Interactions between Brucella melitensis and human phagocytes: bacterial surface O-Polysaccharide inhibits phagocytosis, bacterial killing, and subsequent host cell apoptosis.
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Proteomic analysis of detergent resistant membrane domains during early interaction of macrophages with rough and smooth Brucella melitensisEntrance and survival of Brucella pinnipedialis hooded seal strain in human macrophages and epithelial cellsMolecular targets for rapid identification of Brucella spp.Caspase-2 mediated apoptotic and necrotic murine macrophage cell death induced by rough Brucella abortusFrancisella tularensis Schu S4 O-antigen and capsule biosynthesis gene mutants induce early cell death in human macrophages.Regulation of the mitogen-activated protein kinases by Brucella spp. expressing a smooth and rough phenotype: relationship to pathogen invasiveness.Deep-sequencing analysis of the mouse transcriptome response to infection with Brucella melitensis strains of differing virulence.Smooth and rough lipopolysaccharide phenotypes of Brucella induce different intracellular trafficking and cytokine/chemokine release in human monocytes.Immunization of mice with gamma-irradiated Brucella neotomae and its recombinant strains induces protection against virulent B. abortus, B. melitensis, and B. suis challengeComparative analysis of the early transcriptome of Brucella abortus--infected monocyte-derived macrophages from cattle naturally resistant or susceptible to brucellosisBrucella abortus rough mutants induce macrophage oncosis that requires bacterial protein synthesis and direct interaction with the macrophage.Comparative genomic analysis of Brucella melitensis vaccine strain M5 provides insights into virulence attenuationA B lymphocyte mitogen is a Brucella abortus virulence factor required for persistent infection.Lipopolysaccharide-Deficient Brucella Variants Arise Spontaneously during Infection.Complete genome-wide screening and subtractive genomic approach revealed new virulence factors, potential drug targets against bio-war pathogen Brucella melitensis 16M.Characterization of recombinant B. abortus strain RB51SOD toward understanding the uncorrelated innate and adaptive immune responses induced by RB51SOD compared to its parent vaccine strain RB51Evaluation of Brucella abortus phosphoglucomutase (pgm) mutant as a new live rough-phenotype vaccineSmooth to Rough Dissociation in Brucella: The Missing Link to VirulenceBrucella spp noncanonical LPS: structure, biosynthesis, and interaction with host immune system.Brucellosis vaccines: assessment of Brucella melitensis lipopolysaccharide rough mutants defective in core and O-polysaccharide synthesis and exportToll-like receptor 4-linked Janus kinase 2 signaling contributes to internalization of Brucella abortus by macrophagesN-Formyl-Perosamine Surface Homopolysaccharides Hinder the Recognition of Brucella abortus by Mouse Neutrophils.Persistence of the bacterial pathogen Granulibacter bethesdensis in chronic granulomatous disease monocytes and macrophages lacking a functional NADPH oxidase.Full-Length cDNA Cloning, Molecular Characterization and Differential Expression Analysis of Lysophospholipase I from Ovis aries.OMP31 of Brucella melitensis 16M impairs the apoptosis of macrophages triggered by TNF-αMycobacterium avium MAV2054 protein induces macrophage apoptosis by targeting mitochondria and reduces intracellular bacterial growth.Increased rate of apoptosis and diminished phagocytic ability of human neutrophils infected with Afa/Dr diffusely adhering Escherichia coli strains.Internal affairs: investigating the Brucella intracellular lifestyle.Analyses of Brucella pathogenesis, host immunity, and vaccine targets using systems biology and bioinformatics.Brucella melitensis triggers time-dependent modulation of apoptosis and down-regulation of mitochondrion-associated gene expression in mouse macrophages.Brucella melitensis, B. neotomae and B. ovis elicit common and distinctive macrophage defense transcriptional responsesPromotion and Rescue of Intracellular Brucella neotomae Replication during Coinfection with Legionella pneumophilaBrucella dissociation is essential for macrophage egress and bacterial dissemination.Entry and intracellular replication of Escherichia coli K1 in macrophages require expression of outer membrane protein A.Evidence of Brucella abortus OPS dictating uptake and restricting NF-kappaB activation in murine macrophages.Cytotoxicity in macrophages infected with rough Brucella mutants is type IV secretion system dependent.High susceptibility of human dendritic cells to invasion by the intracellular pathogens Brucella suis, B. abortus, and B. melitensis.Inhibition of MHC-I by Brucella abortus is an early event during infection and involves EGFR pathway.Trichomonas vaginalis-induced apoptosis in RAW264.7 cells is regulated through Bcl-xL, but not Bcl-2.Different responses of macrophages to smooth and rough Brucella spp.: relationship to virulence
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P2860
Interactions between Brucella melitensis and human phagocytes: bacterial surface O-Polysaccharide inhibits phagocytosis, bacterial killing, and subsequent host cell apoptosis.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
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2003年學術文章
@zh-hant
name
Interactions between Brucella ...... ubsequent host cell apoptosis.
@en
Interactions between Brucella ...... ubsequent host cell apoptosis.
@nl
type
label
Interactions between Brucella ...... ubsequent host cell apoptosis.
@en
Interactions between Brucella ...... ubsequent host cell apoptosis.
@nl
prefLabel
Interactions between Brucella ...... ubsequent host cell apoptosis.
@en
Interactions between Brucella ...... ubsequent host cell apoptosis.
@nl
P2093
P2860
P921
P1476
Interactions between Brucella ...... ubsequent host cell apoptosis.
@en
P2093
Carmen M Fernandez-Prada
David L Hoover
Elzbieta B Zelazowska
Gregory L Robertson
Mikeljon Nikolich
R Martin Roop
Ted L Hadfield
P2860
P304
P356
10.1128/IAI.71.4.2110-2119.2003
P407
P577
2003-04-01T00:00:00Z