Controlled cortical impact traumatic brain injury in 3xTg-AD mice causes acute intra-axonal amyloid-β accumulation and independently accelerates the development of tau abnormalities
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Chronic traumatic encephalopathy in blast-exposed military veterans and a blast neurotrauma mouse model.The spectrum of disease in chronic traumatic encephalopathyChronic impact of traumatic brain injury on outcome and quality of life: a narrative reviewMechanical Stress as the Common Denominator between Chronic Inflammation, Cancer, and Alzheimer's DiseaseAcute and chronic traumatic encephalopathies: pathogenesis and biomarkers.Animal models of traumatic brain injuryUsing Drosophila as an integrated model to study mild repetitive traumatic brain injury.The liver X receptor agonist GW3965 improves recovery from mild repetitive traumatic brain injury in mice partly through apolipoprotein EThe Controlled Cortical Impact Model: Applications, Considerations for Researchers, and Future DirectionsSystems biology of neurodegenerative diseasesMolecular mechanisms of cognitive dysfunction following traumatic brain injuryAssociation of traumatic brain injury with subsequent neurological and psychiatric disease: a meta-analysisPharmacological characterization of memoquin, a multi-target compound for the treatment of Alzheimer's diseaseImmediate, but not delayed, microsurgical skull reconstruction exacerbates brain damage in experimental traumatic brain injury modelAlzheimer’s disease and chronic traumatic encephalopathy: distinct but possibly overlapping disease entitiesEffects of low-level blast exposure on the nervous system: is there really a controversy?Chronic traumatic encephalopathy: clinical-biomarker correlations and current concepts in pathogenesis.Acute blast injury reduces brain abeta in two rodent species.Presenilin controls kinesin-1 and dynein function during APP-vesicle transport in vivoPerivascular AQP4 dysregulation in the hippocampal CA1 area after traumatic brain injury is alleviated by adenosine A2A receptor inactivation.Microglial Activation in Traumatic Brain Injury.Dementia resulting from traumatic brain injury: what is the pathology?Distinct temporal and anatomical distributions of amyloid-β and tau abnormalities following controlled cortical impact in transgenic miceAxonal pathology in traumatic brain injury.Inflammation and white matter degeneration persist for years after a single traumatic brain injury.Ablation of matrix metalloproteinase-9 gene decreases cerebrovascular permeability and fibrinogen deposition post traumatic brain injury in mice.Traumatic brain injury precipitates cognitive impairment and extracellular Aβ aggregation in Alzheimer's disease transgenic mice.RNA-sequencing reveals oligodendrocyte and neuronal transcripts in microglia relevant to central nervous system disease.Histological quantitation of brain injury using whole slide imaging: a pilot validation study in mice.Closed head injury in an age-related Alzheimer mouse model leads to an altered neuroinflammatory response and persistent cognitive impairment.The pathophysiology of repetitive concussive traumatic brain injury in experimental models; new developments and open questionsAmyloid precursor protein revisited: neuron-specific expression and highly stable nature of soluble derivatives.A neurovascular perspective for long-term changes after brain trauma.Administration of COG1410 reduces axonal amyloid precursor protein immunoreactivity and microglial activation after controlled cortical impact in mice.Lithium reduces BACE1 overexpression, β amyloid accumulation, and spatial learning deficits in mice with traumatic brain injury.Modeling Chronic Traumatic Encephalopathy: The Way Forward for Future Discovery.Polypathology and dementia after brain trauma: Does brain injury trigger distinct neurodegenerative diseases, or should they be classified together as traumatic encephalopathy?Dendritic Spine Loss and Chronic White Matter Inflammation in a Mouse Model of Highly Repetitive Head Trauma.Rapid accumulation of endogenous tau oligomers in a rat model of traumatic brain injury: possible link between traumatic brain injury and sporadic tauopathiesAmyloid-β Peptides and Tau Protein as Biomarkers in Cerebrospinal and Interstitial Fluid Following Traumatic Brain Injury: A Review of Experimental and Clinical Studies.
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P2860
Controlled cortical impact traumatic brain injury in 3xTg-AD mice causes acute intra-axonal amyloid-β accumulation and independently accelerates the development of tau abnormalities
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh
2011年學術文章
@zh-hant
name
Controlled cortical impact tra ...... velopment of tau abnormalities
@en
Controlled cortical impact tra ...... elopment of tau abnormalities.
@nl
type
label
Controlled cortical impact tra ...... velopment of tau abnormalities
@en
Controlled cortical impact tra ...... elopment of tau abnormalities.
@nl
prefLabel
Controlled cortical impact tra ...... velopment of tau abnormalities
@en
Controlled cortical impact tra ...... elopment of tau abnormalities.
@nl
P2093
P2860
P1476
Controlled cortical impact tra ...... velopment of tau abnormalities
@en
P2093
David L Brody
Frank M LaFerla
Hien T Tran
P2860
P304
P356
10.1523/JNEUROSCI.0858-11.2011
P407
P577
2011-06-01T00:00:00Z