The p65 subunit of NF-kappaB binds to PGC-1alpha, linking inflammation and metabolic disturbances in cardiac cells.
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Skeletal muscle PGC-1α is required for maintaining an acute LPS-induced TNFα responseZBTB2 increases PDK4 expression by transcriptional repression of RelA/p65FGF21 and Cardiac PhysiopathologyRedox regulation of mitochondrial biogenesisRegulation of mitochondrial biogenesis and its intersection with inflammatory responsesRedox signaling in skeletal muscle: role of aging and exerciseTraining-induced mitochondrial adaptation: role of peroxisome proliferator-activated receptor γ coactivator-1α, nuclear factor-κB and β-blockadeSalidroside stimulates mitochondrial biogenesis and protects against H₂O₂-induced endothelial dysfunction.Role of PGC-1α in exercise training- and resveratrol-induced prevention of age-associated inflammationLoss of Pgc-1α expression in aging mouse muscle potentiates glucose intolerance and systemic inflammationThe interplay between NF-kappaB and E2F1 coordinately regulates inflammation and metabolism in human cardiac cells.PGC-1α induces mitochondrial and myokine transcriptional programs and lipid droplet and glycogen accumulation in cultured human skeletal muscle cells.Suppression of mitochondrial biogenesis through toll-like receptor 4-dependent mitogen-activated protein kinase kinase/extracellular signal-regulated kinase signaling in endotoxin-induced acute kidney injury.PGC1α plays a critical role in TWEAK-induced cardiac dysfunction.Conditional knockout of prolyl hydroxylase domain protein 2 attenuates high fat-diet-induced cardiac dysfunction in mice.Peroxisome proliferator-activated receptor γ coactivator 1α and FoxO3A mediate chondroprotection by AMP-activated protein kinase.Toll-like receptor-mediated inflammatory signaling reprograms cardiac energy metabolism by repressing peroxisome proliferator-activated receptor γ coactivator-1 signaling.Astragaloside IV protects against isoproterenol-induced cardiac hypertrophy by regulating NF-κB/PGC-1α signaling mediated energy biosynthesisFenofibrate improves renal lipotoxicity through activation of AMPK-PGC-1α in db/db mice.Adenosine prevents TNFα-induced decrease in endothelial mitochondrial mass via activation of eNOS-PGC-1α regulatory axis.PGC-1α overexpression by in vivo transfection attenuates mitochondrial deterioration of skeletal muscle caused by immobilization.miR-146a targets Fos expression in human cardiac cellsA novel statistical approach for identification of the master regulator transcription factor.Mitochondrial Alterations in Peripheral Mononuclear Blood Cells from Alzheimer's Disease and Mild Cognitive Impairment Patients.Overexpression of human selenoprotein H in neuronal cells enhances mitochondrial biogenesis and function through activation of protein kinase A, protein kinase B, and cyclic adenosine monophosphate response element-binding protein pathwayAcute Response of PGC-1α and IGF-1 Isoforms to Maximal Eccentric Exercise in Skeletal Muscle of Postmenopausal Women.Chronic ethanol consumption increases cardiomyocyte fatty acid uptake and decreases ventricular contractile function in C57BL/6J miceSustained elevation of NF-κB activity sensitizes offspring of maternal inflammation to hypertension via impairing PGC-1α recovery.The NO/ONOO-cycle as the central cause of heart failure.Postmortem mRNA expression patterns in left ventricular myocardial tissues and their implications for forensic diagnosis of sudden cardiac death.AMP-activated protein kinase inhibits NF-κB signaling and inflammation: impact on healthspan and lifespan.Transcriptional control of mitochondrial biogenesis and its interface with inflammatory processes.AMP-activated protein kinase: an emerging drug target to regulate imbalances in lipid and carbohydrate metabolism to treat cardio-metabolic diseasesRole of PGC-1α signaling in skeletal muscle health and disease.Functional crosstalk of PGC-1 coactivators and inflammation in skeletal muscle pathophysiology.Acute rhabdomyolysis and inflammation.A Review of the Molecular Mechanisms Underlying the Development and Progression of Cardiac Remodeling.A Role for Peroxisome Proliferator-Activated Receptor Gamma Coactivator-1α in Nucleus Accumbens Neuron Subtypes in Cocaine Action.An overview of the inflammatory signalling mechanisms in the myocardium underlying the development of diabetic cardiomyopathy.Reduced expression of PGC-1α partly underlies mitochondrial changes and correlates with neuronal loss in multiple sclerosis cortex.
P2860
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P2860
The p65 subunit of NF-kappaB binds to PGC-1alpha, linking inflammation and metabolic disturbances in cardiac cells.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
The p65 subunit of NF-kappaB b ...... disturbances in cardiac cells.
@en
The p65 subunit of NF-kappaB b ...... disturbances in cardiac cells.
@nl
type
label
The p65 subunit of NF-kappaB b ...... disturbances in cardiac cells.
@en
The p65 subunit of NF-kappaB b ...... disturbances in cardiac cells.
@nl
prefLabel
The p65 subunit of NF-kappaB b ...... disturbances in cardiac cells.
@en
The p65 subunit of NF-kappaB b ...... disturbances in cardiac cells.
@nl
P2093
P356
P1476
The p65 subunit of NF-kappaB b ...... disturbances in cardiac cells
@en
P2093
Arthur M Feldman
David Alvarez-Guardia
Manuel Vázquez-Carrera
Mercy M Davidson
Teresa Coll
Xavier Palomer
P304
P356
10.1093/CVR/CVQ080
P577
2010-03-07T00:00:00Z