ROS accumulation and IGF-IR inhibition contribute to fenofibrate/PPARalpha -mediated inhibition of glioma cell motility in vitro.
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Fenofibrate induces apoptosis of triple-negative breast cancer cells via activation of NF-κB pathwayCharacterization of fenofibrate-mediated anti-proliferative pro-apoptotic effects on high-grade gliomas and anti-invasive effects on glioma stem cells.Molecular mechanisms of fenofibrate-induced metabolic catastrophe and glioblastoma cell death.Anti-tumoral effects of miR-3189-3p in glioblastomaInhibition of intracerebral glioblastoma growth by targeting the insulin-like growth factor 1 receptor involves different context-dependent mechanisms.Activation of peroxisome proliferator-activated receptor α (PPARα) suppresses hypoxia-inducible factor-1α (HIF-1α) signaling in cancer cells.PPARα modulates gene expression profiles of mitochondrial energy metabolism in oral tumorigenesisMetformin stimulates IGFBP-2 gene expression through PPARalpha in diabetic states.Peroxisome proliferator activated receptor α ligands as anticancer drugs targeting mitochondrial metabolismFenofibrate Suppresses Oral Tumorigenesis via Reprogramming Metabolic Processes: Potential Drug Repurposing for Oral Cancer.Targeting 3-phosphoinositide-dependent protein kinase 1 by N-acetyl-cysteine through activation of peroxisome proliferators activated receptor alpha in human lung cancer cells, the role of p53 and p65.Behavioral paradigms to evaluate PPAR modulation in animal models of brain injury.Cyclin D1 represses peroxisome proliferator-activated receptor alpha and inhibits fatty acid oxidation.PPAR-γ Agonists As Antineoplastic Agents in Cancers with Dysregulated IGF Axis.Anticancer activity of salicin and fenofibrate.Peroxisome proliferator-activated receptor α (PPARα) contributes to control of melanogenesis in B16 F10 melanoma cells.Glioblastoma-synthesized G-CSF and GM-CSF contribute to growth and immunosuppression: Potential therapeutic benefit from dapsone, fenofibrate, and ribavirin.Fenofibrate subcellular distribution as a rationale for the intracranial delivery through biodegradable carrier.Involvement of ROS-alpha v beta 3 integrin-FAK/Pyk2 in the inhibitory effect of melatonin on U251 glioma cell migration and invasion under hypoxia.Fenofibrate induces G0/G1 phase arrest by modulating the PPARα/FoxO1/p27 kip pathway in human glioblastoma cells.Fenofibrate enhances barrier function of endothelial continuum within the metastatic niche of prostate cancer cells.A critical overview of long non-coding RNA in glioma etiology 2016: an update.Novel MEK inhibitor trametinib and other retinoblastoma gene (RB)-reactivating agents enhance efficacy of 5-fluorouracil on human colon cancer cells.Differential Effects of MicroRNAs on Glioblastoma Growth and Migration.Fenofibrate-induced nuclear translocation of FoxO3A triggers Bim-mediated apoptosis in glioblastoma cells in vitro.Regulation of IGFBP-2 expression during fasting.Fenofibrate Induces Ketone Body Production in Melanoma and Glioblastoma Cells.Fenofibrate triggers apoptosis of glioblastoma cells in vitro: new insights for therapy.SLC7A11 Overexpression in Glioblastoma Is Associated with Increased Cancer Stem Cell-Like Properties.Changes in mitochondrial morphology induced by calcium or rotenone in primary astrocytes occur predominantly through ros-mediated remodeling.Anticancer potential of aminomethylidene-diazinanes I. Synthesis of arylaminomethylidene of diazinetriones and its cytotoxic effects tested in glioblastoma cells.The transcription factor PPARα is overexpressed and is associated with a favourable prognosis in IDH-wildtype primary glioblastoma.Acute Ethanol Increases IGF-I-Induced Phosphorylation of ERKs by Enhancing Recruitment of p52-Shc to the Grb2/Shc Complex.DHA induces apoptosis of human malignant breast cancer tissues by the TLR-4/PPAR-α pathways.Hints on ATGL implications in cancer: beyond bioenergetic clues.Anticancer Properties of Fenofibrate: A Repurposing Use.
P2860
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P2860
ROS accumulation and IGF-IR inhibition contribute to fenofibrate/PPARalpha -mediated inhibition of glioma cell motility in vitro.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
ROS accumulation and IGF-IR in ...... glioma cell motility in vitro.
@en
ROS accumulation and IGF-IR in ...... glioma cell motility in vitro.
@nl
type
label
ROS accumulation and IGF-IR in ...... glioma cell motility in vitro.
@en
ROS accumulation and IGF-IR in ...... glioma cell motility in vitro.
@nl
prefLabel
ROS accumulation and IGF-IR in ...... glioma cell motility in vitro.
@en
ROS accumulation and IGF-IR in ...... glioma cell motility in vitro.
@nl
P2093
P2860
P50
P356
P1433
P1476
ROS accumulation and IGF-IR in ...... glioma cell motility in vitro.
@en
P2093
Ewa Wybieralska
Justyna Drukala
Katarzyna Urbanska
Krzysztof Reiss
P2860
P2888
P356
10.1186/1476-4598-9-159
P577
2010-06-22T00:00:00Z
P5875
P6179
1043322084