Role of epidermal growth factor receptor degradation in cisplatin-induced cytotoxicity in head and neck cancer - Wikidata - awgldk - TriplyDB

Role of epidermal growth factor receptor degradation in cisplatin-induced cytotoxicity in head and neck cancer

Role of epidermal growth factor receptor degradation in cisplatin-induced cytotoxicity in head and neck cancer

http://www.wikidata.org/entity/Q39730112

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EGFR nuclear translocation modulates DNA repair following cisplatin and ionizing radiation treatment Improving the efficacy of chemoradiation with targeted agents Protein-intrinsic and signaling network-based sources of resistance to EGFR- and ErbB family-targeted therapies in head and neck cancer Epidermal growth factor protects squamous cell carcinoma against cisplatin-induced cytotoxicity through increased interleukin-1β expression Membrane Transition Temperature Determines Cisplatin Response YM155 induces EGFR suppression in pancreatic cancer cells.Ubiquitination-dependent regulation of signaling receptors in cancer.Molecular mechanism of the schedule-dependent synergistic interaction in EGFR-mutant non-small cell lung cancer cell lines treated with paclitaxel and gefitinib.Enhanced anticancer effect of the combination of cisplatin and TRAIL in triple-negative breast tumor cells Effect of erlotinib on epidermal growth factor receptor and downstream signaling in oral cavity squamous cell carcinoma.Early tumor progression associated with enhanced EGFR signaling with bortezomib, cetuximab, and radiotherapy for head and neck cancer.STAT1 Activation Is Enhanced by Cisplatin and Variably Affected by EGFR Inhibition in HNSCC Cells Optical molecular imaging of multiple biomarkers of epithelial neoplasia: epidermal growth factor receptor expression and metabolic activity in oral mucosa.Degradation of epidermal growth factor receptor mediates dasatinib-induced apoptosis in head and neck squamous cell carcinoma cells.Wild-type EGFR is stabilized by direct interaction with HSP90 in cancer cells and tumors.Tetrathiomolybdate mediates cisplatin-induced p38 signaling and EGFR degradation and enhances response to cisplatin therapy in gynecologic cancers.High efficacy of CpG-ODN, cetuximab and cisplatin combination for very advanced ovarian xenograft tumors.Destabilization of the epidermal growth factor receptor (EGFR) by a peptide that inhibits EGFR binding to heat shock protein 90 and receptor dimerization.Targeted in vivo delivery of EGFR siRNA inhibits ovarian cancer growth and enhances drug sensitivity.Cbl-b enhances sensitivity to 5-fluorouracil via EGFR- and mitochondria-mediated pathways in gastric cancer cells.Efficacy of an EGFR-specific peptide against EGFR-dependent cancer cell lines and tumor xenografts.Phosphoproteomics Reveals HMGA1, a CK2 Substrate, as a Drug-Resistant Target in Non-Small Cell Lung Cancer.Differential protein stability of EGFR mutants determines responsiveness to tyrosine kinase inhibitors.Molecular biology of squamous cell carcinoma of the head and neck: relevance and therapeutic implications.EGFR targeted therapies and radiation: Optimizing efficacy by appropriate drug scheduling and patient selection.Mechanisms of Resistance to EGFR Tyrosine Kinase Inhibitors and Therapeutic Approaches: An Update.Constitutively activated ERK sensitizes cancer cells to doxorubicin: Involvement of p53-EGFR-ERK pathway.Soluble guanylate cyclase stimulators increase sensitivity to cisplatin in head and neck squamous cell carcinoma cells.Antiglioma effects of N6-isopentenyladenosine, an endogenous isoprenoid end product, through the downregulation of epidermal growth factor receptor.Cisplatin and ultra-violet-C synergistically down-regulate receptor tyrosine kinases in human colorectal cancer cells Regulation of EGFR protein stability by the HECT-type ubiquitin ligase SMURF2.Molecular imaging of akt enables early prediction of response to molecular targeted therapy.Mechanisms for DNA-damaging agent-induced inactivation of ErbB2 and ErbB3 via the ERK and p38 signaling pathways.Erlotinib-induced generalized eczema craquelé and follicular rash sparing the previous radiation field.Small Molecules Identified from a Quantitative Drug Combinational Screen Resensitize Cisplatin's Response in Drug-Resistant Ovarian Cancer Cells.Dicer affects cisplatin‑mediated apoptosis in epithelial ovarian cancer cells

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Role of epidermal growth factor receptor degradation in cisplatin-induced cytotoxicity in head and neck cancer

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2010年论文

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Role of epidermal growth facto ...... xicity in head and neck cancer

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Role of epidermal growth facto ...... icity in head and neck cancer.

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Role of epidermal growth facto ...... xicity in head and neck cancer

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Role of epidermal growth facto ...... icity in head and neck cancer.

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Role of epidermal growth facto ...... xicity in head and neck cancer

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0008-5472.CAN-09-4294

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Cancer Research

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Role of epidermal growth facto ...... xicity in head and neck cancer

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Aarif Ahsan

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Abigail Helman

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Ashok Hegde

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Mahaveer S Bhojani

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Mukesh K Nyati

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Radhika Menawat

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Shyam Nyati

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Susmita G Ramanand

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Theodore S Lawrence

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P2860

Survival of cancer cells is maintained by EGFR independent of its kinase activity

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Crystal structure of double-stranded DNA containing the major adduct of the anticancer drug cisplatin

Recurrent fusion of TMPRSS2 and ETS transcription factor genes in prostate cancer.

Signal transduction by receptors with tyrosine kinase activity

Ubiquitin ligase activity and tyrosine phosphorylation underlie suppression of growth factor signaling by c-Cbl/Sli-1

c-Cbl/Sli-1 regulates endocytic sorting and ubiquitination of the epidermal growth factor receptor

Induction chemotherapy plus radiation compared with surgery plus radiation in patients with advanced laryngeal cancer.

Epidermal growth factor regulates the in vitro sensitivity of human ovarian carcinoma cells to cisplatin.

The resurgence of platinum-based cancer chemotherapy.

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Susan M Hiniker

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20215522

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