FGF-2 and Anosmin-1 are selectively expressed in different types of multiple sclerosis lesions.
about
The innate immune system in demyelinating diseasePlasma biomarkers discriminate clinical forms of multiple sclerosisFGF-2 released from degenerating neurons exerts microglial-induced neuroprotection via FGFR3-ERK signaling pathway.Integrated analysis of recurrent properties of cancer genes to identify novel driversAngiogenesis in multiple sclerosis and experimental autoimmune encephalomyelitis.The role of immune cells, glia and neurons in white and gray matter pathology in multiple sclerosis.Activation of endogenous neural stem cells for multiple sclerosis therapy.Protocol to isolate a large amount of functional oligodendrocyte precursor cells from the cerebral cortex of adult mice and humans.A new in vitro mouse oligodendrocyte precursor cell migration assay reveals a role for integrin-linked kinase in cell motilityFibroblast growth factor signaling in oligodendrocyte-lineage cells facilitates recovery of chronically demyelinated lesions but is redundant in acute lesions.Expression and Genetic Analysis of MicroRNAs Involved in Multiple Sclerosis.Modulation of neural stem/progenitor cell proliferation during experimental Herpes Simplex encephalitis is mediated by differential FGF-2 expression in the adult brainWhole-Genome Expression Analysis and Signal Pathway Screening of Synovium-Derived Mesenchymal Stromal Cells in Rheumatoid Arthritis.MicroRNAs as active players in the pathogenesis of multiple sclerosis.Signalling pathways that inhibit the capacity of precursor cells for myelin repair.Multiple sclerosis: does Nogo play a role?Regulation of oligodendrocyte precursor migration during development, in adulthood and in pathology.Olfactory system and demyelination.Fibroblast growth factor-2 signaling in neurogenesis and neurodegeneration.The effect of glia-glia interactions on oligodendrocyte precursor cell biology during development and in demyelinating diseasesAnosmin-1 over-expression increases adult neurogenesis in the subventricular zone and neuroblast migration to the olfactory bulb.Thyroid Hormone Potentially Benefits Multiple Sclerosis via Facilitating Remyelination.Dysregulated fibroblast growth factor (FGF) signaling in neurological and psychiatric disorders.Extracellular cues influencing oligodendrocyte differentiation and (re)myelinationThe role of growth factors as a therapeutic approach to demyelinating disease.Contribution of the oligodendrocyte lineage to CNS repair and neurodegenerative pathologies.Modulation of subventricular zone oligodendrogenesis: a role for hemopressin?IL-17-triggered downregulation of miR-497 results in high HIF-1α expression and consequent IL-1β and IL-6 production by astrocytes in EAE mice.Dietary cholesterol promotes repair of demyelinated lesions in the adult brain.Fibroblast growth factor signalling in multiple sclerosis: inhibition of myelination and induction of pro-inflammatory environment by FGF9.Anosmin-1 over-expression regulates oligodendrocyte precursor cell proliferation, migration and myelin sheath thickness.Strategies for repair of white matter: influence of osmolarity and microglia on proliferation and apoptosis of oligodendrocyte precursor cells in different basal culture media.The Role of SDF-1/CXCR4/CXCR7 in Neuronal Regeneration after Cerebral Ischemia.ERK1/2 signaling is essential for the chemoattraction exerted by human FGF2 and human anosmin-1 on newborn rat and mouse OPCs via FGFR1.Achievements and obstacles of remyelinating therapies in multiple sclerosis.Kallmann syndrome patient with gender dysphoria, multiple sclerosis, and thrombophilia.Oligodendroglial fibroblast growth factor receptor 1 gene targeting protects mice from experimental autoimmune encephalomyelitis through ERK/AKT phosphorylation.Intraventricular injection of FGF-2 promotes generation of oligodendrocyte-lineage cells in the postnatal and adult forebrain.Critical role of Ror2 receptor tyrosine kinase in regulating cell cycle progression of reactive astrocytes following brain injury.The cysteine-rich region and the whey acidic protein domain are essential for anosmin-1 biological functions.
P2860
Q24597958-4C4F88C6-FD25-405D-A0B1-09FF6D151F78Q28262774-364AD77D-995D-4FE7-8699-7EC231318CF9Q33616048-D6D5A2C4-AB9A-4DE9-96ED-44FD170A81A2Q33742981-2FD114FF-4728-493D-9DD4-F073CCB06A61Q34109028-30782290-E97C-4E43-A562-205E39B9F77DQ34468304-7B7A3950-369E-4D06-B975-CC7C1F48FAD3Q34986269-384E8019-DB03-4512-AD83-79DCCEE71910Q35058625-5F1A8F18-BBF5-46BE-B5BF-533B0580D7A7Q35910607-AE6EE639-77CE-423D-9BAD-4CF1C73D8FF9Q35946418-B182E186-3E50-431D-8E8D-482DCA17949FQ36790311-521D8F28-F731-418A-A2AC-99FCB031692CQ37107215-2CDE9DD1-FC45-4475-BAFE-C12CEDC4B6F2Q37239644-8ABD3E7C-CAED-4BB5-ABD4-F827EAF11C6DQ38064320-918E946A-DDF4-4CF9-B91F-69D63ABF8C8EQ38072291-AD662CEB-3774-4DA2-98E0-6FAEE7F11AD8Q38082999-EE14F910-01A9-4536-8C0D-5B3FA05BE794Q38108182-D36AABF1-5C15-45E8-9125-B13C45982600Q38125663-BFC33D6C-2F90-44BC-8441-B34C9208CE71Q38140658-2CEE4A4D-F126-4321-BC2F-349CAC16AABDQ38175970-AB083D7D-91F2-4AC8-B9F6-A4C87D72A04FQ38303609-42BD2458-0739-48DB-91BA-2E29A8F14FD8Q38560936-0938FA9D-FA86-4F15-A0ED-E1B198E2619BQ38603624-0446C0E5-F2F5-4E69-943A-753BDC9B99A0Q38789322-4EB2C7DD-1570-46AD-AED8-15ABAD6DCA18Q38789326-394DBBA3-3CC6-402A-ADEE-142D9C7AC466Q38815878-CE6A705F-F2C6-47E6-BC9F-26A8B4CE5591Q39242960-D03593F4-018B-4C09-AF25-5321616F2E40Q40231889-05DA93C6-82B3-4312-B5CD-E5981EB7425EQ40360370-99FE5E03-898E-4108-928D-645EDD9D13FAQ41020735-EC9115B6-CDC1-47E3-B287-1B380B3BE79AQ41463008-CF33ED7D-AF40-42C2-879F-44164EF18E34Q42204562-C58B5D91-22BB-4887-AEB5-23CED1F93FE4Q45119433-E27941D0-1D4D-42CC-AC54-6460FF055397Q46621500-3B75B596-641A-4BF9-85D3-73A0B1952DF3Q47835493-47A6C4DC-D03D-478F-85E9-738DBBB04FA6Q48297715-6E46A3DB-5132-4CD3-B014-C53878174AA8Q48342467-EE307B47-2EB3-4C75-AB25-5A8DF54E5DCAQ48387856-99BEAE4D-3B05-4BF9-B1D5-6200DEB91F9DQ48482418-C552012D-9FE2-40ED-B32D-7C2085660C83Q54469037-14BF93EC-8EF3-4288-A530-4D852F8CDAC0
P2860
FGF-2 and Anosmin-1 are selectively expressed in different types of multiple sclerosis lesions.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
FGF-2 and Anosmin-1 are select ...... of multiple sclerosis lesions.
@en
FGF-2 and Anosmin-1 are select ...... of multiple sclerosis lesions.
@nl
type
label
FGF-2 and Anosmin-1 are select ...... of multiple sclerosis lesions.
@en
FGF-2 and Anosmin-1 are select ...... of multiple sclerosis lesions.
@nl
prefLabel
FGF-2 and Anosmin-1 are select ...... of multiple sclerosis lesions.
@en
FGF-2 and Anosmin-1 are select ...... of multiple sclerosis lesions.
@nl
P50
P1476
FGF-2 and Anosmin-1 are select ...... of multiple sclerosis lesions
@en
P2093
Francisco Javier Arenzana
P304
14899-14909
P356
10.1523/JNEUROSCI.1158-11.2011
P407
P577
2011-10-01T00:00:00Z