Repression of IP-10 by interactions between histone deacetylation and hypermethylation in idiopathic pulmonary fibrosis - Wikidata - awgldk - TriplyDB

Repression of IP-10 by interactions between histone deacetylation and hypermethylation in idiopathic pulmonary fibrosis

Repression of IP-10 by interactions between histone deacetylation and hypermethylation in idiopathic pulmonary fibrosis

http://www.wikidata.org/entity/Q39713121

about

Genetic and epigenetic regulation of intestinal fibrosis Recent advances in understanding idiopathic pulmonary fibrosis Epigenomics of idiopathic pulmonary fibrosis Personalized medicine: applying 'omics' to lung fibrosis Beyond the genome: epigenetic mechanisms in lung remodeling Evolving genomic approaches to idiopathic pulmonary fibrosis: moving beyond genes Immunomodulation by mesenchymal stem cells in treating human autoimmune disease-associated lung fibrosis Matrix metalloproteinases as therapeutic targets for idiopathic pulmonary fibrosis γδ T cells attenuate bleomycin-induced fibrosis through the production of CXCL10 Mechanisms of fibrosis: therapeutic translation for fibrotic disease Suv39h1 mediates AP-2α-dependent inhibition of C/EBPα expression during adipogenesis.Pathogenesis of idiopathic pulmonary fibrosis: review of recent findings.Combined pulmonary fibrosis and emphysema.Density and duration of pneumococcal carriage is maintained by transforming growth factor β1 and T regulatory cells Epigenetic control of gene expression in the lung.Relationship of DNA methylation and gene expression in idiopathic pulmonary fibrosis.Smoking and pulmonary fibrosis: novel insights.Integrating mechanisms of pulmonary fibrosis Epigenetic regulation of microRNAs controlling CLDN14 expression as a mechanism for renal calcium handling Smoking and idiopathic pulmonary fibrosis Cytokine-like factor 1 gene expression is enriched in idiopathic pulmonary fibrosis and drives the accumulation of CD4+ T cells in murine lungs: evidence for an antifibrotic role in bleomycin injury.Epigenetics in lung fibrosis: from pathobiology to treatment perspective.Methylation-mediated BMPER expression in fibroblast activation in vitro and lung fibrosis in mice in vivo.Profibrotic activities for matrix metalloproteinase-8 during bleomycin-mediated lung injury Microarray profiling reveals suppressed interferon stimulated gene program in fibroblasts from scleroderma-associated interstitial lung disease.Mechanistic links between aging and lung fibrosis.IDIOPATHIC PULMONARY FIBROSIS IS A COMPLEX GENETIC DISORDER.The pathogenesis of idiopathic pulmonary fibrosis.Histone methylation in myelodysplastic syndromes.Epigenetics: Concepts and relevance to IBD pathogenesis.Epigenetics within the matrix: a neo-regulator of fibrotic disease.Influence of age on wound healing and fibrosis.Molecular basis of organ fibrosis: potential therapeutic approaches.Epigenetics in immune-mediated pulmonary diseases.Epigenetics of idiopathic pulmonary fibrosis.Epigenetic targets for novel therapies of lung diseases.Epigenetics in idiopathic pulmonary fibrosis.Epigenetic regulation of cardiac fibrosis.Thrombin Induces CCL2 Expression in Human Lung Fibroblasts via p300 Mediated Histone Acetylation and NF-KappaB Activation.Histone deacetylase inhibitor restores surfactant protein-C expression in alveolar-epithelial type II cells and attenuates bleomycin-induced pulmonary fibrosis in vivo.

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Repression of IP-10 by interactions between histone deacetylation and hypermethylation in idiopathic pulmonary fibrosis

http://www.wikidata.org/entity/Q39713121

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

2010年论文

@zh

2010年论文

@zh-cn

name

Repression of IP-10 by interac ...... idiopathic pulmonary fibrosis

@en

Repression of IP-10 by interac ...... idiopathic pulmonary fibrosis.

@nl

type

label

Repression of IP-10 by interac ...... idiopathic pulmonary fibrosis

@en

Repression of IP-10 by interac ...... idiopathic pulmonary fibrosis.

@nl

prefLabel

Repression of IP-10 by interac ...... idiopathic pulmonary fibrosis

@en

Repression of IP-10 by interac ...... idiopathic pulmonary fibrosis.

@nl

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Molecular and Cellular Biology

P1476

Repression of IP-10 by interac ...... idiopathic pulmonary fibrosis

@en

P2093

Carol A Feghali-Bostwick

http://www.w3.org/2001/XMLSchema#string

Keira Watts

http://www.w3.org/2001/XMLSchema#string

Linhua Pang

http://www.w3.org/2001/XMLSchema#string

William R Coward

http://www.w3.org/2001/XMLSchema#string

P2860

Regulation of chromatin structure by site-specific histone H3 methyltransferases

The HP1alpha-CAF1-SetDB1-containing complex provides H3K9me1 for Suv39-mediated K9me3 in pericentric heterochromatin

The DNA methyltransferases associate with HP1 and the SUV39H1 histone methyltransferase

SETDB1: a novel KAP-1-associated histone H3, lysine 9-specific methyltransferase that contributes to HP1-mediated silencing of euchromatic genes by KRAB zinc-finger proteins

Methylation of histone H3 lysine 9 creates a binding site for HP1 proteins

Selective recognition of methylated lysine 9 on histone H3 by the HP1 chromo domain

DNA methyltransferase Dnmt1 associates with histone deacetylase activity

CoREST is an integral component of the CoREST- human histone deacetylase complex

Set domain-containing protein, G9a, is a novel lysine-preferring mammalian histone methyltransferase with hyperactivity and specific selectivity to lysines 9 and 27 of histone H3

Molecular cloning of ESET, a novel histone H3-specific methyltransferase that interacts with ERG transcription factor

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2874-2886

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scholarly article

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10.1128/MCB.01527-09

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12

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43201459

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20404089

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pulmonary fibrosis

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2876687

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