
@en
G Protein-Coupled Receptor-G-P ...... and Fibrosis in Heart Failure.
@nl
prefLabel
G Protein-Coupled Receptor-G-P ...... and Fibrosis in Heart Failure.
@en
G Protein-Coupled Receptor-G-P ...... and Fibrosis in Heart Failure.
@nl
P2093
P2860
P356
P1476
G Protein-Coupled Receptor-G-P ...... and Fibrosis in Heart Failure
@en
P2093
Burns C Blaxall
Fadia A Kamal
Joshua G Travers
P2860
P304
P356
10.1681/ASN.2015080852
P577
2016-06-13T00:00:00Z