Cystic fibrosis transmembrane conductance regulator (CFTR) expression in human platelets: impact on mediators and mechanisms of the inflammatory response.
about
DHA- and EPA-derived resolvins, protectins, and maresins in airway inflammationImpaired phagocytosis in localized aggressive periodontitis: rescue by Resolvin E1Mechanisms of ATP release and signalling in the blood vessel wall.Platelets in pulmonary vascular physiology and pathology.Important role of platelets in modulating endotoxin-induced lung inflammation in CFTR-deficient mice.Purinergic signaling in early inflammatory events of the foreign body response: modulating extracellular ATP as an enabling technology for engineered implants and tissuesPhysiological impact of abnormal lipoxin A₄ production on cystic fibrosis airway epithelium and therapeutic potential.Pro-resolving lipid mediator Resolvin D1 serves as a marker of lung disease in cystic fibrosis.Resolvin D1 and Resolvin E1 Promote the Resolution of Allergic Airway Inflammation via Shared and Distinct Molecular Counter-Regulatory Pathways.The role of Lipoxin A4 in Cystic Fibrosis Lung Disease.Platelet-leukocyte interactions in cardiovascular disease and beyond.The unique contribution of ion channels to platelet and megakaryocyte function.The serum- & glucocorticoid-inducible kinase in the regulation of platelet function.Proteomics investigation of human platelets in healthy donors and cystic fibrosis patients by shotgun nUPLC-MSE and 2DE: a comparative study.Insulin signaling via the PI3-kinase/Akt pathway regulates airway glucose uptake and barrier function in a CFTR-dependent manner.Lipoxin A4 prevents tight junction disruption and delays the colonization of cystic fibrosis bronchial epithelial cells by Pseudomonas aeruginosa.Platelet proteomics applied to the search for novel antiplatelet therapeutic targets.Anti-inflammatory effects of tobramycin and a copper-tobramycin complex with superoxide dismutase-like activity.Decreased TGF-β1 and VEGF Release in Cystic Fibrosis Platelets: Further Evidence for Platelet Defects in Cystic Fibrosis.CFTR regulates acute inflammatory responses in macrophages.MicroRNA-181b regulates ALX/FPR2 receptor expression and proresolution signaling in human macrophages.microRNA-181b is increased in cystic fibrosis cells and impairs lipoxin A4 receptor-dependent mechanisms of inflammation resolution and antimicrobial defense.Annexin A1 and specialized proresolving lipid mediators: promoting resolution as a therapeutic strategy in human inflammatory diseases.Establishment and long-term culture of human cystic fibrosis endothelial cells.Specialized Pro-Resolving Lipid Mediators in Cystic Fibrosis
P2860
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P2860
Cystic fibrosis transmembrane conductance regulator (CFTR) expression in human platelets: impact on mediators and mechanisms of the inflammatory response.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Cystic fibrosis transmembrane ...... of the inflammatory response.
@en
Cystic fibrosis transmembrane conductance regulator
@nl
type
label
Cystic fibrosis transmembrane ...... of the inflammatory response.
@en
Cystic fibrosis transmembrane conductance regulator
@nl
prefLabel
Cystic fibrosis transmembrane ...... of the inflammatory response.
@en
Cystic fibrosis transmembrane conductance regulator
@nl
P2093
P50
P921
P356
P1433
P1476
Cystic fibrosis transmembrane ...... of the inflammatory response.
@en
P2093
Bianca Rocca
Francesca Pardo
Giovanni Davì
Luca Battistini
Maria Lucia Furnari
Nicola Martelli
Sara Di Silvestre
Serena Quattrucci
Stefano Lancellotti
Stefano Manarini
P304
P356
10.1096/FJ.10-159921
P407
P50
P577
2010-06-08T00:00:00Z